If you responded to a person who is sick at home and that patient has not been taking their K-Dur medicine for a few days and you find your CHF patient, who had been prescribed Labetalol, Lasix and K-dur, and showed monomorphic V-tach on the monitor, would you synchonize cardiovert the patient first if he was hemodynamically unstable knowing that he is low on potassium which might convert your patient into unwanted and nonperfusing rhythm? Would we harm the patient if we shock them?
When a person is in v-tach and the v-tach degenerates into v-fib, is the transition abrupt (like the transition from normal sinus rhythm to v-tach) or is it gradual? By gradual, I mean the v-tach becomes progressively finer and faster until eventually it can no longer be defined as v-tach.
I have looked for these stats everywhere but I can't find much of value.
I was recently diagnosed with a running v-tach. I had been to the emergency room 3 times within two weeks because of my symptoms, which are dizzyness, blurred vision, fainting or passing out, sometimes I have tingling in my face or hands. I was admitted to the hospital the first time, they did some tests. Stress test, CT scan, some blood work. They said I was fine.
But having a wide complex does not mean that you necessarily have V-tach, even if your wide complex appears regular. Even if you do have V-tach, it does not mean you are necessarily in danger.
If you have wide complex, an electrophysiologist has to look at your ECG to interpret whether you might have v-tach or something else. A doctor also need to help you decide how to handle this.
Basically,
If you start having multiple PvC's in a row, it is called Ventricular Tachycardia. Now some people have V-Tach for a few seconds are maybe a little longer and it goes away and you go back to sinus rhythm. However, if your heart does not go back into normal rhythm, V-Tach can lead to the deadly ventricular rhythm called Ventricular Fibrillation. If you do not receive immediate medical attention (CPR, De fibrillation) this rhythm is always fatal.
In the future I will be a bit faster on atropine, sedation and pacing as necessary. My concern with giving atropine before controlling the PVCs was that it might instigate V-tach from this obviously irritable focus, but your explaniaion makes sense. The 12-leads were all non-diagnostic for STEMI or NSTEMI, though this did happen after the pt was doing lots of yard work, so ischemia and ACS were definitely on my list of concerns. Thank you again for your response.
Once when I got tooth anesthetic, I began trembling. I don't know what it was, but Im know that I don't tolerate epinephrine or adrenaline. Since that time I always ask such medication which does not cause trembling or tachycardia.
My understanding is that once a heart is damaged, the heart is more prone to v-tach and v-fib. In my Dad's case, after 4 heart attacks his heart was so damaged he developed cardiomyopathy and needed an ICD. That would zap his heart back to normal rhythm when it went out of control.
Not sure how to stop v-fib. I think v-tach can be controlled to some degree with the proper medications and a pacemaker or ICD.
When a person is in v-tach, is the pulse rate high just before or just after the episode. The reason I ask is that yesterday I felt a fluttering in my chest and I started to feel a major head rush , kind of like a light headed feeling and when I went to take my pulse I could not find it for at least 3 seconds and then it kicked back in and my heart was beating normally but it was not fast. Heart rate was about 85 and within a few minutes was down to 72.
The later ICDs are more sophisticated and do a better of of identifying V-tach correctly and attempt to pace me out of V-tach before shocking. In the course of replacing tone ICD, I got an infection and the ICD and leads had to be removed and placed on the right side. Through all of this, I continued to exercise, but gave up competitive racing.
In the last couple years, the V-tach returned about 1 or 2 times per month.
Im a 24yr old male, Im going tomorrow morning for an EP study and possible ablation for my v-tach episode, a 27 beat run at 127bpm. non sustained. Im sooooooooooo scared of complications, does anyone know the death rate during this test? or any other complications? if they induce v-fib whats the chances they can get you back into normal rythm? is this a VERY safe procedure that they want to do for my v-tach? please HELP!!!!
could an EP study and ablation make my v-tach worse or cause it to become dangerous later on in life? 2.Also how safe are EP studies? 3.Is ablation safe? 4.Whats the mortality rates for this procedure? 5.Should I get the EP study done or can this be benign? 6.Whats the chances of V-Fib and SCD during EP study and ablation? 7.How bad is it to have a 26 beat run of v-tach??
I am 36 yrs. old. I have three normal echos on file before my last c-section (two were taken during the last trimester of my last pregnancy & one during the second trimester of the last pregnancy). DURING THE LAST C-SECTION SURGERY, the anaesthesiologist said I had an episode of v-tach straight out of an episode of House.
so far SVT is not fatal but a consisted SVT for long periods of time can be harmful and produce complications. fatal Arrthymias known as V-tach ventricular tachycardia which basically means that the lower ventricle chambers are rapidly contracting and not letting enough blood flow through the heart lungs and brain this is the cause of being in shock and losing 100% blood pressure which than causes the heart to go in V-fib or induced cardiomyopathy .
t be good,lol. Dr. said that I was having runs of V-tach everyday and he wanted to expedite the ICD plans. I have been hopeful that aggressive med diet would increase EF. He humored me and said to have MUGA on wed and if Ef is under 35, no if's about it. So....
Oh, back to your question, if this helps. When I spoke to his office to set up MUGA, his assistant said that if company (cardio-net) contacted me, they probably had my # down as contact, rather than Dr's office.
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