If you responded to a person who is sick at home and that patient has not been taking their K-Dur medicine for a few days and you find your CHF patient, who had been prescribed Labetalol, Lasix and K-dur, and showed monomorphic V-tach on the monitor, would you synchonize cardiovert the patient first if he was hemodynamically unstable knowing that he is low on potassium which might convert your patient into unwanted and nonperfusing rhythm? Would we harm the patient if we shock them?
When a person is in v-tach and the v-tach degenerates into v-fib, is the transition abrupt (like the transition from normal sinus rhythm to v-tach) or is it gradual? By gradual, I mean the v-tach becomes progressively finer and faster until eventually it can no longer be defined as v-tach.
I have looked for these stats everywhere but I can't find much of value.
I was recently diagnosed with a running v-tach. I had been to the emergency room 3 times within two weeks because of my symptoms, which are dizzyness, blurred vision, fainting or passing out, sometimes I have tingling in my face or hands. I was admitted to the hospital the first time, they did some tests. Stress test, CT scan, some blood work. They said I was fine.
Could this cause v-fibb and SCD in my situation? 2. What could have caused this? 3. Is it possible for v-tach to be benign or is it always deadly? why haven't this ever been caught with the multiple of test that i had, holters and event monitors?? How dangerous is this? My pulse is always jumping around up and down.
Is it possible for v-tach to be benign or is it always deadly? why haven't this ever been caught with the multiple of test that i had, holters and event monitors??
t think that it was V-Tach or at least i never asked. The only suggestion that i would have in trying to find a strip is to contact other EMS rigs and ask them if they may have one. I think everyone is going to be a little leary other then community members to share much because of HIPPA but it couldn't hurt to try or to maybe contact an EP doc to see if they may have one...are you doing a comparison for a class you are instructing?
it and chose to have an ablation done for v tach and a couple of other things and the ablation is in alot of cases successful in the 80% range. And no it isn't always deadly and like i said it can be treated. I personally felt like i was sitting on a time bomb just like it sounds like you are feeling and decided to get pro active and do something about it because i didn't want to live my life feeling jumpy every single time i could feel my heart for one reason or another.
But having a wide complex does not mean that you necessarily have V-tach, even if your wide complex appears regular. Even if you do have V-tach, it does not mean you are necessarily in danger.
If you have wide complex, an electrophysiologist has to look at your ECG to interpret whether you might have v-tach or something else. A doctor also need to help you decide how to handle this.
You hit it on the head - just because you have seen a doctor deosn't mean a thing. I was "diagnosed" with anxiety by my PC several years ago, but he never mentioned a monitor and I didn't know any better (I was in my mid 20's). I just lived with it, didn't take any meds or anything. My "anxiety" subsided until about three years ago, but when it came back about three years ago I figured anxiety again.
Basically,
If you start having multiple PvC's in a row, it is called Ventricular Tachycardia. Now some people have V-Tach for a few seconds are maybe a little longer and it goes away and you go back to sinus rhythm. However, if your heart does not go back into normal rhythm, V-Tach can lead to the deadly ventricular rhythm called Ventricular Fibrillation. If you do not receive immediate medical attention (CPR, De fibrillation) this rhythm is always fatal.
In the future I will be a bit faster on atropine, sedation and pacing as necessary. My concern with giving atropine before controlling the PVCs was that it might instigate V-tach from this obviously irritable focus, but your explaniaion makes sense. The 12-leads were all non-diagnostic for STEMI or NSTEMI, though this did happen after the pt was doing lots of yard work, so ischemia and ACS were definitely on my list of concerns. Thank you again for your response.
I have had bad reactions to regular novocaine in the past, prior to my SVT diagnosis in 2001, so he has given me lidocaine since then. For the past ten years prior to my ablation in April 2011, I was on beta blockers (atenolol) so there were no issues. I was taken off beta blockers 3 weeks after my ablation. Have had dental work requiring lidocaine since then, with no issues until now.
While he was injecting the second shot of novocaine, an SVT episode suddenly triggered.
My understanding is that once a heart is damaged, the heart is more prone to v-tach and v-fib. In my Dad's case, after 4 heart attacks his heart was so damaged he developed cardiomyopathy and needed an ICD. That would zap his heart back to normal rhythm when it went out of control.
Not sure how to stop v-fib. I think v-tach can be controlled to some degree with the proper medications and a pacemaker or ICD.
You may be having a ventricular response to the A-Flutter, Like supraventricular tachycardia. My device will pick up SVT and V-tach or V-fib only. you may need to have some adjustments in your settings or maybe give the beta-blockers time to take effect. It sure isn`t fun being shocked hope your doctors can figure it out.
You are the first person I have seen who has had an ICD longer than me, my first implant was in 1991. BEST OF LUCK TO YOU.
When a person is in v-tach, is the pulse rate high just before or just after the episode. The reason I ask is that yesterday I felt a fluttering in my chest and I started to feel a major head rush , kind of like a light headed feeling and when I went to take my pulse I could not find it for at least 3 seconds and then it kicked back in and my heart was beating normally but it was not fast. Heart rate was about 85 and within a few minutes was down to 72.
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