pacemaker cells of gi

i never felt better after receiving the pacemaker than before i got it. my wife was persistent with my doctor, after two weeks of complaing, she finally got his attention when she noted that my heart rate kept dropping innthe 30's during a medical procedure. she confronted the doctor. i was brought into the er, where they checked my pacemaker, one lead was off. when they went in to replace that lead they found the other lead off, they also found a black oily substance on my pacemaker.

This allows the pacemaker cells within the SA node to be in control of generating and pacing the heartbeat. But, if a particular group of cells in areas remote from the SA node become extra- or hyper-excitable they can take over as the pacemaker for one or several beats, or for a longer term. You might think of the heart's cells as a schoolyard full of young boys playing soccer. Some are inherently quick and fast and are always on top of the game, while others usually play a little slower.

Dehydration is by far the biggest killer with fever, and fluid loss can be counteracted by monitoring the patient and using IV to push more fluids in. With loss of fluids, cells will start to die, blood pressure will drop and this is not a good situation to be in. From reports I've read with brain cells, they may start to die at 42c. From the patients I've observed in intensive care, they keep the heart rate low, 70-80 to keep it from overworking over long periods of time.

hypokinesis can also be the result of stunned cells. Stunned heart cells affected by a heart attack can be revitalized if blood/oxygen is quickly provided within a few hours to the area affected by an acute heart attack. Akinesis is different from hypokinesis in that the heart cells affected are necrotic (dead). Stem cell therapy may be helpful for that condition. Five years ago I had hypokinesis of the left ventricle wall and a low EF.

Since my step-mother had been hospitalized a week before with GI bleeding due to long term (20 years) effects of Coumadin, my father is reluctant to keep on with the medication now he's at home. The doctor stated to my father that the Coumadin was related to the pacemaker insertion, but I couldn't think of any reason that clotting would be a danger. Incidently, the day my father was being discharged from the rehab nursing facility, staff drew him aside and said your wife just died.

The sinoatrial node (SA node) is a group of cells positioned on the wall of the right atrium, near the entrance of the superior vena cava. Cells in the SA node will naturally discharge at about 70-80 times per minute. Because the sinoatrial node is responsible for the rest of the heart's electrical activity, it is sometimes called the primary pacemaker, the natural pacemaker.

Usually, the heart muscle cells keep time more slowly than normal pacemaker cells. This allows the pacemaker cells within the SA node to be in control of generating and pacing the heartbeat. But, if a particular group of cells in areas remote from the SA node become extra- or hyper-excitable they can take over as the pacemaker for one or several beats, or for a longer term. You might think of the heart's cells as a schoolyard full of young boys playing soccer.

Usually, the heart muscle cells keep time more slowly than normal pacemaker cells. This allows the pacemaker cells within the SA node to be in control of generating and pacing the heartbeat. But, if a particular group of cells in areas remote from the SA node become extra- or hyper-excitable they can take over as the pacemaker for one or several beats, or for a longer term. You might think of the heart's cells as a schoolyard full of young boys playing soccer.

The sinus atrial node is a clump of cells in your heart that put out an electrical current that causes the rest of your cells in the atrial center of your heart to contract. The electrical current continues into the ventricular node and causes the bottom of the heart to contract immediately after that.

Yes, the condition can infect the heart's natural pacemaker. The heart's "natural" pacemaker is called the sinoatrial (SA) node or sinus node. It's a small mass of specialized cells in the top of the heart's right atrium (upper chamber). It makes the electrical impulses that cause your heart to beat.

If the cells are dead, a cath would not be of any help. If the cells are hibernating due to low blood flow to the area, medication can increase the blood flow to the area....angina with stress and rest normal blood flow. Your symptoms, if any, would be the issue to correct. If you are having chest pain (angina) with exertion and rest relieves the chest pain that would be stable angina and medication can be effective.

do you have an ICD/defrillator also?

I am 58 yrs old woman. Five years ago I had a sudden onset of acute ischemic colitis. No cardiac issues were present at that time. Approx. 6 mos. later I suffered an episode of syncope followed by a second bout of ischemia; at that time a heart arrythmia was diagnosed as well as a PFO. The PFO has not been closed. Neither my gastroenterologist nor my cardiologist can say with any certainty that the bouts of ischemia are directly related to the cardiac issue.

My understanding is that it takes 2 - 4 hours from the time food enters your stomach, until it leaves. I recently had an upper endoscopy (for other reasons) I hadn't eaten in 12 hours and the GI dr. said there was a lot of food in my stomach. So, I have gastroparesis and would like to talk to a knowledgable dr about a pacemaker for my stomach. The GI dr prescribed Reglan to help the stomach digest. I really don't think it is doing anything, although it has only been around a week.

s very worst is based in part on the fact that there are several sites in the stomach where the main pacemaker cells to trigger stomach contractions are found. In traditional chinese medicine, the deeper organs can be accessed by more peripheral local points on the arms, legs, and body - some of those points connect up with the stomach. So it's very worth a try. There are also chinese herbs that could be tried.

These were stem cells made from my OWN STEM CELLS not embryonic. So, no chance of rejection, no extra pills to take. There is over an 80% chance of improvement. My EF was between 12 and 15% before new stem cells. Have not had another EF test yet. I am diabetic(insulin dependent.) I do have a defibrillator and pacemaker. Would be only too happy to share my experiences - they will be on-going!! It is a shame one has to go out of the country to have this procedure done.

My Doc gave me meds two days ago it seems tobe working. dose anyone have an idear what could be causeing the vomiting? My upper GI has been cleared Small bowel has delayed diegustion.

Hi mcalderon91723, If this were me, one of the things I'd like to know would be what terminated the 7.5 second pause? My understadning is that every cell in your heart can become a pacemaker. It's nature's wonderful backup system in case the regular pacemaker signal doesn't reach the ventricles. The noraml pacemaker is a group of cells in the atrium called the "sino atrial node" or SA node.

I dont know if the lead can cause such effects, but hypokinesis is sometimes reversible. It depends on whether heart cells are dead or hibernating. It of course negatively influences ejection fraction (depending on position in heart), which can make you feel tired. In time, things still can improve. I understand your frustration.

He got a GI test done and he still had his food in his stomache 15 hours after eating, his colonoscopy showed that he had inflammation of his bowels... crohns is a possiblity. Any other options??

Having a heterogeneous liver would suggest that there are some irregular cells within the liver. The texture or composition of the cells in the liver would not be uniform. Heterogeneity in the liver mass may not necessarily be a serious problem in itself - it depends on the circumstances of the condition.

LBBB is very common, and as we age there is more chance of developing it. By the age of 80, 11% of people will have it. LBBB is quite common during angioplasty when the catheter tickles the right bundle branch, but things return to normal pretty quickly when the catheter is removed. If a patient suffers LBBB, a temporary pacemaker is usually fitted for the procedure. When working hard, RBBB seems to have little/no effect on performance, quite the opposite to LBBB.

If you have had negative biopsies but GI can see Barrets during Endoscopy, it might just mean that the cells that were taken during the biopsies were not Barrets cells as then it is possible for Pathologists to miss this. The protocol for Barrets surveillance (no dysplasia) is every 3 years. Only once there are signs of dysplasia (cancer stages) then you need to go for annual scopes and then it increases to 6 monthly if there are activity.

my father had a heart attack in 1984, a pacemaker four years ago andnow is diagnosed with heart failure, is this normal?

heart cells are probably trying to take up for the loss. However, not being the natural pacemaker center, these cells fire at inappropriate times causing what you are feeling as palps. As for what to do, I can only tell you what I would do. I would get the thing fixed or replaced ASAP.

Pacemaker cells of gi

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