t know anything about that - all I know is that before Aricept, I was in a gray mist, and after five months of Aricept, everything seemed to make more sense. Before Aricept, it was as if I had low water pressure - I just couldn't get enough juice in my brain to put thoughts together. On Aricept, suddenly my brain could make connections.
The only trippy thing I noticed was an almost hi-def moire behind my eyeballs when I went to sleep.
If a person has not been diagnosed with alzheimers but is taking aricept, is it okay? will it hurt to be taking it?
my mum was diagnosed with alzaheimer in October 2008, she was prescriped dogmatile fort 200, aricept, trittico and lustral, I wonder if this combination goes well as she is more depessed since she took the medication.
My husband has been taking 10mg of Aricept for 2 years now and I have not noticed any significant difference in his memory. Actually, my observation is his memory is becoming worse. I'm struggling with whether I should discontinue the medication since I don't see improvement and because it is quite costly. I also know Aricept is designed to slow down the dementia so if I discontinue it I'm wondering if he will become worse much faster.
I have been taking aricept for about 9 months. It is hard to tell if there is an improvement. It has been a while since I forgot my way home but I still forget a lot of little stuff. I keep taking it because I am convinced it cant hurt anything and it just might help a little.
I think you should discuss with your PCP regarding a drug called Aricept. This should be started only on prescription of your treating physician. It is used for treatment of Alzheimer’s, but is helpful for short term memory loss.
Aricept is a drug of choice to treat Alzheimer's, but its use is not restricted. It has been observed that Aricept improves cognition and function, which includes effects on memory and performing everyday tasks, hence it would have been prescribed.
I think you should discuss with your PCP regarding a drug called Aricept. This should be started only on prescription of your treating physician. It is used for treatment of Alzheimer’s, but is helpful for short term memory loss.
Aricept is a drug of choice to treat Alzheimer's, but its use is not restricted. It has been observed that Aricept improves cognition and function, which includes effects on memory and performing everyday tasks, hence it would have been prescribed.
I read your post to me about how you don't like block/replace therapy, and that you like MMI titration, what is that? HOw do you keep your patients from going hypo thyroid on only ATD's? and if they do go hypo, how do you help them if you don't use replacement? Also, does using ATD's burn out your thyroid? and does replacement burn out your thyroid? Do you consider 5mg of MMI to be a high dose of ATD?
I am confused as to which is the most important issue and how I approach each. I see a neurologist this week and have been placed on Aricept. Yet still the confusion lingers. Please give me some directive on which is the most imperative issue.
When I was having some serious cog fog, I asked my neurologist for a sample of Aricept. Had to argue with him, actually. It worked! I got back some of my cognitive function - not all, but enough that I felt confident about my decisions.
I took it for four months, total. I still have one month left over on the scrip, so if I really need it, I'll have it.
She has moderate to severe short term memory loss and has been started on Aricept. She has also recently had an MRI of the brain.
Could you explain: "moderate diffuse cerebral atrophy and chronic white matter small vessel disease"?
Thank you!
I am a healthcare professional and can not afford to seek professional help as it may compromise my job. Please let me know if titration is an effective way to come down. My thoughts are to decrease my dose every three days as follows:
-Currently taking 4, 10 mg-325 acetaminophen at 5-6 pm and 4 more (same concentration) around bed time.
Hi Margypops,
Axona is a brand-new prescription therapy launched in March 2009 for the treatment of mild-moderate Alzheimer's disease. As Alzheimer's is marked by a decrease in glucose in the brain (the primary source of food for the brain), Axona acts by providing/metabolizing glucose in the body to act as an alternate source of food for the brain.
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