Eithne01,
The treatment of chronic myeloid leukemia (CML) has changed significantly since the late 1990s, with the development and subsequent approval by the Food and Drug Administration of imatinib (in 2001), the first of a class of medications called tyrosine kinase inhibitors, which target the specific abnormality that causes CML.
Before that time, patients were treated with other therapy, including interferon and bone marrow transplant, and had very poor outcomes.
Are you currently on any medication like imatinib? The neutropenia can be due to your current medication. You can ask your hematologist for G-CSF injection if you have persistent neutropenia. Patients with CML are immunocompromised and can easily acquire infections. Neutropenia also puts a patient in an immunocompromised state.
For now, it is very important to prevent acquiring any infection. You should avoid crowded places and persons with ongoing infection.
Good luck.
Hi.
Chronic Myelogenous Leukemia (CML) is a blood disorder caused by an acquired genetic defect in the pleripotent stem cell. It has several phases: indolent chronic or stable phase, aggressive or advanced phase, and accelerated and blastic phase. The transition between phases may take years (on the average, 4 to 6 years from the stable phase to aggressive phase).
Average survival rates are generalizations, not applicable to a particular patient. The prognosis is relatively good. Newer techniques are being tried, including imatinib and stem cell transplantation.
All the best, and God Bless!
TY for the reply. Yes I was told and am thinking that its parasitc or allergy. I went to aruba in June but that wouldn't have an affect on me now, would it?
Therapy with corticosteroids is used for the treatment of most and additional therapy is available and dependent upon the type of HS. Newer medicines such as imatinib and mepolizumab are said to be promising. So, should your son have HS, there is reason to be optimistic regarding a positive response to currently available therapy.
You should request, of his doctor, an additional explanation for the tentative diagnosis of pre-hypertension.
Good luck.
//en.wikipedia.org/wiki/Trophoblast
"The placenta functions as an immunological barrier between the mother and the fetus, creating an immunologically privileged site. For this purpose, it uses several mechanisms:
It secretes Neurokinin B containing phosphocholine molecules. This is the same mechanism used by parasitic nematodes to avoid detection by the immune system of their host.[2]"
http://en.wikipedia.
//en.wikipedia.org/wiki/Foreskin
The penis head is properly called the "glans penis".
https://en.wikipedia.org/wiki/Glans_penis
You are blessed and fortunate to have an intact foreskin.
I love wiki! I noted that lab results of my recent ER visit showed high levels of neutrophils, but supposedly within the normal range. Following that, I discovered that it isn't the bacteria or filaria, whichever I have, sealing themselves in. It's my immune system trying to seal them out via a process discovered in 2004, known as Neutrophil extracellular traps. Webs! I couldn't understand what was making the webs, now I know. Something new everyday.
//en.wikipedia.org/wiki/Gliosis
Basically when they use 'gliotic' or 'gliosis' they're talking about neuron damage. So it sounds like you have some neuron damage in the right frontal lobe, at the back, and you have some mild volume loss. Volume loss means that your brain has shrunk slightly - or atrophied.
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