Stroke volume training

Common Questions and Answers about Stroke volume training


Avatar m tn Over many, many years, with high blood pressure and being very overweight, the inside of your ventrical gets thicker, and the volume inside therefore decreases, decreasing stroke volume and causing your heart rate to become higher and your heart then has to work harder to pump the same amount of blood, which over many years causes the ventricular walls to stiffen. I hope that clarifies some of it.
Avatar n tn I recently began a training program with a significant higher volume of training than the past 7-8 years. My issue is that when I am at rest, periodically I can feel a significant thumping in my chest. I understand that for my resting heart rate to remain so low I have a high stroke volume. I just had a full medical evaluation from my employer and I have no other medical concerns. I do not take any meds, my BP is 110/70. I am 6ft, 196# with 18% body fat.
Avatar f tn AF is not acutely dangerous but you do want to maintain the aspirin per the doctors instruction to reduce the risk of a stroke for now and ask the doctor about switching to anti-coagulation meds. Because yours is paroxysmal AF, it is hard to correlate it to anything else including strep. For some people, there are triggers such as caffeine, alcohol, hormonal changes, etc ... Also, endurance training could trigger remodeling of heart muscles and some will develop AF from excessive training.
Avatar m tn I suspect that with the lower heart rate, there is more complete filling of the heart chambers or more blood volume per stroke which helps to compensate for the reduced rate. Last July I completed a catheter ablation and have not had any symptoms since the procedure. I have discontinued all meds and returned to my former level of exercise.
Avatar f tn With regular training, your heart becomes stronger and pumps a greater volume of blood with less effort, lowering your heart rate at rest and during exercise. Your muscle cells grow more mitochondria and become more efficient at extracting and using oxygen, decreasing demand during exercise.
Avatar n tn i have no family history of stroke or heart attack, however, i do have family history of diabetes and psychological disease. i've always had a HR that recovered slowly from exertion, but now my resting HR is between 80 and 95. during sleep it falls to 65. my BP is normal to slightly low. i exercise almost every day, though, high-intensity and aerobic exercise. my HR spikes as soon as i start exercising, and can get as high as 190 when i'm wrestling or boxing.
Avatar m tn Because chamber size is the metric for volume of blood pumped with each stroke, you can evaluate the heart's LV functionality by what is called fractional shortening. LV diastolic (end point) minus LV systolic (end) divided by LV diastolic times 100. Reference range is 18 to 42%. Above 30 is considered normal, 26 to 30% represents mild decrease in ejection fraction, below is abnormal.
Avatar n tn I would want to look at all the measurements that can tell you how well your heart is pumping blood like ejection fraction and stroke volume. If you get the all clear from your doc lose the weight. I used to be very althletic but had put on 40 pounds after having kids. All I did was lose the weight, I didn't even work out to lose the weight. But when I did exerciseafter losing the weight my tolerance went right back to normal, it was very noticable.
Avatar m tn -An increase in sympathetic stimulation to the heart increases contractility and heart rate. -An increase in contractility tends to increase stroke volume and thus a secondary increase in preload. -An increase in preload results in an increased force of contraction by -Starling's law of the heart; this does not require a change in contractility. -An increase in afterload will increase contractility (through the Anrep effect).
Avatar m tn Your so-called pulse pressure is also fairly high (difference between systolic and diastolic), it may be caused by a high stroke volume from the heart, or unflexible arteries. Not easy to say, but ask your doctor. If your blood pressure is in the 140-160 range it's too high, so it's a good idea to follow the advice you got from the doctor answering your post..
Avatar m tn 8cm systolic diameter, 22% FS, 117ml diastolic volume, 56.2 ml systolic volume, EF% 52, Stroke Volume 60.8, LVPW thickness .82cm, IVP thickness .88 So, my questions are the following: -Can you have some form of systolic dysfunction without ending up with an enlarged heart or an EF% lower than 50? -Could I have systolic dysfunction that is being controlled by my exercise, which is why I have been able to have an EF% in the low 50’s and no structural remodeling?
Avatar m tn The etiology would be if the carotid bodies sense a decrease in blood pressure they send a message to increase the heart rate and stroke volume of the heart. If the pressure increases, they tell the heart to slow down. This is why a person can press on specific areas of the neck and have a response in their heart. This procedure, called carotid massage, can induce changes in blood pressure and can even cause a person to stop having an arrhythmia, or irregular heart beat.
3197167 tn?1348972206 The tempo, or pace, of the music had the greatest effect on relaxation. Speeds Post-Stroke Recovery A daily portion of one's favorite pop melodies, classical music or jazz can speed recovery from debilitating strokes, according to the latest research.
Avatar n tn Other symptoms of Parkinson's disease include decreased facial expression and voice volume, slowness of movement, changes of gait including slowing down of walking and shuffling, and other symptoms. Shoulder pain is sometimes the first symptom of Parkinson's disease and is due to increased muscle tone (called rigidity) in the shoulder.
Avatar n tn Hi all, I had a stroke when I was 21 years old, It was a small stroke and due to my age and stamina only took me 4 months to recover, since then I have been on no medication. I am now 30 years old and now looking back have begun to realise that the problems I am experiencing could be due to TIA's and side effects of the stroke. I am very "up and down", have (which is the opposite to some) very bad hearing.
Avatar n tn Since than I also had a muga test done which showed no change in my stroke volume at rest and during exercise. Currently I have a echo done every 6 months. My last echo was on 12/10/98 which showed that my heart has grew slightly from the last echo. My cardiologist has just prescribed medication which I think is Vazotec. I am currently very active with both aerobic and weight training activity. I am not experiencing any symptoms at this time. My blood pressure is 130/50.
Avatar n tn 60 or 80 mmHg, the most likely basis is stiffness of the major arteries (as markmsn states), an echo would rule out aortic regurgitation (a leak in the aortic valve) as a cause and that would have been a possibility, arteriovenous malformation (an extra path for blood to travel from a high pressure artery to a low pressure vein without the gradient of a capillary bed), hyperthyroidism or some combination.
Avatar n tn For example, if systemic venous return is suddenly increased (changing from standing to supine position), right ventricular blood input (preload) increases leading to an increase in stroke volume and pulmonary blood flow. Now the left ventricle will experience an increase in oxygenated blood from the lungs, which in turn increases left ventricular preload and stroke volume by the Frank–Starling mechanism.
Avatar f tn You need to have a 24H Holter ECG and a heart volume test (your blood volume could be 5-50% below normal levels.) The doctor must be an LLMD (Lyme Literate Medical Doctor) and knows about pathogenic mycoplasmas. Check Dr. Garth Nicolson's website: He's one of the few doctors anywhere that can treat pathogenic mycoplasma infections! Most Doctors do not have a clue. And your life ends up in Limboland, because of this.
Avatar n tn I was never bothered about heart rate until I read about what the supposed maximum should be for my age, and after using the hrm on the training bike at my gym. I carry out a High Intensity Interval Training routine for about 40 mins which I finish on the bike, and it gets my ticker up to 175-180, there's no pain although I'm a little out of breath (that's the point of HIIT). I can confirm this figure as I have double checked it with a borrowed chest band. My resting pulse is in the low 50's.
214864 tn?1229718839 I realize you do not have my chart with my history, but I have had an mi and stroke simultaneously in 2003, and another stroke in 2005. Hi BP and tachycardia under control. My second question has to do with former ulcers in my stomach and small intestine eight years ago, due to abuse of NSAIDs. Now I cannot tolerate even an enteric coated baby aspirin without very bad "Gut" pain or ulcer pain. So I have been taking Plavix sine June of 2003.
Avatar n tn Thanks for the input Ken. The only conflicating data with stroke volume being, even though I'm athletically conditioned by running, weight training, swimming, boxing etc... I still find that at rest, ie. lying on the couch and having a BPM of 50 and getting up and walking up a flight of stairs, my BPM will reach 120 within 3-4 seconds. I though an increase stroke volume will definatly result in a lower heart rate upon minimal exericse.
Avatar n tn It's the beta blocker. I'm taking 75 mg of metoprolol daily, I was a bike racer for years. I live in Phoenix, it was 113 degrees, my BP gets down to as low as 75/50 after a long ride in the heat. Ever since going on metoprolol, my legs have been like lead, my training response sucks, and when I go to altitude, it's like I've never been on a bike before. But, if I look at my training performance, it's not really so bad, my real performance is only down about 10% or so.
Avatar n tn Is it possible that I was the unknowing author of my own problems with all the heavy exercise? Do I need to cut my exercise volume and intensity now? Is Zestril the best approach? I can't get past the feeling every time I work out that I am shortening my lifespan rather than prolonging it.
Avatar f tn Even if you end up in AF, there is no acute risk of dying although the risk for stroke goes up. I suggest keeping a journal of whether your PACs correlate with what you eat, drink and how you exercise (over training can do it). Make sure you magnesium and potassium levels are in range. Even in the worse case where you do end up with AF years down the road, ablation can cure it if you catch it while it is still paroxysmal, if you find a center that does a high volume.
Avatar n tn ejection fraction 68% shortening 39% left ventrical mass 190 grams with a stroke volume 98ml lv diastolic volume 144ml lv systolic volume 46ml lv 5.4cm (enlarged) lv wall thickness 1.0cm left atrium 3.8cm estimated pulmonary artery pressure slightly elevated mild aortic insufficiency mild to moderate tricuspid regurgitation right ventrical slightly enlarged but no value given Can you please explain these findings to me and how serious are they?
Avatar dr m tn Untreated obstructive sleep apnea can lead to chronic fatigue, depression, anxiety, weight gain, high blood pressure, heart disease, heart attack and stroke. ____________________________________________ Steven Y. Park, M.D., author of Sleep, Interrupted: A physician reveals the #1 reason why so many of us are sick and tired. Endorsed by New York Times best-selling authors Dr. Christiane Northrup, Dr. Dean Ornish, Dr. Mark Liponis, and Mary Shomon. www.doctorstevenpark.
489725 tn?1280056153 Education should include the latest medical recommendations and guidelines and the rationale for them (i.e. meds or ablation) for the patient's situation, and some training regarding possible modes of decision making. I know this is a lot to expect, but these decisions we make are of great significance and need to be weighed very carefully.
Avatar n tn At that time I had a echocardiogram which showed that my heart was slightly enlarged. Since than I also had a muga test done which showed no change in my stroke volume at rest and during exercise. Currently I have a echo done every 6 months. My last echo was on 12/10/98 which showed that my heart has grew slightly from the last echo. My cardiologist has just prescribed medication which I think is Vazotec. I am currently very active with both aerobic and weight training activity.
572651 tn?1531002957 Spasticity is caused by upper motor neuron lesions and isn’t exclusive to MS - spasticity is also seen in stroke patients, head injuries, and cerebral palsy. I really liked his explanation about spasticity that it is similar to our cars. We push the accelerator to go and the brake pedal to stop. If we press both we get the equivalent of what our muscles are doing with the spasticity. Spasticity needs to be treated only if it impacts daily activities.