modic changes

thank you so much for your help i have been having trouble holding my urine do you think that has anything to do with my back alsoi have degenerative narrowing and modic changes at c5-c6 and degenerative changes at vertebral body endplates lower and mid thoracic accompanid by modic changes what does modic changes mean again thank you

L5-S1. Stable Modic type I changes @L5-S1. Modic type I changes are most often sterile with vascular granulation tissue producing the endplate edema. Some patients with Modic type I changes ultimately demonstrate chronic discitis or osteomyelitis with low virulence pathogens. Back to the original question, my family practitioner today said, I was getting better? I have reached out to my Neurosurgeon in AZ & awaiting a call from another surgeon 3 hours from here.

It states, end plate irregularity and mild modic endplate changes. Posteriorannular fissure and small broad-based posterior disc protrusion. Mild facet and ligament hypertrophy. Moderate to moderately severe metformin narrowing. Abutment of the exiting left L4 nerve root. Subarticular recess narrowingwith abutment of descending L5 nerve root.

l3-l4 minimal bulging disc broad base disc protrusion at l4-l5 mild central canal and minimal bilateral neuroformanial stensis mild broad disc protusion at l5-s1 central canal stensis mild narrowing of right neuroforamina and advanced ddd at c5-c6 with siinal canal stensis degenerative changes at vertebral body endplates lower thoracic and midthoracic with modic changes narrowing an modic changes at t7-t8-t9-t10 what are HEMANGIOMAS thank anyone for your help and do i need surgery

There is straightening of the lumbar spine lordosis and advanced Modic endplate changes at L4-L5 with discogenic disease. -L3-L4: Mild broad-disc bulge extending 1-2 mm beyond the vertebral endplate. There is early tear of the annulus fibrosis of the right para-lateral margin. There is moderate left and early mild right neuroforaminal narrowing. Compression of the exiting left L3 nerve is suspected. Fluid is noted within the facets.

The impression of MRI REPORT Degenerated L/S spine with L Para-central disc protrusion at L5-S1 causing compression of L sided existing nerve roots with diffuse central disc protrusion at L4-5 disc bulges at L2-3 and L3-4 with disc desiccations atL2-3 L3-4 ,L4-5 and L5-S1 levels, with modic type II changes at L4, L5, and S1 with posterior marginal Osteo puytoses seen at multiple levels.

Subchondral degenerative change of vertebral end-plates in lower-lumbo-sacral spine is seen. There is Modic Type II degenerative change of the end-plate detected at L5-S1 with ostephyte formation. The posterior articular pillars and facet joints show moderate degenerative hypertrophic changes with ligamentum flavum buckling. The neural foramina are patent. The disc spaces show normal hydration except at the lower lumbar spine.

HETEROGENEOUS MARROW SIGNAL WITH T1 MARROW HYPOINTENSITY IS RE IDENTIFIED. THERE ARE INCREASED MODIC TYPE 1 ENDPLATE CHANGES POSTERIORLY AT L2-L3, L3-L4, AND L4-L5. THE CONUS MEDULLARIS IS NORMAL IN CONTOUR AND TERMINATES AT L1. L1-L2: THERE IS NO EVIDENCE OF DISC HERNIATION OR SPINAL STENOSIS. L2-L3: DISC BULGE AND SMALL RIGHT POSTEROLATERAL DISC HERNIATION WITH CAUDAL DISC EXTRUSION. THE DISC HERNIATION HAS DECREASED SLIGHTLY COMPARED TO THE PRIOR MRI.

L4-5 broad based disc buldge early posterior arthropathy at L5-S1 broad based disc buldge bilateral facet arthropathy narrowing of the bilateral neural foramen secondary to facet arthropathy, posterior osteophytosis and disc buldge.Active modic-type endplate degenerative changes. Dr. says my results weren't bad and that I can try nerve blocks which I did and it helped for a limited time.

- congenital pedicle shortening - Spondylarthrosis from L3 and downwards with smaller efusion in facet joints, ligamentum flavum hypertrophy and indicating fatty infiltration of pedicles (modic type 2 degeneration) --> considerably bilateral neural foraminal stenosis from L3 downwards - spinal channel is normally wide at all levels - conus medularis has a normal signal - shown skeleton and discs have normal signal

Disc desiccation with Modic type 1 endplate changes. Disc bulge with a right paracentral protrusion does not significantly indent the ventral thecal sac although there is asymmetric approximation and likely abutment of the anterior margin of the decending right S1 nerve root (series 6 image 25). This along with facet joint hypertrophy moderately narrows the bilateral foramen. The visualized sacal nerve root are other wise unimpinged.

C2 - 3 level is unremarkable C3-4 level demonstrates mild bilateral uncovertebral degenerative changes as well as a small posterior disc osteophyte complex. No Sign central canal stenosis is identified, Mild right neuroforaminal stenosis. C4-5 level demonstrates mild to moderate uncovertebral degeneration and associated moderate bilateral neuroforaminal stenosis. C5-6 level demo marked disc hight loss and end plate osteophytosis as well as Modic type 2 changes.

At the L4-L5 there is significant disc space height loss along with MODIC type ll changes present at the endplates. A broad-based posterior disc protusion is identified with surperimposed high signal. Findings compatible with annular tearing.

Loss of disk height and disk desiccation with endplate irregularity. Modic type 1 endplate edema. Central disk protrusion exerts minimal mass effect on bilateral transverse nerve roots. No signifian't spinal stenosis. Mild bilateral forminal narrowing. Mild facet arthropathy bilaterally. I have tingling and numbness in both legs. I have had this for 2 years. An epidural steroid injection helped the left leg for awhile but not the right leg. I still do my physio.

There are Modic Type of endplate changes present at L4-L5 level with endplate bone marrow edema; otherwise, no focal bonme marrow abnormalities are seen. Please I'm only asking someone to please explain to me how injured am I and if any surgery can help me.

The disc is desiccated, partially collapsed, and aggravated by patchy Modic type 2 reactive end plate changes and intradiscal gas, not to mention grade 1 anterolisthesis of L5 on S1, allowing uncovering of the posterior disc margin and formation of bilateral foraminal protrusions. In conjuction with marked bilateral L5-S1 facet arthrosis and pars interarticularis defects, there is a severe "up-down" biforaminal encroachment and paradoxical widening of the central spinal canal.

AP alignment shows mild anterior subluxation of L5 on S1. Modic I-II changes are seen at L3-4; elsewhere, marrow appears within normal limits. Discs are desiccated below L3. Conus medullaris is visualized at about T12-L1. L1-2: Mild facet overgrowth. Otherwise, negative. L2-3: Greater amount of facet overgrowth. No foraminal stenosis or thecal sac distortion. L3-4: Left hemilaminectomy at L3. There is marked right lateral subluxation of L3 on L4.

bilateral disk endplate spurs and bilateral neural narrowing and mild central canal stenosis as a result.the prevertabral soft tissiues are unremarkable.there are signal changes around the c6-c7 intertvertabral disc,in the subcortical bone,consistant with modic-type degenerative signal change,secondary to degenerative disc disease.impression:dicognative degenarative disease,most accentuated c5-c-6,c6-c7 with mild central canal stenosis.

AP alignment shows mild anterior subluxation of L5 on S1. Modic I-II changes are seen at L3-4; elsewhere, marrow appears within normal limits. Discs are desiccated below L3. Conus medullaris is visualized at about T12-L1. L1-2: Mild facet overgrowth. Otherwise, negative. L2-3: Greater amount of facet overgrowth. No foraminal stenosis or thecal sac distortion. L3-4: Left hemilaminectomy at L3. There is marked right lateral subluxation of L3 on L4.

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