Multiple sclerosis onset of disease
Common Questions and Answers about Multiple sclerosis onset of disease
multiple-sclerosis
Approximately 50% of patients with RRMS convert to Secondary Progressive Multiple Sclerosis (SPMS) within 10 years of disease onset. After 30 years, this figure rises to 90%.
At any one time, the Relapsing-Remitting form of the disease accounts around 55% of all people with multiple sclerosis.
SPMS is characterised by a steady progression of clinical neurological damage with or without superimposed relapses and minor remissions and plateaux.
There is no known connection, but many of us suffer with extremely cold fingers and toes as part of our MS, but don't have the Raynaud's symptoms of white, waxy digits.
And as Bob points out many people have more than one disease gong on.
Scattered cerebral hemispheric white matter FLAIR hyperintensities are nonspecific but may be sequelae of chronic small vessel ischemic disease and can also be seen in the setting of migraine headaches. However, the presence of a left thalamic lesion raises suspicion for an underlying demyelinating process.
He had an MRI of the brain that showed multiple foci of T2 and FLAIR hyperintensity in the white matter of both cerebral hemispheres including approximately 5 small lesions in the periventricular white matter, lovated at the callosal sptal margin and oriented perpendicular long axis of the lateral ventricles. THere are 2 Flair hyperintense lesions along the undersurface of the copus callosum. Focus of T2 hyperintensity is seen in the deep white matter of the right frontal lobe.
Late onset multiple sclerosis (LOMS), defined as the first presentation of clinical symptoms in patients over 50, is not a rare phenomenon as previously thought, since the prevalence ranges between 4% and 9.6% in different studies. The course of the disease is often primary progressive and pyramidal or cerebellar involvement is observed in 60%-70% of the patients at presentation.
Hi Kathy,
Mary Kay is right, there are so many mimics of the disease and it's the Neuro's job to rule these things in, or out during a thorough workup.
Do you know what this workup consists of so far? Was the MRI of the brain only? Do you know the strength of the machine used?
What labs were run? Sorry for all the questions, but it takes so much digging, and knowing this, we can help you put together a plan for your next steps if this Dr.
It is similar to primary-progressive MS (PPMS) because both types of MS are steadily progressive from disease onset. With PPMS, there is steadily worsening of neurologic function from the very beginning, although the rate of progression may vary over time with the patient experiencing occasional plateaus or minor temporary improvements.
Hello.
There have been debates about childhood onset multiple sclerosis. The lesions in your son's brain MRI hint towards a slow and probably progressive disease. I guess he has headache as the only symptoms. A lot of researchers have studied childhood onset multiple sclerosis. As young as 5 year olds have been diagnosed to have MS.
The MRI report described here suggests a possible demyelination. The diagnostic criteria used for adults can be used for children also.
Good morning.
My name is Viviane, I'm a 26 years old female. I've been seriously depressed and feeling anxious for the last couple of years.
All through this time I've been feeling symptoms like tension in my muscles all the time, tachycardia, short breath, among others (common for this kind of condition). Lately however I got a new one which is really puzzling me, I have a mild tingling sensation in my legs and a few times also in the upper limbs.
Using the EAE mouse model for multiple sclerosis (MS), the inventors have shown that the disease course and severity can be ameliorated with daily administration of antihistamine. This approach has the potential for treating MS with less side-effects and more convenient dosing than current therapeutic interventions.
Stage of Research
EAE mice treated daily with anti-histamine receptor 1 have decreased disease severity and delayed onset of the disease compared to controls.
These palsies are often caused by diabetes, but can also be the result of a head injury, tumor, multiple sclerosis, meningitis, high blood pressure or an aneurysm. The diplopia is usually resolved when the underlying condition is treated but some people may require additional help such as vision therapy, special eyeglasses or surgery.
http://www.eyedoctorguide.com/eye_problems/double_vision_diplopia.
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