Stroke volume fit

Common Questions and Answers about Stroke volume fit

stroke

Avatar m tn CO is a product of blood volume per heart stroke and heart rate. The higher/better the stroke volume (weak, strong, stronger contractions) the fewer heart beats (more for weak, less for strong, least for stronger) are required to meet demand (held constant for illustration) for blood oxygen. A well-condition individual requires fewer heartbeats due to strong contractions. It is the stroke that determines EF...not the CO, as CO can vary by an increase or decrease of HR.
1124887 tn?1313754891 EF is a compensating factor and helps maintain a balancing flow of blood between the right and left side of the heart. Stroke volume is inverse to the heart rate. Slow heart rate the greater the stroke volume which indicates the heart rate is also a compensating factor...the slower the heart rate the more time to fill. Wide pulse pressure (no medication, etc) can be the outcome of increased CO due to fitness, anxiety, hyperthyroidism, etc.
Avatar f tn I am a healthy, fit, and very active 47 year old woman--healthy lifestyle and diet, runner, and I ride my bike for transportation. Resting HR of 52. PACs started a few weeks ago, so got a Holtor which reported over 4000 PACs/24 hours (that was half of what they had been previously), sleeping HR went as low as 41. Had an echocardiogram the other day, and everything was normal, except: "severely increased left atrial volume." Mysteriously, left atrial *size* (i.e., diameter) was normal.
Avatar n tn i have been suffering from shortness of breath for 5 months.recently i have a echocardiogram. it shows that my stroke volume is 47 ml . is this is a normal value or it is a sign of a heart disease? my ef is 61%.
Avatar n tn Some enlargement is expected if you are quite fit. The more intense the exercise regime, the more the heart will naturally adapt, up to a limit. This is why athletes hearts can pump blood so much more efficiently around the body. The heart muscle increases and the internal volume increases, allowing more blood to be pushed out with each stroke.
Avatar n tn I am in the UK. I was very, very fit, ran, lived at the gym, windsurfed & kart raced. I had a very, very bad stroke in March 04, I was 42. I nearly died, which is an effect! Lol. Now in a wheelchair, speach is very, very bad & I am virtually paralysed. I wrote a 68,000 word book, which I cannot advertise here. Doctors said I would just have eye movement. I believe I am still recovering. Tim.
Avatar m tn This happens by a system of vasodilation and vasoconstriction coupled with the pumping rate and stroke volume of the heart. Rather than make frequent changes in volume, the body changes the configuration of the total container (all the vessels) and increases the speed at which the resource is renewed.
Avatar m tn It's been 2yrs since my stroke and I have no voluntary movement in my left arm or hand. Is there any possibilty that i could recover any movement? My neurlogist told me that if within 6month of the stroke if I didnt have any movement that my hand will stay paralyzed?
Avatar m tn A faster heartbeat does not provide enough time to fill as at rest, and the timing of contractions are less. The percent of blood pumped with each heartbeat is less reducing stroke volume (lower EF). The heart rate has increased to provide the necessary blood/oxygen to meet the system's demand for the diminished stroke volume. The CT scan indicates there is substanital soft plaque buildup within the linings of the vessels.
Avatar n tn Diastolic volume (filling phase) per stroke is blood volume measured at end of diastolic (maximum filling capacity...relaxed state) minus the volume at the end of systole (minimum amount of blood left in the chamber). Divide end diastolic volume into the difference between end systole and end diastole is the fraction (%) pumped with each stroke. CHF by definition is an EF below 29% (left side non-compliance) indicating there may be an insufficient blood/oxygen supply meeting system demand.
Avatar m tn The volume of blood left in a ventricle at the end of contraction is end-systolic volume. The difference between end-diastolic and end-systolic volumes is the stroke volume, the volume of blood ejected with each beat. Ejection fraction (Ef) is the fraction of the end-diastolic volume that is ejected with each beat; it is stroke volume divided by end-diastolic volume. Normal in a man is 50-68%.
Avatar m tn For a perspective, diastole dysfunction compromises left ventricle filling volume, inadequate stroke volume and operating at a high pressure regimen. LV filling volume is reduced due to increased wall size crowding out available space. Inadequate stroke volume is usually due to wall thickening and stiffening and loss of elasticity. High pressures can dilate upper chamber to compensate for gradient pressure across the mitral valve. That is the pathology.
1591333 tn?1297733419 These mechanisms would be interrrelated. The low blood supply (volume in circulation) the kidneys will increase the volume (an example would blood loss, or a low cardiac output). The left venticle will dilate to increase blood flow with each stroke and the heart rate will increase the cardiac output (amount of blood pumped per minute). If the system is dysfunctional, medication, etc. can change the dynamics.
Avatar n tn My niece had an MRI recently and one of the findings said volume loss of the posterior fossa. What does that mean exactly? She has speech, vision, hearing and balance problems that we have never really had explained as to why she has them? Are they all releated to what they found on the MRI?
720516 tn?1230646225 d expect the stroke volume to be inverse to the Heart Rate - e.g. a well-conditioned person would have a low HR with a low BP but a high stroke volume with every beat. As far as poisoning, it'd seem natural for the system to go into high gear to remove the poison - but 4 months would sure be a long time for that, unless it created some semi-permanent change in you somehow.
Avatar f tn what happens with significant MVR some blood fails to pumped into circulation with each stroke. To maintain adequate blood flow with a decrease in stroke volume is to increase the heart rate. The math for cardiac output is stroke volume times heart rate for one minute. Frankly, I don't believe you have significant MVR because you should also have the symptoms of shortness of breath with exertion and muscle fatigue.
Avatar m tn An echo calculates heart chamber diminsions and volume of blood pumped with each stroke. The math equation is diastole volume (peak filling) minus systole (volume after pumping) divided by diastole for the percentage. You can calculate your fraction shortening (FS) which is similar to EF but relies on dimensions rather than volume. To calculate subtract systole dimension from diastole dimension and divide by diastole for percentage. FS is an estimate of myocardial contractility.
Avatar m tn Hey, i just got this off some Dr's site ( http://www.netwellness.org/question.cfm/35267.htm ) The cause of white matters signal abnormality could be caused by: Aging and risk factors such as hypertension and diabetes. Sleep apnea, with the episodes of hypoxia, is another cause of these findings. They are not exactly "normal" findings, as they are associated with increased risk for stroke and cognitive decline in the long term, but are not always indicative of a stroke.
Avatar n tn CO (volume of blood pumped with each heartbeat) there is a variation of volume of blood pumped with each stroke to maintain a balance between left and right side blood flow) and abviously PAP will vary accordingly (stronger heartbeat,higher PAP. If there are blockages (clots) in the lungs or pulmonary artery (increase of resistance the heart pumps against, and/or if there is narrowing, stiff pulmonary vessel the more resistance (increase of resistance will cause higher PAP).
Avatar m tn For instance, how are systemic vascular resistance, blood volume, stroke volume, cardiac output, etc affected. I am wondering if an HOCM heart relies on a hyperdynamic state from hyperthyroidism, and if going euthyroid will harm the cat.
Avatar n tn So I am thinking that I am just very deconditioned and my stroke volume is low and I need to wait for changes in muscle growth and circulation in my legs too. Am I too old to really increase my heart efficiency and stroke volume now?
1527234 tn?1291771442 In this phase due to F/S phenomonon the ventricular myocardium is no longer able to contract adequately to compensate for the volume overload of mitral regurgitation (volume overload can be do to other causes), and the stroke volume of the left ventricle will decrease. The decreased stroke volume causes a decreased forward cardiac output and an increase in the end-systolic volume dilates left ventricle.