A full hemodynamic assessment was made and his cardiac output was 6.8 liters per minute. His PA pressure is normal at 28/15 as was the RV pressure normal at 29/14. His pulmonary wedge pressure is a bit high at a mean pressure of 21, his RA mean pressure is also high at 17. He also had full oximetry assessment to rule out intracardiac shunting. There is no evidence of any intracardiac shunting seen.
The pain lasts anywhere from a few minutes to several hours. I have been prescribed nitroglycerin to take as needed for chest pain and this medicine surely helps. I've had several EKGs and cardiac enzymes done which were all normal. I also had a threadmill/stress test done which was normal. My cardiologist thinks the the probability of me having an occluded/blocked coronary artery is very low.
The difference of normal cardiac output has a variation of about 34%. Therefore. just based on cardiac output variation the PAP can vary by 34%.
I have read your doctor's report (what you provide) to you, and there is not any opinion that is not in keeping with a good analysis and evaluation. I think you may be reading information that is confusing, however, if you don't trust your doctor's evaluation then see another doctor.
QUOTE:"by PikaPika88, 3 hours ago
A doctor works at the country side told me..... MI won't affect the blood pressure. Many MI patient remain in normal range of blood pressure. She didn't mention about the heart rate (pulse)."
How can it not effect vital signs? There has to be a misunderstanding!
The system compensates when there is low cardiac output. MI damages heart cells and impedes left ventricle contractions. Impeded left ventricle reduces cardiac output. ....
I was a patient with heart failure and hospitalized for several days. The discharge from the hospital provided written instruction saying weigh yourself every morning and if there is a few pounds gained over night to contact the doctor as that could indicate being in heart failure mode and fluids were backing up into the lungs and/or peripheral locations... the heart would not be strong enough to output the amount of blood received from the lungs. .
Myocardial infarction pain is characteristically crushing retrosternal chest pain which may move to arms, shoulder, neck, teeth, jaw, belly area or back. This can be squeezing, lasting for 20 minutes. Rest and nitroglycerin may help relieve the pain to some extent. Other symptoms are anxiety, cough, dizziness, palpitations, shortness of breath etc. your doctor would like to check the cardiac enzymes like troponin and CPK, CPK MB.
t ever heard of an increase of the EF when pulse rate increases. Cardiac output (CO) is increased and that metric is the total amount of blood pumped into circulation based on heart rate for one minute. A pacemaker would control the heart rate.
QUOTE: "My GP told me to cut the Atenolol in half and gave me an RX for Enalapril Maleate 5mg daily. He said hopefully the Enalapril would dilate the output artery(my words?) and increase EF and blood flow thus reducing fatigue".
My health history has alway been proper weight, almost normal blood pressure (slightly high systole (if I had "stiff" arteries, it would show a high systole abnormally), cholesterol below 149, etc. Don't judge by age! My parents had a long healthy life without ever being on any medication until the final days..
I took good health for granted at a very young age. Smoked, binge drinking. improper diet, all night parties, etc.
If MVP is serious such as to reduce the cardiac output, the cardiac output is reduced, the heart will over compensate with an enlarged heart. An enlarged heart can effect the electrical pathways and that with RBBB can cause ventrical tachycardia.
"People who have a heart attack and develop a bundle branch block have a higher chance of complications and death than do people who have heart attacks and don't develop a bundle branch block.
The contribution of the heart to the regulation of blood pressure is through changes in cardiac output. Cardiac output is defined as the product of heart rate and stroke volume.
Either can be modified by climatic forces. The heart rate component is regulated by the autonomic nervous system, so its level of activity depends on the intensity of the stimulus at the time.
I was under enormous stress when the pains started and the fear that it was cardiac in origin added to it. I had the nitroglycerin sublingual tablets. I took them, sometimes they helped, other times they didn't. To this day I have no definitive answer as to the cause of my pains, but they seem to have subsided for the past 6-7 months. Try the nitro and see if it helps. My guess is that it won't help indigestion, but would if it was due to vasospasm or other cardiac cause.
I recently had a right heart cath due to continuing symptoms of shortness of breath, chest pain on exertion, cold/tingly hands, swelling in ankles, fatigue (especially on stairs), "foggy" thinking episodes, pounding heartbeat at times. The results were a bit confusing to me and the doc didn't discuss much of it over the phone. All I have are a limited set of the test results as follows: PA Pressure - 88.9; Cardiac Output - 16.2; SBP - 150.
Extreme fatigue is a condition that exists. I have been told by my cardiologist and cardiac surgeon that these conditions should not exist related to my valve regurgitations. I also have been diagnosed with Fibromyalgia and a peripheal (sp?) sensory neuropathy.
Should I be seeking a second opinion?
Thank you.
My experience for heart failure was normal blood pressure and a heart rate sustained at 115 bpm. The higher heart rate is to increase the cardiac output (amount of blood pumped/per minute). The increase is due to a failure of the heart to meet the system's demand for oxygenated blood with normal heart rate, and the increase of the heart rate is compensation for a low volume per heartbeat.
Also the cardiac output may be inadequate, hence the breathlessness. For relief from your symptoms the blood flow and the cardiac output need to be improved. I would suggest to limit your walking, keep high risk factors like hypertension and diabetes under control and discuss the treatment options with your doctor.
Hope this helped and do keep us posted.
Eventually reduced cardiac output causes the left side of the heart to enlarge to compensate for the reduced cardiac output, and the left ventricle will begin to lose its contractility and as a consequence the systole EF will decrease.(Frank/Starling phenomonon).
By and large, diastolic dysfunction is chronic process. When this chronic condition is well tolerated by an individual, no specific treatment may be indicated.
1- What was the reason for this raised enzymes that affect the cardiac for the next time
2- Is there any way to raise the Ejection fraction to be 50% or above
3- Can the Patient make another bypass surgery
4- Overall , what can be done with Patient to perform his cardiac activity
Description: the Patient IS 61 YEARS old , heavy smoker , who underwent a coronary artery bypass surgery in 2001 wherein he received one vein graft to diagonal, PDA and marginal and one LIMA to LAD
Current: the
I agree with the above. Nitroglycerin only works if the problem is coronary artery disease.Make sure you've had a cardiac evaluation performed.
The following day I had the nuclear and echo gram test. The 3rd day I had chest discomfort again and was given nitroglycerin and immediately the discomfort was gone (first time I ever had a nitro tablet). This happened twice in my 4 day stay in the hospital.
First told I had a cardiomyopathy because my stress test showed diffuse hypokinesis, stress fraction of 37% and resting fraction of 42%.
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