Eithne01,
The treatment of chronic myeloid leukemia (CML) has changed significantly since the late 1990s, with the development and subsequent approval by the Food and Drug Administration of imatinib (in 2001), the first of a class of medications called tyrosine kinase inhibitors, which target the specific abnormality that causes CML.
Before that time, patients were treated with other therapy, including interferon and bone marrow transplant, and had very poor outcomes.
Are you currently on any medication like imatinib? The neutropenia can be due to your current medication. You can ask your hematologist for G-CSF injection if you have persistent neutropenia. Patients with CML are immunocompromised and can easily acquire infections. Neutropenia also puts a patient in an immunocompromised state.
For now, it is very important to prevent acquiring any infection. You should avoid crowded places and persons with ongoing infection.
Good luck.
Hi.
Chronic Myelogenous Leukemia (CML) is a blood disorder caused by an acquired genetic defect in the pleripotent stem cell. It has several phases: indolent chronic or stable phase, aggressive or advanced phase, and accelerated and blastic phase. The transition between phases may take years (on the average, 4 to 6 years from the stable phase to aggressive phase).
Average survival rates are generalizations, not applicable to a particular patient. The prognosis is relatively good. Newer techniques are being tried, including imatinib and stem cell transplantation.
All the best, and God Bless!
TY for the reply. Yes I was told and am thinking that its parasitc or allergy. I went to aruba in June but that wouldn't have an affect on me now, would it?
Children up to 5
For children up to age 5, normal blood sugar targets are 100 to 200mg/dL. Fasting blood sugars should be near the low end of the range.
Children 5-11
For children who are between the ages of 5 and 11, normal blood sugar targets are 70 to 150mg/dL. Fasting blood sugars should be nearer to 70mg/dL. Blood sugar after meals and before bedtime should be near 150mg/dL.
I have used it and noticed it especially targets the stomach fat. I drink one glass around 8am everyday.
I have another questioned for you, do you know what copaxone is and what it targets in a person? Here is a little info for you. I went and looked on the box of copaxone that I have stored in the fridge for any opiate and the only word is glatiramer. So I went to Bing and typed in disposal of glatiramer and and the 5th one down was this:
COPAXONE®(glatiramer acetate injection)
copaxone (glatiramer acetate) injection [Aventis Pharmaceuticals Inc.] DESCRIPTION ...
Small HBV pharma usually do not have HBV experts, so some of them wasted a lot of time and effort in pursuing the wrong targets. Take Arrowhead, the experts would have advised them that silencing of HBV x-gene and precore gene are just as good targets as the s-gene.
These experts are all recruited late in the process, hope they can help to design better targets and clinical trials.
Just my opinion.
Paxil is an antidepressant that blocks the enzyme that breaks down serotonin and targets serotonin to only certain receptors while bypassing others, allowing serotonin to wash longer in the receptors. Xanax is a benzodiazepine, an anti-anxiety and anti-seizure medication that targets GABA receptors. Benzos act only for a short time, some longer, some shorter, while antidepressants work all day while you're on them.
No. They do different things, and target different neurotransmitters. Zoloft is an ssri, which targets selected serotonin receptors while also blocking the body's natural process for breaking down and eliminating used serotonin so used serotonin can circulate longer in the targeted receptors (and only those receptors -- other receptors will shut down because the brain will perceive them as no longer needed). Xanax targets GABA receptors, a completely different neurotransmitter.
Therapy with corticosteroids is used for the treatment of most and additional therapy is available and dependent upon the type of HS. Newer medicines such as imatinib and mepolizumab are said to be promising. So, should your son have HS, there is reason to be optimistic regarding a positive response to currently available therapy.
You should request, of his doctor, an additional explanation for the tentative diagnosis of pre-hypertension.
Good luck.
The tests you had were designed to detect antibodies to the HIV virus. Sometimes antibodies recognize targets other than their specific targets, causing reactions that do not indicate of a true infection – these reactions are called “false positive”. That is what happened to you. The flu vaccine caused your body to produce antibodies that reacted withthe targets present in the HIV test even though you did not have HIV.
Once again, you do not have HIV.
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