My dentist gives me my shot and see another patient, by the time he got back to me I was fully numb. and the numbness last for 8 - 12 hours. I am highly allergic to lidocaine it puts me into anaphylactic shock, I have an epipen but sa that I don't have to use that I just make sure everyone is doing their job and I always wear my medic alert bracelet.
My understanding is that once a heart is damaged, the heart is more prone to v-tach and v-fib. In my Dad's case, after 4 heart attacks his heart was so damaged he developed cardiomyopathy and needed an ICD. That would zap his heart back to normal rhythm when it went out of control.
Not sure how to stop v-fib. I think v-tach can be controlled to some degree with the proper medications and a pacemaker or ICD.
There is no relationship between afib and v-fib. A-fib is a very common rhythm disorder that occurs sometimes in people with healthy hearts. The abnormal rhythm is initiated in the atrium. V-fib is a ventricular rhythm disorder, initiated in a different part of your heart. Atrial arrhythmias are not going to cause you to drop dead and are not considered as serious as the ventricular ones. Sounds like you have had all the tests to rule out a serious issue. Need to try to stop worrying.
Then, within 3-4 minutes of this very rapid, but regular heartbeating (SVT), it converted to this crazy, irregular rhythm. It was a distinct change (this was the A-fib). Called 911! For the next 2-3 hours, I stayed in A-fib/SVT until I was cardioverted by Cardizem injections.
I have a long history of panic disorder(genetically stacked!), and have struggled with all of this since the birth of my first child in 1994. You have my complete sympathies.
So, my verdict on Sotalol is positive and I would definitely give it a try if your cardiologist is recommending it. As for the risk of dangerous V-fib, my understanding is that the risk is low with this drug but, because of it, Sotalol is usually started in the hospital, at least in the US. Patients are monitored for any dangerous heart rhythms for 3 days. For some reason, this isn't done in Canada where I live. I was simply given the prescription and sent home to start the drug.
Hi. I'm 24 male dip a can of tobacco a day. I know. I have an anxiety and panic disorder. I think I have been getting these pvcs. It feels like my heart skips or stops for a minute. Then back to normal or heart rate may rise a little. During the skipped heart beats if lying on my left side I get a regular beat the pause then regular beat pause. I usually only get them once a day. Couple times a week. My blood pressure raises a little during pvcs plus I can feel it.
Stroke CAN kill. V-fib without prompt treatment WILL kill. Within 45 seconds after you go into v-fib - i.e. your heart stops pumping - you WILL lose consciousness. Within 3 minutes of onset of v-fib without treatment (CPR or cardioversion) your brain starts to die. Stroke CAN kill but absent VERY PROMPT treatment v-fib WILL INVARIABLY KILL YOU. Hence medication for rate control.
It showed short runs of a-fib and v-tach, but very inconsistently, but surprising and a bit worrisome. I have a 15yr history of arrhythmia and also have chd and have had strokes because of the chd. I am currently on aspirin as well as other meds including a new prescription for multaq, but does the a-fib warrant coumadin? I just read about a-fib and I don't want to have another stroke.
When I had my ablation in January 08 for a-fib and flutter I was admitted to hospital on Tuesday morning, the procedure was Wednesday morning and then I was released Friday evening. I have read from other people who have had ablations for a-fib at different hospital or EP that were admitted the same day as their procedure and only stayed overnight. I did have to stay until my coumadin levels were normal.
Okay, well this turned out to be a pretty information-packed, long response, but I believe it can do a lot of good for you in understanding your situation if you go ahead and read it. I would also like to warn you that I am not a doctor. I am just a student, who happens to have a particular area of interest in the heart. Maybe someday I'll be a cardiologist or something, but I'm not one now, so take everything that I say with the knowledge that I am not a licensed doctor or anything.
s and other weird heartbeats that waken me make me more likely for v-fib?These episodes happen most that waken me from sleep,and early morning hours,especially after night of drinking, but also without drinking.when they happen I feel LONG pauses between heartbeats,and slow pulse with random spurts of tachy on and off.5.what could that be?
http://www.ncbi.nlm.nih.
Plus, in the absence of a good signal from the atria you can easily go into ventricular tachycardia (v-tach) which can escellate into ventricular fibrillation (v-fib). FYI, absent cardioversion by a defibrillator v-fib is pretty much 100% lethal. When the ventricals are in fib they aren't pumping. That tends to be lethal. I suspect that you're being given atenolol to control your heartrate to keep you out of v-tach. Again, I got used to it pretty quickly.
The second event, which is less common (although it becomes more common as we age into later adulthood), is what occurs if you have atrial fibrillation (a-fib) or atrial flutter, which are also types of SVT. A-fib and atrial flutter are very fast arrhythmias of that occur in the atria, in which the atrial rate can go 200 to 400, or more, beats/minute. Our A-V node can’t conduct that quickly, so our ventricles don’t beat much over 200 to 220 beats/minute.
Without a shock from an ICD or paddles, you would be dead within minutes if you had v-fib. V-fib is the deadliest arrhythmia you can have short of total asystole (which at that point, can it really be called arrhythmia? Is music without sound still music?), and is just about 100% fatal without quick medical intervention.
And Jerry's right, a-fib in and of itself is not life threatening. V-fib is life ending. Google ventricular fibrillation for yourself.
When a person is in v-tach and the v-tach degenerates into v-fib, is the transition abrupt (like the transition from normal sinus rhythm to v-tach) or is it gradual? By gradual, I mean the v-tach becomes progressively finer and faster until eventually it can no longer be defined as v-tach.
I have looked for these stats everywhere but I can't find much of value.
t prevent me from going into a V-Tach or even V-Fib (which is my main concern). I saw the EP doc as well and he was less than reassuring, stating that the PACs could lead to A-fib, but that I should just 'go back to cardio if I felt like I was having more PACs"?
Is there a reason why I'd have these changes (in echo & now w/ PACs) in such a short amount of time? Is it dangerous to have these changes - will they progress to V-fib or A-fib?
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