Stroke volume women
Common Questions and Answers about Stroke volume women
stroke
5 % reference 54-74(LV end-diastolic volume/bsa (H) value 148 ml/m\s\2 reference 29-61(LV end-systolic volume/bsa (H) value 122ml/m\s2 reference 8-24(LV ejection fraction, 1-p A4C (L) value 16.3% reference 46-78(Stroke volume, 1-p A4C value 30 ml (Stroke volume/bsa, 1-pA4C value 16.1 ml/m/s\2(LV end-diastolic volume 2-p (h) value 189 ml reference 46-106(LV end systolic volume 2-p (H) value 158 ml reference 14-42(LV ejection fraction 2-p (L) value 16.
CO is a product of blood volume per heart stroke and heart rate. The higher/better the stroke volume (weak, strong, stronger contractions) the fewer heart beats (more for weak, less for strong, least for stronger) are required to meet demand (held constant for illustration) for blood oxygen. A well-condition individual requires fewer heartbeats due to strong contractions. It is the stroke that determines EF...not the CO, as CO can vary by an increase or decrease of HR.
MPV reflects the average volume of your platelets. A high MPV is associated with a high risk of heart attack and stroke. The normal range is 7.5 to 11.5. MPV will vary with platelet production; younger platelets are larger than older ones.
I don't know why you were having these test done. Your history and symptoms are a major deciding factor as to whether or not your values are an indicator of illness. There are a multitude of ways to interpret your results.
This happens by a system of vasodilation and vasoconstriction coupled with the pumping rate and stroke volume of the heart. Rather than make frequent changes in volume, the body changes the configuration of the total container (all the vessels) and increases the speed at which the resource is renewed.
It's been 2yrs since my stroke and I have no voluntary movement in my left arm or hand. Is there any possibilty that i could recover any movement? My neurlogist told me that if within 6month of the stroke if I didnt have any movement that my hand will stay paralyzed?
A faster heartbeat does not provide enough time to fill as at rest, and the timing of contractions are less. The percent of blood pumped with each heartbeat is less reducing stroke volume (lower EF). The heart rate has increased to provide the necessary blood/oxygen to meet the system's demand for the diminished stroke volume.
The CT scan indicates there is substanital soft plaque buildup within the linings of the vessels.
The volume of blood left in a ventricle at the end of contraction is end-systolic volume. The difference between end-diastolic and end-systolic volumes is the stroke volume, the volume of blood ejected with each beat. Ejection fraction (Ef) is the fraction of the end-diastolic volume that is ejected with each beat; it is stroke volume divided by end-diastolic volume. Normal in a man is 50-68%.
For a perspective, diastole dysfunction compromises left ventricle filling volume, inadequate stroke volume and operating at a high pressure regimen. LV filling volume is reduced due to increased wall size crowding out available space. Inadequate stroke volume is usually due to wall thickening and stiffening and loss of elasticity. High pressures can dilate upper chamber to compensate for gradient pressure across the mitral valve. That is the pathology.
My niece had an MRI recently and one of the findings said volume loss of the posterior fossa. What does that mean exactly? She has speech, vision, hearing and balance problems that we have never really had explained as to why she has them? Are they all releated to what they found on the MRI?
what happens with significant MVR some blood fails to pumped into circulation with each stroke. To maintain adequate blood flow with a decrease in stroke volume is to increase the heart rate. The math for cardiac output is stroke volume times heart rate for one minute.
Frankly, I don't believe you have significant MVR because you should also have the symptoms of shortness of breath with exertion and muscle fatigue.
An echo calculates heart chamber diminsions and volume of blood pumped with each stroke. The math equation is diastole volume (peak filling) minus systole (volume after pumping) divided by diastole for the percentage.
You can calculate your fraction shortening (FS) which is similar to EF but relies on dimensions rather than volume. To calculate subtract systole dimension from diastole dimension and divide by diastole for percentage. FS is an estimate of myocardial contractility.
Hey, i just got this off some Dr's site
( http://www.netwellness.org/question.cfm/35267.htm )
The cause of white matters signal abnormality could be caused by:
Aging and risk factors such as hypertension and diabetes. Sleep apnea, with the episodes of hypoxia, is another cause of these findings. They are not exactly "normal" findings, as they are associated with increased risk for stroke and cognitive decline in the long term, but are not always indicative of a stroke.
CO (volume of blood pumped with each heartbeat) there is a variation of volume of blood pumped with each stroke to maintain a balance between left and right side blood flow) and abviously PAP will vary accordingly (stronger heartbeat,higher PAP.
If there are blockages (clots) in the lungs or pulmonary artery (increase of resistance the heart pumps against, and/or if there is narrowing, stiff pulmonary vessel the more resistance (increase of resistance will cause higher PAP).
If you are consistently clinically dehydrated (really, really dehydrated, like you just crawled in from the desert where you were dying of thirst), it might theoretically reduce your semen volume, just the way it would reduce your blood or tears.
If you're asking because you know for a fact (because you measured) that you used to produce more volume, and now you have measured again and aren't producing as much, for that question see your doctor.
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