The results of stenosis of each coronary artery and LVEF (heart failure) were correlated to carotid artery stenosis but no association was found to cerebrovascular parameters. Only severity of coronary artery disease and the Gensini score (a scoring system for evaluating collateral circulation of the coronary arteries) associated with disturbed cerebrovascular reactivity. Carotid artery stenosis was associated with decreased cerebrovascular parameters.
Faint opacification of the proximal renal artery is demonstrated. Features is suspicious of renal artery stenosis.
Simple cortical cysts are noted at left kidney.
No aggressive bony lesion.
Impression
1.8.0cm aortic arch aneurysm distal to the origin of left subclavian artery. Mixed hyper- and hypo-dense intra-mural density, suspicious of subacute intra-mural hematoma. No CT evidence of dissection or rupture.
2.
Statins are excellent but they are more likely to prevent disease progression in that same artery or other ones. they are less likely to cause actual regression of the stenosis. I would have to agree with you doctor that one should only intervene on severe stenosis (>80) or on lesser stensis (<80) only when symptoms are present.
It bothered me so told gp who said sounded like angina and sent me for CT of heart. This revealed mild stenosis in left descending artery and advised me to do on aspirin and statins which i have been taking since last july. I had CT with contrast in august which confirmed other CT and cardiologist put me on 5mg bisoprolol which i have been taking. I feel my throat tight/tense still when waling for a while - up, straight and downhill.
What tests are able to determine if there is renal artery stenosis? Is it via blood test or CT scan/ultrasound? Which medication would you consider increasing? In regards to the prograf, currently taking 2.5mg 2x daily. Almost one year post transplant. Very worried. As always, thank you for answering my questions.
The left main coronary artery (LMCA) reveals thin soft plaques with no significant stenosis and trifurcates into left anterior descending (LAD), ramus intermedius (RI) and left circumflex (LCx). The proximal LAD reveals calcified plaques causing 30% stenosis. Rest of the LAD reveals occasional plaques with no significant stenosis. The diagonal branches reveal no significant stenosis. Ramus reveal occasional calcified plaques with no significant stenosis and supplies the OM territory.
Normal
LAD : Type III LAD has 50% proximal stenosis followed by eccentric 95% stenosis before D1 and 90% - 95% long segment stenosis astride D2. Distal LAD exhibits TIMI - III flow D2 has 90% ostial stenosis
LCX : Non-dominant LCX has 70% proximal stenosis followed by 90% stenosis after OM1. Major OM1 is normal.
RCA : Dominant RCA has mid luminal irregularities in the proximal and mid segment.
Or was the test ECG-gated 16-slice CT scan angiogram of the whole chest showing coronary artery occlusion? It is not clear why there was CT scan angiogram and now stress test angiogram, but the CT scan was not very decisive according to the report.. Usually the CT scan is not used when there are symptoms of heart vessel occlusions, instead a cath. But now that a CT angiogram has shown occlusions, why would there be another test for occlusions.
Many heart attacks occur with the rupture of the inner lining (endothelium cells) of the artery due to plaque buildup between the inner and outer wall of the artery. A cath angiogram does not show the risk, but a CT scan 64 slice can/will evaluate the anatomy of vessels for the risk of a heart attack from that source (many more attacks from rupture than occluded vessels).
I am assuming the MI is ischemic (lack of blood flow to the heart).
The right artery is blocked however the angiogram and CT angiogram contradict each other. One says the blockage is at the top, the other says the blockage is in the middle. Personally, I would believe the angiography report because this has seen retrograde filling and it makes more sense. Your right artery then is blocked near the top, but the vessel is being fed by natural bypasses from the left side.
If it turns out that you have substantial compression of the artery, then medication is the first line of defence. Surgery is an option, but only usually if you suffer lots of symptoms which medication In some cases, where the bridge is thin and short, stents are used to hold the artery open. The options will be evaluated by the cardiologist who does the angiogram.
The calcium score is a calculation of the soft plaque that resides between the layers of the blood vessel. The significance of high score is the risk of rupture of the soft plaque into the blood flow channel and cause a heart attack. The score may represent degree of plaque in the left anterior artery
Left main artery with 20-30% narrowing with hard plaque is not considered significant asnd should not be any problem.
In May 08, I had an ultrasound and an ERCP and there was nothing abnormal. Blood tests are also normal. My Gastro has recommended that I have a CT Scan of my abdomen to rule out Diverticulosis and then thinks we should stop there as it doesn't seem to be bowel related. My worry is that I had RAI Uptake Scan and RAI 131 for Graves Disease in 2005. X-rays for gallbladder in 2001, a mammogram in 2007 and two other CT Scans over 10yrs ago.
A calcium score can help RISK stratify you for major cardiac events over the long term, but it DOES not tell you specifics about the extent of your current blockages. A CT angiogram or a cath angiogram can do that--cath is still the gold standard.
Recommendations for asymptomatic individuals based on their coronary calcium sore can be summarized as follows. Calcium scores of 0 identify individuals in the "lowest-risk" group with 10 year risk for cardiac events less than 5%.
The disease you have will not be cured, but you certainly have a high chance of stopping it getting any worse. Artery disease is all about inflammation of the artery, this is how it begins, then the immune system makes it worse. So you need to stop inflammation of the coronary arteries, that's the key. A lot of research has gone into this and once an artery is damaged, it inflames, then receptors call for repairs. This is when lipids carrying cholesterol get trapped in the damaged vessel.
It makes no sense that people would have to die trying to find out if they have fibromuscular dysplasia or renal artery stenosis or carotid artery stenosis. I am blocked in my left renal artery and they want to go straight to an angioplasty (which they need dye for) and my right renal is start to narrow. My left carotid has homogenous plaque and my right carotid is looped. There is no hope for me at this point since I can't have dye.
Dear Lilly Lou,
A CT scan of the chest, including the blood vessels, will still show the blood vessels downstream of a stenosis, unless the stenosis is so bad that it is preventing ANY blood from getting beyond it. There is no "dormancy" of a blood vessel. Without evaluating you, I cannot tell exactly what needs to be done for you specifically. However, I definitely would recommend that you have several opinions for what sounds like fairly severe disease at this point.
Also, you refer to process in discussion is a CT scan. It is a CT angiogram and involves the anatomy of vessels (cross section) and is as good as a cath angiogram and a CT scan (256 slice is 3D) is much more inclusive and can include respiratory system, the heart, aorta (ascending, arch, and descending), etc.
Renal artery stenosis is usually due to atherosclerosis due to aging. But there is a genetic risk of Renal artery stenosis. If untreated it can progress to renal failure. Surgical management is considered for >80% bilateral stenosis and stenosis in a solitary functioning kidney. This is correlated with blood creatinine levels to assess the renal function. So, based on the degree of stenosis and the renal functioning surgical therapy is planned and the outcome is good.
I was in the hospital recently due to vertigo issues. They ran a CT angiogram with contrast. The only thing listed on the radiology report was “minimal calcified plaque in the internal carotid arteries, no significant stenosis”. Does this mean I have heart disease. Also I read that if I have any plaque in my carotids then I most definitely have it in my coronary arteries and it is probably worse. Is this true?
(Left anterior descending coronary artery- There is a mild to moderate calcified and noncalcified plaque noted involving the proximal left anterior descending coronary artery, resulting in approximately 50% stenosis noted of the proximal left anterior descending coronary artery due to predominantly calcified focal atherosclerotic plaque. No additional stenosis are identified.)
I'm 48 and my LDL is 80 and HDL is 44.
Right Coronary Artery -
Dominant - Proximal segment has 90% stenosis.Mid segment is totally cut off.
Remaining RCA,PDA & PLV are visualized thro, ipsilateral collaterals from proximal RCA.
FINAL IMPRESSION - Diffuse Triple Vessel Disease
Advise -Early CABG.
This was the Report.
Medicine Prescribed by the doctor :
Atrovastatin Tablets IP 20 mg - CAAT 20
Nitrolong 2.6 -Nitroglycerin controlled Release Tablets 2.
The results were primary diagnoses IHD
Secondary diagnoses COPD, HDN, Cholestrolaemia,Gout, Osterarthritis, Urinary retention secondary to BPH,, Chest pain releived by GTN
Course in hospital Angioram organised revealed 80-90% stenosis of RCA (non-dominant) can any one explain what all of this means as we are very confused no one has said will he need a stent or bypass or can they do anything for his as he has been getting so much chest pain at rest and cant do much he is 74years old I would b
Recommended
