She mentioned my ultrasound shows multiple stones in both of my kidneys. She said that she thinks I could have MSK or renal tubular acidosis. I am not going to lie, I am scared and confused. My grandpa died from ESRD when I was about 12. I remember a lot of trips for dialysis. I also remember he had a lot of pain. My question is where do I start and what can I expect? I have two young daughters, two grown children, and a grand daughter who need me. Please help!
These include medullary calcification which is seen in renal tubular acidosis, hyperoxaluria, hypercalciuria, primary hyperparathyroidism, hypervitaminosis A and sarcoidosis. Focal calcification is seen in infections including tuberculosis and metabolic abnormalities.
I hope it helps. Take care and regards.
Disorders of calcium metabolism, such as hypercalcemia and hypercalciuria, may induce the formation of calcium renal stones and deposition of calcium salts in the renal parenchyma (nephrocalcinosis). The extensive deposition of calcium may lead to chronic tubulointerstitial disease and renal insufficiency. The first signs of damage induced by hypercalcemia are seen at the intracellular level, in the tubular epithelial cells.
I saw a nephrologist in Dallas last week who believes we have renal tubular acidosis. He has seen all of the ultrasound pictures and urine test results and has ordered further testing so hopefully we will have an answer soon. This has been a very frustrating process. Three more quick questions - if I have bilateral renal cortical thinning does that mean that my kidney function is already being affected? Without treatment, will the thinning just continue and lead to renal failure?
1) Kidney losses: Certain kidney disorders such as renal tubular acidosis (for example, chronic kidney failure and acute kidney failure), 2) Magnesium deficiency , 3) Leukemia, 4) Loss of potassium through stomach and intestines, Vomiting, Enemas or excessive laxative use , Diarrhea, After ileostomy operation, 5) Effect of medicines: Water pills (diuretics) , Medicines used for asthma or emphysema (beta-adrenergic agonist type of drugs such as bronchodilators, steroids, or theophylline), Ami
By RTA do you mean renal tubular acidosis? I have definitely seen cases of 6th nerve palsy improve over time but I am a little concerned about the duration here. Usually things would start to improve in a 2-3 months. I still am not clear as to exacly why you developed the bilateral 6th nerve palsy in the first place, Not knowing all your details I would have to direct you to a neuro-ophthalmologist.
Hi,
There are a few conditions which may be associated with both the diarrhea as well as the stones in the kidneys. Examples of the same are Crohn’s disease/ulcerative colitis, renal tubular acidosis etc. For the confirmation of the same, you’ll need further investigations like a CT scan, colonoscopy, urine analysis etc. Please do consult a urologist/gastroenterologist for the same. I sincerely hope that helps. Take care.
A check of you urine pH on several occasions and some blood work may reveal that you have renal tubular acidosis, but that is realy a “long shot.”
The mucus that you mention is most likely prostatic fluid forced out with straining. I would agree with you that you are overly concerned.
Hope this helped.
S.A.Liroff, M.D.
Renal impairment, including cases of acute renal failure and Fanconi syndrome (renal tubular injury with severe hypophosphatemia), has been reported with the use of VIREAD [See ADVERSE REACTIONS].
It is recommended that creatinine clearance be calculated in all patients prior to initiating therapy and as clinically appropriate during therapy with VIREAD. Routine monitoring of calculated creatinine clearance and serum phosphorus should be performed in patients at risk for renal impairment.
Possible causes would include kidney conditions such as RTA (renal tubular acidosis) which can be diagnosed through blood and urine tests. I believe a nephrologist consult is also in order since the diagnosis and treatment for these conditions requires a different level of expertise. Low potassium levels do not cause strokes.
Hemorrhagic strokes are most commonly caused by dangerous elevations in blood pressure.
You must try and find a good nephrologist. Recurrent kidney stones are found renal tubular acidosis, medullary sponge kidney, Dent's disease (genetic disease of the kidney), hyperparathyroidism, primary hyperoxaluria and Crohn's disease. For oxalate stones you need to restrict (not totally stop) foods which are high in oxalate like beans, legumes, dark green leaves, egg plant, spinach, beets, chocolates, wheat barn, rhubarb and nuts and fruit cake.
The drug is also used in clinical trials to treat Post Traumatic Stress Disorder.[15] A pilot study suggests that Topiramate is possibly effective against infantile spasms.[16] A study by Harvard recommends topiramate as an effective treatment in the prevention of Periventricular leukomalacia in preterm infants after an hypoxic-ischemic injury.[17] In May 2006 the U.S. National Institutes of Health web site clinicaltrials.
We had one neonatologist say it was extremely unlikely, and another that said he would likely have renal damage before neurological damage, and that no neurological damage is seen until base excess is >20 and ph <6.9. Just wanted to know your thoughts on his risk for neurological issues later in life including ADD and if it is significant that base excess and ph dropped so much in first 17 minutes of life. Thanks for your help.
15-17 Urinary pH generally reflects the serum pH, except in patients with renal tubular acidosis (RTA). The inability to acidify urine to a pH of less than 5.5 despite an overnight fast and administration of an acid load is the hallmark of RTA. In type I (distal) RTA, the serum is acidic but the urine is alkaline, secondary to an inability to secrete protons into the urine. Type II (proximal) RTA is characterized by an inability to reabsorb bicarbonate.
I would ask your urologist to evaluate you for RTA (renal tubular acidosis) which will require some blood work. Actually a stone workup involves both blood and urine tests. The blood at least for: uric acid, electrolytes( sodium, potassium, chloride, hco2), creatinine, phosphorus, calcium and the urine at least for: uric acid, magnesium, calcium, citrate, oxalate, creatinine and sodium.
temporal lobe seizures, then it was taken away. I have been diagnosed with malabsorption syndrome; renal tubular acidosis; secondary hyperaldosteronism; interstitial cystitis; delayed gastric emptying; GERD; difficulty swallowing due to muschle probelms at the back of my tongue as well as flashing liquid into my wind pipe. I have edema in my ankles and my face. I can go on and on with the different types of infections I have had. I have diabetes controlled with diet.
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However, it may be severe enough to cause (as well as be caused by) 