Eithne01,
The treatment of chronic myeloid leukemia (CML) has changed significantly since the late 1990s, with the development and subsequent approval by the Food and Drug Administration of imatinib (in 2001), the first of a class of medications called tyrosine kinase inhibitors, which target the specific abnormality that causes CML.
Before that time, patients were treated with other therapy, including interferon and bone marrow transplant, and had very poor outcomes.
Are you currently on any medication like imatinib? The neutropenia can be due to your current medication. You can ask your hematologist for G-CSF injection if you have persistent neutropenia. Patients with CML are immunocompromised and can easily acquire infections. Neutropenia also puts a patient in an immunocompromised state.
For now, it is very important to prevent acquiring any infection. You should avoid crowded places and persons with ongoing infection.
Good luck.
Hi.
Chronic Myelogenous Leukemia (CML) is a blood disorder caused by an acquired genetic defect in the pleripotent stem cell. It has several phases: indolent chronic or stable phase, aggressive or advanced phase, and accelerated and blastic phase. The transition between phases may take years (on the average, 4 to 6 years from the stable phase to aggressive phase).
Average survival rates are generalizations, not applicable to a particular patient. The prognosis is relatively good. Newer techniques are being tried, including imatinib and stem cell transplantation.
All the best, and God Bless!
TY for the reply. Yes I was told and am thinking that its parasitc or allergy. I went to aruba in June but that wouldn't have an affect on me now, would it?
Therapy with corticosteroids is used for the treatment of most and additional therapy is available and dependent upon the type of HS. Newer medicines such as imatinib and mepolizumab are said to be promising. So, should your son have HS, there is reason to be optimistic regarding a positive response to currently available therapy.
You should request, of his doctor, an additional explanation for the tentative diagnosis of pre-hypertension.
Good luck.
The two phenomena of leukotriene antagonist use and the occurrence of Churg-Strauss syndrome are probably related more to the reduction in corticosteroid dose afforded by leukotriene antagonists (and the resultant unmasking of latent Churg-Strauss syndrome) than to any truly causal relationship. Patients with mild disease may be treated with prednisone.
In reality that is the dose exposed to at the skin on the beam side, and the dose decreases with tissue depth as some of the radiation is absorbed at each level as it penetrates tissue and is therefore gone and cannot expose the deeper tissues. Many people don't understand this, but tissues actually absorb radiation, and only a small % of the radiation exits the body to form the image. Say 100% radiation enters the skin, maybe a few percent leaves on the backside.
A quarter grain is a very conservative dose, but I could be more specific if you post the lab work that led to her decision (with reference ranges from your own lab report, please).
"She is also waiting 6 weeks to recheck and then only doing TSH as she says I don't need the T3 and T4 checked anymore since surgery." Six weeks is too long when actively adjusting meds. Four weeks is plenty of time. She's dead wrong about just testing TSH.
i am a non responder,after 2 previous tratments with other interferons,now after having a liver biopsy my dr wants me to go on pegasys with copegus, he wants to prescribe the 180mcg/0.5ml shot along which is prefilled but he wants me to take some of the other prefilled shot which would make it more than the normal 180 mcg. i did not ask him exactly how much it will total cause i have doubts about this,i have never heard of this. and may just stick with the 180 once a week.
Welp...Took my final dose (ledipasvir/sofosbuvir/riba) this morning and gave my 12 week blood draw. We shall see. Doc gave me a realistic 80's % chance of svr given my prior Tx's and the fact I am cirrhotic. I have a better chance than ever before.
Yes, you are being too impatient. The 50 mcg is a starting dose. Dependent on the levels of your biologically active thyroid hormones, Free T3 and Free T4, it may take a while to ramp up your meds to achieve symptom relief. In addition, if your doctor only tests for TSH and medicates accordingly, you may never get there.
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