Don't wait. I have been through many caths. They aren't a big deal and it is the only difinitive way for a Doctor to tell. Because the actually see the issue. Most caths are done on simple out patient procedure and if everthing is OK then go on the vacation stress free. Don't gamble with your life. Take it from a guy who had a sudden death heart attack at 37. Another MI at 38 and ended up with 7 stents. I had stress tests, 3d cat scans of my heart, ekgs, echocardiograms.
In (7/05), Nuc study revealed small area of ischemia and global hypokinesis in inferior wall. Now, 12/2010- EF of 48 percent with inf wall hypokinesis to akinesis. Data demonstrates large inferior wall perfusion defect which is predominately fixed. There is a small amount of reversibility at edges, in particular, inferolateral and inferoseptal wall that may be consistent with peri infarct ischemia.
Hi I am 34 Year old , I had an Acute inferior MI on May - 4 at around 8.00 pm last and there was complete blockage in RCA, on 5th May 2007 around 1.00 am my Echo was done and my ECHO showed LVEF = 35% with LA Dilated, I later got a stent implanted in my RCA. I want to know if this LVEF = 35% and LA dilated was due to MI and if LVEF is improved after stent is implanted or if any way by which it can be improved.
Immediate post stress imaging demonstrates augmented contractility of all wall segments - inferior and psoterio walls remain relatively hypokinetic.
Abnormal stress echo demonstrating a previous non transmural inferior/posterior MI with some contractile reserve. There is no reversible ischemia.
What does this really mean? The cardiologist said that I have a suspect spot on the back of my heart - not to worry about it and he had no reason for my chest pain.
I just found out that I have a large sized, mild to moderate myocardial perfusion abnormality at apical to basal inferior and inferolateral wall, with partial reversibility at the inferolateral wall. Seems the mediastinal border between chambers is slightly thickened, but only by .2cm so that's hardly anything.
Stress tmographic images of the left ventricle revealed inferior and lateral wall defects seen of moderate size. The resting tomographic images revealed normal perfusion. Gated images revealed abnormal systolic thickening in the inferior and lateral walls. The ejection fraction is calculated to be 36%.
CONCLUSIONS: A) No anginal symptoms in response to exercise and with evidence of ischemia.
B) The isotope study revealed evidence of ischemia in the inferior and lateral wall of large size.
Fixed Apical/Inferoseptal Defect with Prior MI in the LAD or RCA distribution; no stree defect, mild LV enlargement, Inferior/septal hypokinesia, LVEF 56%. I was sent to a cardiologist for a cath which came back with a normal left coronary system and 20% irregularity at the sheperd's crook. Ejection 55%. Risk considered low by the cardiologist.
I attempted to explain to you, on another identical post as well, isosorbide is generic for sorbitrate and is taken to PREVENT angina due to ischemia. If you want medication handy in the event you are experiencing angina, nitro is fast acting and within seconds or so there is relief. Doesn't make sense to keep sorbitrate handy as there is not immediate relief.
My husband passed away 2yrs ago from a inferior wall mi in his sleep, when I got up he went into what seemed like convaultions and quit breathing. It took 15 mins for the ambulance to arrive and they worked on him for 15 mins before they got him back. He continued to have siezures and had to keep him heavily sedated cause siezure meds alone did not work. My step daughters seem to think that over time his brain could have repaired it's self. My? Is.
The report reads
Mild left ventricular Dilation, basal septal and basal inferior wall motion hypokinesia,Overall LV Systolic function preserved,
No evidence of aortic valvulopathy
trivial mitral reguurgitation
Normal right ventricular systolic function, right chamber not dilated, no significant pulmonary hypertension 25 mmhg systolic pap, Trivial pulmonary valve regurgitation, no evidence pulmonary stenosis, no patent foramon ovale.
The EKG came back as abnormal, possible inferior wall infarct, age-untermined. Surgery has been cancelled because of this report. I have a history of high blood pressure which is normally in control with my medications. I don't recall ever having any type of serious chest pain in the past. I do tend to become short of breath with activity, especially with climbing stairs and going up hills, but I tended to relate this to my weight and being very out of shape.
I am a 48 year old male, 6 ft and 76 kg. I had Ant wall MI in August 2007 with inferior wall ischaemia. Stent was inplanted in LAD with 100% blockage. OM2 100% blockage but filled by collaterals. By december 2007 I was taking 12.5mgx2 betablocker(carvidilol), 5mgx2 ACE-I(ramipiril), Monotrate 10mgx1, Aspirin 150x1, diuteric and Clopidrogel(anti platlet) 75x1, drug for cholestrol. I was able to walk 1.5 miles (35 minutes) and do my office worl(table job) without getting tired by evening.
i am a 66-year-old man with recent inferior wall MI and following are my angiography details:
left main: normal bifurcates into LAD and LCx
LAD: type III vessel shows two tandem proximal 40% stenosis followed by 80% stenosis.
LCx: nondominant vessel and is normal.
RCA: dominant vessel and shows 75% stenosis immediately after RV branch and 40% stenosis before bifurcation of PDA and PLV.
renals: normal
LIMA/RIMA: normal.
I had a MI last October 2012. Since than I am on medication and there are no major problems. Only problem is I get tired easily and at times feel there in no strength in the body.
Recently I have undergone 2-D Echocardiography. The impression of the report is as under:
1. Bi-Ventricular Systolic Function Adequate.
2. No Diastolic Dysfunction.
3. Inferior Wall Hypokinesia noted at Rest.
4. LVEF= 61%
What does this suggest ? Is there anything to worry about? What further test required?
My mom was suffered from Inferior MI and she was thrombolysed. Now her general condition is fair. Her BP is maintained at 110/80 mmHg and her heart rate is round about 65.She has no DM and Hypertension. But her cholesterol level was higher than normal. But now within normal limit.Now, cardiologist gave her metoprolol 100 mg bd , aspirin, clopilet, atovarstain 20 mg hs , ivabaradine 5mg half bd and monotrate bd and multivitamins.
focal region of dyskenisis involving basal septum and akinesis of basal inferior wall, EF is normal. Left Atrium at 4 cm, upper end of normal; tricuspid valve regurgitation identified. Results rule out heart failure with the statement overal function is well preserved.
There is some wall movement impairment (rom prior MI) but that doesn't effect the functionality of the heart.
There is also the pericardium, which is the protective layer around the heart. Now, your Mom has basal and and mid-epicardial enhancement of the interior wall. This means that the inner wall (i.e. interior not lateral) of the ventricle in question has some thickening of the outer most layer of her heart. So, if you picture her heart, on the along the outer portion of the inside wall of her ventricle heart there is some thickening.
2) Fixed perfusion defect at apex,antroseptal wall,apical anterior segments and almost whole of the inferior wall showing no sign of viability scan. All other segments are viable including mid and basal anterior wall,IVS and lateral wall.
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These are the angiography and viability scan reportf my father...
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