Diabetes insipidus and hypernatremia

Common Questions and Answers about Diabetes insipidus and hypernatremia


Avatar n tn Hello and hope you are doing well. Diabetes insipidus causes excessive thirst and polyuria. This can result in dehydration and electrolyte imbalance. So, it results in hypotension and hypernatremia (increased sodium). Sometimes excess of fluid intake can cause a suppression of ADH secretion. Excessive fluid intake in dipsogenic diabetes insipidus can lead to water intoxication, a condition that lowers sodium concentration in your blood, which can damage your brain.
Avatar f tn Decreased vasopressin release or decreased renal sensitivity to vasopressin leads to diabetes insipidus, a condition featuring hypernatremia (increased blood sodium content), polyuria (excess urine production), and polydipsia (thirst). Diabetes isn't the only thing that can cause problems. If she hit her head, an injury could have "broken" the membrane or gland (I can't remember exactly how it works) in the brain that controls proper regulation.
Avatar n tn Diabetes insipidus can occur after brain surgery or trauma, and is usually self-limiting and resolves after days to weeks. In a minority it may persist, and produces the symptoms that you describe. There may be partial production of ADH resulting in some residual ability to concentrate urine. You should be checked for adrenal insufficiency as this also results in an inability to concentrate the urine and dizziness etc.
Avatar f tn During a couple of years I have sometimes had hypernatremia (maybe dehydration), and often hypercalcemia (due to hyperparathyroidism). Maybe I have partial diabetes insipidus. I am wondering whether I could also have adrenal insufficiency. But I don't have low sodium or cravings for salt. On the contrary I don't like/tolerate salt. My S-Corsol has been normal. I don't have hyperaldosteronism. I am 65 years old. I have had much stress and burnout in my history.
1522652 tn?1291241954 serum and urine osmolality, S Na+ and copeptin, which is a marker of ADH. On next week S aldosterone, renin, Na+, K+ anf Mg++ will be measured (to find possible hyperladosteronism; I often have hypernatremia). I think that my kidneys cannot concentrate urine because HPT causes them not to react to ADH. They waste water, and my serum becomes concentrated. I have problems with my ears. I am afraid that hypercalcemia could change the ionic compositon of the endolymph in my vestibular apparatus.
Avatar f tn hypothyroidsm overmedication (upper abdominal pain sometimes during walking, polyuria, dehydration) hypercalcemia (primary hyperparathyroidism diagnosed, hypercalcemia, high PTH, polyuria) partial central diabetes insipidus (could not be excluded, polyuria, dehydration, nocturia) muscular dystonia (diagnosed) coronary artery disease (suspection, ST segment depression in ECG during recovery) chronic disease anemia (have microcytosis, high erythrocyte count) hemoglobinopathy (a heamotolgist sugge