At day 22, i took raltegravir from the box but im not sure whether i drank the pill with the water or not. Normally, I remember the pills that I drank through glass of water but i cannot remember this time whether i drank this pill with glass of water or not but i counted pills and i concluded that their numbers are as expected.
I searched couple of papers and i concluded not to double the dose in the case of i used that pill.
combining emtricitabine and tenofovir from the NRTI class, and raltegravir, from the integrase inhibitor class. There ARE alternative drugs available and possibly in Thailand they use lamivudine in place of raltegravir. I am definitely NOT a chemist, so I can't tell you for sure if the combination you're taking is appropriate. You should discuss your concerns with the doctor who prescribed PEP or a pharmacist/chemist. I wish you the best.
s quite impossible to trust my mind after many days. I was bottom and we had a short penetration, because I felt too much pain. But I'm not sure if there was ejaculation or not, I couldn't find the guy in the club later that night and I have no contact with him at all. Because I'm living in another country, it was so hard to get the Pep medication.
I started PEP 2 hrs later (truvada, norvir, raltegravir, and preszita), my initial results are negative. I do not know the viral load of the patient yet, i can only pray that it is low. I know odds are 0.3% but that doesnt settle my fears, i am young and want to live a happy healthy life with KIDS! DR. please tell me if you have honstly heard of any cases like mine that have converted. I am in awe and feel like my life is totally over and ruined.
I am currently taking truvada and isentress (raltegravir) is this enough I couldn't get my prescription for dolutegravir filled...is this pep enough?
Dear jterwin,
Thanks for writing in.
Leukaemia of the central nervous system is extremely rare (in patients who do not have leukemia and presenting with headache only). I won't consider it as a possibility in your case.
Since all your exams and test results are normal, it is very unlikely that you have a serious disorder.
Given your symptoms, it is more likely that you could be suffering from a simple tension headache or sinusitis. These can be easily differentiated on examination.
I am a 57 year old female who has been diagnosed with CNS inflammation. After reading a posting from this forum, Heidi - 1-24-1999, the response stated that a patient should not be left with a diagnosis of CNS inflammation; however, that is how I have been left as 2-6-08. On 11-11-07, I had a seizure while at a pro football game and was taken to HUMC where I was admitted for 11 days.
I was just now seen by a nurologist who said that my pathology report showed CNS Vasculitis as the reason for the hemorrage in Oct. and that is very bad. He wants to look for any other secondary disease that may be causing this like Lupus. So I have done a Spinal Tap and blood work and will be doing a Brain Angiograph depending on these results. I am so bad today I can barely walk.
m confused on the prescriptions given to me I have a 7 day dose of raltegravir and dolutegravir...thought it was supposed to be 28 days?
I appreciate your blunt response. I was prescribed Truvada + raltegravir (PEP) but could not obtain raltegravir in Europe. To be fair, I called the US PEP hotline who told me that I could take Truvada as PEP as there are no studies that prove it’s ineffective, so I didn’t start it without advice from the US HIV experts. I agree my anxiety is an issue but I legitimately was seeking an answer.
Anyways I rushed to the hospital and was put on PEP at a maximum of 3 hours post exposure (truvada and raltegravir). I have taken the pills without missing any doses. At 3 weeks post exposure the lymph nodes in my neck swelled up slightly and I felt a tiny bit feverish. Checking my temperature rectally though revealed it was always in the 37s. These strange symptoms lasted only 2 days...could this be ARS?
I actually went to the clinic, got PEP (truvada 200mg and Raltegravir 400mg), also done hiv prick test which was negative. Dr was helpful and explained everything for me. He also said best thing is as u guys suggest, ask him test but also if he is clear 6 weeks back also, but that also depends on trust.
On my side i don't wanna embarrass him as twice he told me not to worry.
I am taking Truvada and Raltegravir. Yesterday, (3 days after exposure and 2 days on PEP) I had protected sex with a female partner (I, the insertive male) and when I ejaculated I am afraid some spilled off the base of the condom.
If I began PEP 24 hours after possible exposure and had protected sex with someone else on Day 2 of PEP, could I have biologically put them at risk for HIV if by any chance the ejaculate spilled off the condom?
Hi, when we are told HIV is transmitted only by 3 conditions: unprotected penetration in anus/vagina;mother to child; IV drugs. I want to ask the unprotected penetration only means penis into anus / vagina, right? not including like oral, tongue even finger and other sex activities, right?
Sedatives, tranquilizers, muscle relaxants, antidepressants, and other central nervous system (CNS) depressants may have additive CNS- and/or respiratory-depressant effects with propoxyphene. Misuse of propoxyphene, either alone or in combination with other CNS depressants, has been a major cause of drug-related deaths, particularly in patients with a history of emotional disturbances, suicidal ideation, or alcohol and drug abuse.
Some other suspicions would be central nervous system related (CNS). CNS controls atrial muscles to assist with blood pressure changes such as when we stand. There are tests that can be done to see if your CNS is not functioning right. I think you'd need to be referred to a neurologist who has background in CNS diagnosis. Something that can cause CNS problems are things like Lyme's disease but there are many others.
What's the differences between CNS depression? May i know what's the causes of depression? Does it relate to the neurotransmitter? Diazepam is an anti-depressant? It enhances the effect of GABA by binding to the benzodiazepine site on the GABA A-receptor. After binding to the receptor, may i know what's the next mechanism? GABA is an inhibitory neurotransmitter? If GABA is an inhibitory neurotransmitter, how does diazepam can cause anti-depress? I heard that it can treat seizure?
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