At day 22, i took raltegravir from the box but im not sure whether i drank the pill with the water or not. Normally, I remember the pills that I drank through glass of water but i cannot remember this time whether i drank this pill with glass of water or not but i counted pills and i concluded that their numbers are as expected.
I searched couple of papers and i concluded not to double the dose in the case of i used that pill.
combining emtricitabine and tenofovir from the NRTI class, and raltegravir, from the integrase inhibitor class. There ARE alternative drugs available and possibly in Thailand they use lamivudine in place of raltegravir. I am definitely NOT a chemist, so I can't tell you for sure if the combination you're taking is appropriate. You should discuss your concerns with the doctor who prescribed PEP or a pharmacist/chemist. I wish you the best.
I started PEP 2 hrs later (truvada, norvir, raltegravir, and preszita), my initial results are negative. I do not know the viral load of the patient yet, i can only pray that it is low. I know odds are 0.3% but that doesnt settle my fears, i am young and want to live a happy healthy life with KIDS! DR. please tell me if you have honstly heard of any cases like mine that have converted. I am in awe and feel like my life is totally over and ruined.
I am currently taking truvada and isentress (raltegravir) is this enough I couldn't get my prescription for dolutegravir filled...is this pep enough?
It was not a problem. You can get a special insulated transporter from Roche - call the Pegasist number and request one. No problem with security but have a copy of your prescription just in case. On the plane, I asked the flight attendants for ice several times and added to the transporter to keep it cool. I kept all my other meds on my carry on bag as well. Smart to get info on any Hep C organizations where you are going just in case and to put your mind at ease.
m confused on the prescriptions given to me I have a 7 day dose of raltegravir and dolutegravir...thought it was supposed to be 28 days?
I appreciate your blunt response. I was prescribed Truvada + raltegravir (PEP) but could not obtain raltegravir in Europe. To be fair, I called the US PEP hotline who told me that I could take Truvada as PEP as there are no studies that prove it’s ineffective, so I didn’t start it without advice from the US HIV experts. I agree my anxiety is an issue but I legitimately was seeking an answer.
Paxiled, your information is almost quite good up until a point. Amitriptyline is indeed a tricyclic 'anti depressant', but it also has many other effects and uses too. The anti-depressant properties comes from its abilities to inhibit the reuptake of serotonin, (making it a serotonin reuptake inhibitor), but it also inhibits the reuptake of norepinephrine, which - whilst not fully understood - helps to regulate nerve pain from misfiring, or damaged nerves.
Anyways I rushed to the hospital and was put on PEP at a maximum of 3 hours post exposure (truvada and raltegravir). I have taken the pills without missing any doses. At 3 weeks post exposure the lymph nodes in my neck swelled up slightly and I felt a tiny bit feverish. Checking my temperature rectally though revealed it was always in the 37s. These strange symptoms lasted only 2 days...could this be ARS?
I actually went to the clinic, got PEP (truvada 200mg and Raltegravir 400mg), also done hiv prick test which was negative. Dr was helpful and explained everything for me. He also said best thing is as u guys suggest, ask him test but also if he is clear 6 weeks back also, but that also depends on trust.
On my side i don't wanna embarrass him as twice he told me not to worry.
In the following day, I finally could find a very good hospital with a specialized area for HIV treatment, and the doctor was quite surprised that they had given me just a Truvada. She added a Isentress (raltegravir) to my Pep treatment. But I could realized that she was quite worried because I had had only Truvada in the begining of my treatment and in the very deadline edge.
I am taking Truvada and Raltegravir. Yesterday, (3 days after exposure and 2 days on PEP) I had protected sex with a female partner (I, the insertive male) and when I ejaculated I am afraid some spilled off the base of the condom.
If I began PEP 24 hours after possible exposure and had protected sex with someone else on Day 2 of PEP, could I have biologically put them at risk for HIV if by any chance the ejaculate spilled off the condom?
So do let me fill you in on its fascinating history, and explain why it's such an important part of the Soothanol X2 formula. DMSO is short for dimethyl sulfoxide -- an organic, sulfur-rich substance found in the woody part of trees. It was first discovered by a Russian researcher back in 1866. But the real excitement started about 100 years later, when American scientists started uncovering some truly startling properties...
First, they found it's absorbed with incredible ease....
We have both been trained in STD/HIV prevention, but we obviously let one time slip. He works as a transporter at a local hospital, and he told me he was tested and HIV- as of two weeks prior to us hooking up. I was tested using the 30-min blood test on February 21st, 2008, allowing myself 8 weeks. The result was negative.
My question is this: In my research I have found that 2 months is, for all intents and purposes, sufficient time for the antibodies to appear.
I started PEP (Truvada and ISENTRESS which is Raltegravir Potassium Tablets) in 47 hours. I also saw some case about PEP with blood transfusion. A 12 years old girl had a blood transfusion with hiv blood, she took PEP( tenofovir, emtricitabine, ritonavir-boosted darunavir (subsequently changed to lopinavir) and raltegravir) after 24 hours and had these PEP for three months. This girl also has cssr5 dna which is a dna that hard to infect hiv.
Wow sorry to hear tht hey hw cn thy do ths to u
I started PEP 2 hrs later (truvada, norvir, raltegravir, and preszita), my initial results are negative. I do not know the viral load of the patient yet, i can only pray that it is low. I know odds are 0.3% but that doesnt settle my fears, i am young and want to live a happy healthy life with KIDS! DR. please tell me if you have honstly heard of any cases like mine that have converted. I am in awe and feel like my life is totally over and ruined.
//www.fda.gov/ForConsumers/ByAudience/ForPatientAdvocates/ucm309863.htm
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Here is another:
The Effect of Telaprevir on the Pharmacokinetics of Buprenorphine in Volunteers on Stable Buprenorphine/Naloxone Maintenance Therapy
Co-administration of telaprevir did not increase withdrawal symptom frequency and there were no serious adverse events reported during or after completion of telaprevir co-administration.
I presume you had anal sex, because oral is not a risk.
HIV is only diagnosed from a test so all your details and questions have no answers.
Since you have passed 28 days, you can just take a 4th gen test for a conclusive result and all the details you mention which are just question marks will become irrelevant.
However, after 2013, three-drug regimen consisting of a nucleoside reverse-transcriptase inhibitor (NTRI), (tenofovir plus emtricitabine), along with a third drug, either an integrase strand transfer inhibitor, raltegravir or a boosted protease inhibitor, darunavir/ritonavir is recommended for PEP.
Ideally, NTRI is an important component of the regimen because if HIV infects a cell, it first attaches to and fuses with the host cell.
my dr is a electro physcist for cardiac prob and wants me to have an ablation done, i work in our hospital and work closely with most areas as a transporter... i trust these people but i am scared to death of the ablation. any comments good or bad will help me make my decision next week. my psvt has been around for 13 years off and on.
To date, CPI-431-32 in vitro completely suppresses HBsAg, suppresses HBeAg, and suppresses intracellular HBV DNA by greater than 90%, while also inhibiting viral uptake by blocking NTCP transporter activity. CPI-431-32 also reduces HBV DNA in the HBV1.3 transgenic mouse model. In contrast to other cyclophilin inhibitors, CPI-431-32 demonstrated a wide selective index (“SI”) as measured by the toxicity:efficacy ratio (CC50:IC50), which is sufficient for clinical development in HBV.
A similar mechanism is envisioned for myrcludex, except that in this case the binding of the pres1 arms to the bile acid transporter is blocked.
Thus it can be reasonably expected that these two independent mechanisms to prevent core uptake into the cytosol will work synergistically and could greatly enhance each others efficiency.
My primary care doc also prescribed Raltegravir. It was added three days in. Does it matter that I started it later? Or was the Trevada enough for the 72-hour window?
Also, he said that I'll get tested 6 weeks after my baseline test, which would be two weeks after I finish PEP. Is that too soon?
I read some information that now they believe that there is a link between not enough of the 5-HT transporter SERT to transport the serotonin out of the gut to other parts of the body including the brain. And that all along, I may have not have had a problem of actually making the serotonin itself. In all my research I could not find that they have found a treatment for this problem as of yet. Is there anything else I can do.
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