Stroke volume cardiac output

Common Questions and Answers about Stroke volume cardiac output


1591333 tn?1297733419 The left venticle will dilate to increase blood flow with each stroke and the heart rate will increase the cardiac output (amount of blood pumped per minute). If the system is dysfunctional, medication, etc. can change the dynamics. For instance heart failure increases blood volume and the kidneys react as if the individual is losing blood...the increase of volume further distresses the heart and kidneys continue to believe the system is losing blood.
Avatar n tn The difference of normal cardiac output has a variation of about 34%. Therefore. just based on cardiac output variation the PAP can vary by 34%. I have read your doctor's report (what you provide) to you, and there is not any opinion that is not in keeping with a good analysis and evaluation. I think you may be reading information that is confusing, however, if you don't trust your doctor's evaluation then see another doctor.
Avatar m tn The contribution of the heart to the regulation of blood pressure is through changes in cardiac output. Cardiac output is defined as the product of heart rate and stroke volume. Either can be modified by climatic forces. The heart rate component is regulated by the autonomic nervous system, so its level of activity depends on the intensity of the stimulus at the time.
Avatar n tn I believe there was some discussion on a prior thread regarding stroke volume at its significance. Stroke volume and the significance is monitored by endthelium cells (lines the vessels). When there is a significant drop in CO the vessel enthelium cells sends a message to increase the blood volume (recognized and not able to distinquish if there is a serious blood loss). The system controls stroke volume by venus return. Stroke volume AND heart rate determine CO (cardiac output).
Avatar m tn __________________________________________________________________ ans. Cardiac output (CO) is stroke volume times HR/min. (4/08...39 ml X 85 + 3.33 l/min) (8/08...56 X 92 + 5.2). Not relevant, but what is relevant is stroke volume. The dilated LV for August is producing stronger contractions as it should do.
Avatar m tn For instance, how are systemic vascular resistance, blood volume, stroke volume, cardiac output, etc affected. I am wondering if an HOCM heart relies on a hyperdynamic state from hyperthyroidism, and if going euthyroid will harm the cat.
Avatar n tn Return of systemic blood to the heart is impeded by the pressure inside the chest. The output of the heart is reduced and stroke volume falls. This occurs from 5 to about 14 seconds in the illustration. The fall in stroke volume reflexively causes blood vessels to constrict with some rise in pressure (15 to 20 seconds). This compensation can be quite marked with pressure returning to near or even above normal, but the cardiac output and blood flow to the body remains low.
487969 tn?1249313291 My blood pressure is just like yours! I used to be ~120/88 or something, but now I'm around 98/65. I thought this was significant--or at least a clue--in figuring out what was wrong with me since this was a significant drop in pressure that has remained constant. But of course, the doctors don't seem to care.
Avatar m tn The heart dilates and this is early compensation for systolic dysfunction and decreased cardiac output is accomplished by increasing the stroke volume, the heart rate, or both (cardiac output = stroke volume X heart rate), which is also accompanied by an increase in peripheral tone (dilation). If the underlying cause for compensation is not treated there is overcompensation and the left ventricle dilates to the degree that impairs contractions.
Avatar f tn It is easy to explain high heart rate and high blood pressure, because the two normally compensate each other to maintain a balance of blood flow between the right and left side of the heart. Lower cardiac output (stroke times heart rate) can/will constrict blood vessels and blood pressure with rise, and that will cause an increase in heart rate rate, etc. and blood volume.
Avatar f tn To maintain adequate blood flow with a decrease in stroke volume is to increase the heart rate. The math for cardiac output is stroke volume times heart rate for one minute. Frankly, I don't believe you have significant MVR because you should also have the symptoms of shortness of breath with exertion and muscle fatigue.
Avatar m tn Either condition will reduce the volume output so output is the correct parameter to estimate the EF. Also, 02 does not directly relate to the EF percentage . However, indirectily low 02 can cause hypoxia and that condition can impair heart wall movement and reduce EF. "Pulse rate and EF": Blood flow dynamics that includes PS and EF are compensatory mechanisms that counter balance each other normally.
1527234 tn?1291771442 In this phase due to F/S phenomonon the ventricular myocardium is no longer able to contract adequately to compensate for the volume overload of mitral regurgitation (volume overload can be do to other causes), and the stroke volume of the left ventricle will decrease. The decreased stroke volume causes a decreased forward cardiac output and an increase in the end-systolic volume dilates left ventricle.
2015762 tn?1329255120 Mathematically, the cardiac output can be calculated by multiplying the heart rate times the stroke volume. In anemia, the cardiac output increases, and that allows more hemoglobin to be exposed to the peripheral tissues, making up for the decreased hemoglobin concentration. Accordingly, the heart rate increases, which gives us one of the cardinal clinical manifestations of anemia, tachycardia, or fast heart rate. The heart does not act alone to increase the cardiac output.
Avatar m tn I have found, read and experienced if one is decompensating/compensating due to a heart problem, the heart rate is almost always above 100 at rest. Cardiac output is stroke volume for one minute. To maintain the same CO to meet the same demand the heart beats faster. My HR was consitently around 115 before going to ER for CHF.
Avatar m tn A stronger heartbeat should/would increase the cardiac output and require fewer heartbeats given the same demand. It can be seen in an equation where cardiac output is stroke (amt of blood pumped with each heartbeat) times the number heartbeats in a 60 second interval...increased stroke fewer heartbeats, a decrease in stroke (weak heart) more heartbeats to meet the same demand.
Avatar m tn In severe cases, the ventricle becomes so stiff that the atrial muscle fails and end-diastolic volume cannot be normalized with elevated filling pressure. This process reduces stroke volume and cardiac output, causing effort intolerance. Figure 117 summarizes the pathophysiology of diastolic heart failure.
1002911 tn?1251595843 If your heart is pumping greater than 100 bpm at rest, and your extremities are cold, it could indicate a low cardiac output. Cardiac output is blood volume pumped with each heartbeat for one minute. When there is a shortage of blood in circulation, circulation is reduced by constricting vessels to the extremities to provide sufficient blood to vital organs. Also, the heart rate increases to the cardiac output.
Avatar m tn Moderate regurgitation can be a problem for some individuals especially if the cardiac output is compromised by DD. There is a reduced cardiac output due to DD and regurgitation further reduces the CO as some of the blood back flows into the upper chamber rather go into circulation. When there is a serious problem with cardia output there should/will be shoertness of breath. My doctor has told me to let him know if I shortness of breath.
1459433 tn?1285693332 Is there anything they can do to cut those risks? Does low cardiac output cause headaches? We already know she has low cardiac output which causes dilated capillaries in her face(the headaches started about the same time as the bright red face). Any info would be helpful as we are not scheduled to see the card or neuro for a couple of months.
Avatar f tn My health history has alway been proper weight, almost normal blood pressure (slightly high systole (if I had "stiff" arteries, it would show a high systole abnormally), cholesterol below 149, etc. Don't judge by age! My parents had a long healthy life without ever being on any medication until the final days.. I took good health for granted at a very young age. Smoked, binge drinking. improper diet, all night parties, etc.
653803 tn?1224248552 I will increase contractility and give relief to heart with more efficient cardiac output. An EF below 29% is considered heart failure mode as the cardiac output usually isn't enough to meet the system's demand for lood/oxygen. When the EF drops into the heart failure range, the cardiac output is less than the blood supply coming from the lungs and blood/oxygen backs up into the lungs causing congested heart failure (condition where fluids leak into the lung tissues (edema).
Avatar f tn The difference would be reflected in the cardiac output. CO is stroke volume time heartbeat for one minute. Obviouisly a skipped beat (s) would significantly reduce the CO, but compensation as you suggest would not be arrhthmia, it would be reduced regular heart rate.. Nevertheless, it is an irregular beat, and the underlying cause should be diagnosed. Your doctor is aware and no concern so it may not be anything serious...just annoying.
Avatar n tn As a consequence, the rapidly contracting ventricles do not have enough time to fill maximally with blood before the each contraction, and the amount of blood that is pumped decreases in spite of the faster heart rate...low cardiac output (CO is heartrate for one minute times stroke volume)....low CO causes low blood pressure. Hope this provides a perspective, and you may want to consult your doctor for an evaluation.