# Stroke volume and ejection fraction

## stroke

Volume of left venticle after pumping contractions subtracted from volume after filling and that result divided by by filling volume provides the ejection fraction. There is a margin of error of about 5% plus or minus and in absolute terms there is a variation of stroke volume to help maintain a balance of blood flow of the leftside of the heart with the rightside.
Normal LA, RA, LV and RV dimensions and wall thickness, normal valve function. Normal ejection fraction 57%. Aorta abdominalis normal with 17mm diameter. Dx: I49.1 Premature atrial depolarization and F45.3 cardiac neurosis. From what I understand and have read, those results are excellent. However, I think my EF is a bit low. Is this caused by a somewhat poor exercise condition? Can it increase with exercise?
i have been suffering from shortness of breath for 5 months.recently i have a echocardiogram. it shows that my stroke volume is 47 ml . is this is a normal value or it is a sign of a heart disease? my ef is 61%.
An echo calculates heart chamber diminsions and volume of blood pumped with each stroke. The math equation is diastole volume (peak filling) minus systole (volume after pumping) divided by diastole for the percentage. You can calculate your fraction shortening (FS) which is similar to EF but relies on dimensions rather than volume. To calculate subtract systole dimension from diastole dimension and divide by diastole for percentage. FS is an estimate of myocardial contractility.
But you can google a chart for her age and sex and what her results should be. Her ejection fraction is on the lower end of normal. Well within normal though. That is ( stroke volume/edv)×100. Stroke volume = amount your heart pumps with each beat Heart rate is= beats per minute Qt or CO ( cardiac output)= sv×hr ( so total pumped in 1 minutes) Edv= is end diastolic volume.
The volume of blood left in a ventricle at the end of contraction is end-systolic volume. The difference between end-diastolic and end-systolic volumes is the stroke volume, the volume of blood ejected with each beat. Ejection fraction (Ef) is the fraction of the end-diastolic volume that is ejected with each beat; it is stroke volume divided by end-diastolic volume. Normal in a man is 50-68%.
In this phase due to F/S phenomonon the ventricular myocardium is no longer able to contract adequately to compensate for the volume overload of mitral regurgitation (volume overload can be do to other causes), and the stroke volume of the left ventricle will decrease. The decreased stroke volume causes a decreased forward cardiac output and an increase in the end-systolic volume dilates left ventricle.
1. Small area of very mild inferior ischemia. 2. No regional wall abnormalities. 3. Calculated EF 76%. At first, I though having an EF higher than 70% was a good indicator since good is 50-70%. But I have since read that "An ejection fraction measurement higher than 75 percent may indicate a heart condition such as hypertrophic cardiomyopathy." Thank you in advance for yout advice.
My ejection fraction is inconsitant, Ranging from a high of 42 in 1979 to a recent low of 16. and yet I play doubles tennis 3 days a weeks for 2 hours with no shortness of breath. Do modest weights, walk a mile a day, 6 foot 2, 195 pounds, 36 waist. I was off meds for 3 days prior to test. Is it possible that attrial fribullation, pvc's can lower my ejection fraction number? I am 74 and feel well. my HDL is 40 , LDL 40 VlDL 8. I take coumadin for attrial fribullation.
Stroke volume (SV) depends on body size. A better measure for heart function is ejection fraction which is also measured on echo. How high is yours? If your ejection fraction is normal and the heart relaxes properly between the beats (diastolic function) you don't have heart failure or cardiomyopathy.
To normally compensate EF increases (range 55-75%) by dilating the LV chamber and that increases contractual strength and blood volume in keeping with physics (FRANK/STARLING mechanism) thereby enhancing performance. The EF usually represents the percentage of blood pumped into circulation with each stroke, but with mitral valve insufficiency there is an amount something less than estimated EF goes into circulation as some blood flows back into the upper chamber.
Coreg 25 x2, and accupril (ace inhibitor). Latest echo shows no improvement of ejection fraction. Still 24 % after 8 months on meds. Anyone have any opinion as to chances of any improvement from meds at this point? LV has reduced moderately from 6.94. I believe my heart suffered damage from serious auto accident (BFT). I was hoping for signs of an improvement.
Cardiac output determines your bodies response. It is a somple math equation. Stroke volume (how strong and effective your pump is) x rate. I had to get a pacemaker to deal with all the meds that are used to treat my heart. I love the thing. I makes daily life a more sure thing. It was the highlight of 2008. Good luck with the meds. It is a really small dose of one of the more innocuous medicines and so it isn't likely to cause you too much problems.
Your doctor says ignore stroke volume?? Gosh! Geewhiz! Stroke volume is the determinate of EF (ejection fraction...means the amount of blood voume pumped out of the left ventricle with each heart stroke. I gave you the formula! It is end diastole (filling phase) and that would be the maximum amount of blood volume for a heart beat.
my father age is 67 his ejection fraction is 34 and fractional shortening is 17 he has cough not severe and some time he has vomiting 4 to 5 ttimes he has breath shortening also kindly guide me what is dangerous level of ejection fraction and fractional shortening and what is survival rate of such patient
Low cardiac output casues a faster heart rate, triggers the kidneys to produce more volume and the additional volume stresses the heart as well and heart failure ensues. Treatment is to reduce fluids, dilate vessels, slow heart rate, sometimes meds to strengthen contractility, etc. The treatment can/will provide relief to the heart's workload and in time the heart will/can reverse the LV enlargement. Currently, my heart is normal size and functionality with the exception of MVR.
An increased preload increases stroke volume through the Frank Starling mechanism and that would be an increase in EF. Frank Starling mechanism can be campared to a hand spring as an analogy. If the hand spring is slightly stretched the recoil is stronger, but if over stretched the hand spring loses its elasticiy as does an overly dilated left ventricle.
For some insight, the assessment of left and right ventricular diastolic function (filling phase) at rest in patients with enlarged heart and/or decreased ejection fraction, can be done in an excellent way using Doppler – echocardiographic methods or other imaging techniques (CT scan, MRI, etc), the assessment of LV diastolic function at Rest & with Exercise in subjects/patients with small LV cavity and normal LV ejection fraction, can at best be done by using an optimal EXTERNAL PRESSURE
For a perspective, diastole dysfunction compromises left ventricle filling volume, inadequate stroke volume and operating at a high pressure regimen. LV filling volume is reduced due to increased wall size crowding out available space. Inadequate stroke volume is usually due to wall thickening and stiffening and loss of elasticity. High pressures can dilate upper chamber to compensate for gradient pressure across the mitral valve. That is the pathology.