Eithne01,
The treatment of chronic myeloid leukemia (CML) has changed significantly since the late 1990s, with the development and subsequent approval by the Food and Drug Administration of imatinib (in 2001), the first of a class of medications called tyrosine kinase inhibitors, which target the specific abnormality that causes CML.
Before that time, patients were treated with other therapy, including interferon and bone marrow transplant, and had very poor outcomes.
I have one of these clival cysts. I read they can cause double vision. I'm curious if it is the cause of my central apnea.
He was then refered to a neurosurgeon that told him this is most likely a inoperable chordoma because of the location in his brain. He told my husband that he would like to consult with his partners and ordered an EMG due to some weakness he feels he has in his hands. That was 12 days ago and we have not heard a word since. We are going out of our minds with worry. I have called the MD's offce six times and cannot get past the receptionist.
1,000,000 cases) and especially at my age, in my early 20s, most chordomas occurring in the 40-70 age group. Also, chordoma seems to go beneath the sacrum/coccyx, whereas this lump appears to exist only above. Any ideas what this could be? Could it be chordoma?
Thanks!
1,000,000 cases) and especially at my age, in my early 20s, most chordomas occurring in the 40-70 age group. Also, chordoma seems to go beneath the sacrum/coccyx, whereas this lump appears to exist only above. Any ideas what this could be? Could it be chordoma?
Thanks!
1,000,000 cases) and especially at my age, in my early 20s, most chordomas occurring in the 40-70 age group. Also, chordoma seems to go beneath the sacrum/coccyx, whereas this lump appears to exist only above. Any ideas what this could be? Could it be chordoma?
Thanks!
Are you currently on any medication like imatinib? The neutropenia can be due to your current medication. You can ask your hematologist for G-CSF injection if you have persistent neutropenia. Patients with CML are immunocompromised and can easily acquire infections. Neutropenia also puts a patient in an immunocompromised state.
For now, it is very important to prevent acquiring any infection. You should avoid crowded places and persons with ongoing infection.
Good luck.
1,000,000 cases) and especially at my age, in my early 20s, most chordomas occurring in the 40-70 age group. Also, chordoma seems to go beneath the sacrum/coccyx, whereas this lump appears to exist only above. Any ideas what this could be? Could it be chordoma?
Thanks!
Hi.
Chronic Myelogenous Leukemia (CML) is a blood disorder caused by an acquired genetic defect in the pleripotent stem cell. It has several phases: indolent chronic or stable phase, aggressive or advanced phase, and accelerated and blastic phase. The transition between phases may take years (on the average, 4 to 6 years from the stable phase to aggressive phase).
Average survival rates are generalizations, not applicable to a particular patient. The prognosis is relatively good. Newer techniques are being tried, including imatinib and stem cell transplantation.
All the best, and God Bless!
Thank you! No, nothing of note had shown up on any prior MRIs. I’d had clivus chordoma and the neuro surgeon didn’t get all of the tumor, but filled the ‘hole’ left with tissue from my leg because of the resection of the tumor. I’d had radiation and proton beam therapy to kill any of the tumor left behind.
sarcoma, chordoma, and teratoma The patient gives a history of bilateral oophorectomy. Therefore, an ovarian origin is considered unlikely.
There is a prominent mass effect seen upon the rectum. No definite fat plane is seen between this mass and the rectum. The remainder of the visualized portions of the bowel are unremarkable, without definite wall thickening or caliber abnormality. No definitely enlarged lymph nodes are seen.
TY for the reply. Yes I was told and am thinking that its parasitc or allergy. I went to aruba in June but that wouldn't have an affect on me now, would it?
Therapy with corticosteroids is used for the treatment of most and additional therapy is available and dependent upon the type of HS. Newer medicines such as imatinib and mepolizumab are said to be promising. So, should your son have HS, there is reason to be optimistic regarding a positive response to currently available therapy.
You should request, of his doctor, an additional explanation for the tentative diagnosis of pre-hypertension.
Good luck.
Patients with hypereosinophilic syndrome should be tested for the presence of the FIP1L1-PDGRFA-mutatition in order to identify patients that could benefit from a treatment with a tyrosine kinase inhibitor such as Imatinib. At present, immunosuppression is still the treatment of first choice for Churg-Strauss syndrome. Novel treatment modalities for both diseases include immunomodulation with interferon alpha and biologics such as antibodies against interleukin 5.
differential diagnostic considerations include chordoma, meningioma, primary ptuitary neoplasm, nasopharyngeal carcinoam with local invasion and/or mestastasis. Note, this lesion does not expand or invade into the posterior fossa or in case the basilar artery. However, it does partially enclircle the cavernous segment of the left internal carotid artery although the cavernous right ICA appears spared.
The cerebral hemispheres are normal in appearance and bilaterally symmetric.
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