I do know there are different anti-platelet mechanisms with each medication you are referring as well which mechanism of action is perferred for a specific malady and the interaction with other medicaion.
My experience is with dual therapy (aspirin and plavix) and the last I heard that therapy is a appropriate post DES implant. It seems plavix helps reduce the risk for clots at implant site, and after a year the risk of clots is less than the risk of excessive bleeding with plavix.
I have beem taking carvedilol for three years and have never seen a description of it so concise and clear.
I take Warfarin as follows: 3mg 2xda for 2 da and 4mg 2xda rest of week. Can't seem to get my levels even. Is the dosage too little or not enough?
The abnormal levels of INR may suggest that there is a problem with the bleeding and clotting mechanism. You must necessarily get the dose of Warfarin adjusted. High or low INR may lead to abnormal clotting or spontaneous bleeding. The medicine should keep the blood just thin enough to prevent clots. If the blood is too thin, you may bleed too easily. Severe headache while being on Warfarin therapy needs immediate medical attention.
is it safe to use blood clotting spray for minor wounds&cuts for warfarin user?
In three months on Warfarin, I thinned over the top and front of my hair (I am a female aged 70) and also my nails went chalky, plus I couldn't sleep well and felt very off colour. The blood tests which were weekly due to an unstable INR became an ordeal when it took two or three attempts. I took Warfarin (for AFib) for about three and a half months and then refused to take it any longer, saying that I would take my chances on aspirin only.
Potassium is a chemical element and it is part of the electrolyte that should be in balance with other lytes to adequately conduct an electrical impulse that originates with the sinus node in the right atrium. The mechanism of action is not completely understood, but what is when a voltage (unit of potential for conduction) and electrolyte there will conduction and passage of electrical impluses.
The proposed mechanism is that continual drinking of large amounts of alcohol induces the hepatic metabolism of anticoagulants. Effects are highly variable and significant INR/PT fluctuations are possible.
MANAGEMENT: Patients taking oral anticoagulants should be counseled to avoid large amounts of ethanol, but moderate consumption (one to two drinks per day) are not likely to affect the response to the anticoagulant in patients with normal liver function.
http://www.nasdaq.
Im 47 & have permanent af -on 14mg warfarin and bisoprolol. Had 2 dc cardioversions which lasted hours and a few days before ectopics with sinus followed by full af set in thereafter. Have slight LV hypokinesis. now AADs suggested sotalol/flecanide. Im concerned about proarrhythmias and toxicity. High Warfarin dose and resistance to remain in sinus suggests to me that Id need quite a high sotolol dose too.
In a conflicting report, the effects of acute and chronic dosing of APAP on warfarin pharmacokinetics and pharmacodynamics was studied in 20 healthy males in a 2-phase, randomized, crossover study. The mean serum concentrations of S- and R-warfarin did not differ significantly between treatments. There was no change in anticoagulation response between treatment and control (4). INR should be monitored more closely when APAP use exceeds 2 g/day or chronic use > 7 days occurs.
References: 1.
I take warfarin (generic coumadin) and have for about 15 years. I discussed changing to Pradaxa with my cardiologist when that drug first came out, I decided not to use it because of cost (warfarin cost about 12 cents a day) and the fact I do well on warfarin, albeit I have to have my blood coagulation tested once a month. Recently I have seen a lot of "bad news" about Pradaxa including law firm adds looking for new participants in a class action suit, as I understand it...
I also suffer from AFib and use Warfarin to hold INR 2.0 - 3.0. I, and I believe most, do not have a bleeding problem in that range.
I have no understanding of what you did you to nose, bu *** is clear you can't doo that. If you have nasal congestion blowing doesn't make it better anyway.
Our nasal tissues strength are not all the same, some have problems with bleeding even with an INR - 1.
AFig can cause the formation of clots in the artial chamber due to the incomplete pumping action characteristic of AFib. If/when these clots move into the blood stream and if they block oxygen/blood to the brain a stroke occurs. An Anticoagulant, such as warfarin/coumadin reduce the chance that such clots will form, thus reducing the possibility of a stroke.
I do not believe the taking of meds to control the heart rate, e.g.
Factor V Leiden is a variation of normal Factor V which is part of the clotting cascade, which is essentially a chain reaction of proteins acting on proteins, ending in the formation of a (fibrin) clot. Platelets are another part of the body's clotting mechanism, they start things off by sticking together to plug the initial hole in the blood vessel & the fibrin clot then adds to this, making it stronger.
ginger and glucosamine sulfate will affect coumadin (warfarin) I'm on warfarin and can't take ginger or glucosamine and a host of other supplements.
You should contact your caregiver for advice regarding your medication as they would have your medical history, general health, etc. There is a noted drug interaction with Viagra and warfarin in certain patients. In patients who have pulmonary hypertension, there is an increased risk of bleeding when taking these medications together, etc..
Thanks for your question, take care.
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