Multiple sclerosis onset to death

Common Questions and Answers about Multiple sclerosis onset to death

multiple-sclerosis

382218 tn?1341181487 Factors predisposing to benign multiple sclerosis included younger age at onset, shorter disease duration and a lower number of relapses. We conclude that a substantial proportion of patients with multiple sclerosis from Crete follow a rather benign disease course, and this may relate to the genetic background of the population and/or to environmental factors.
Avatar m tn but the health is not improving, before admitting to the hospital she was walking, but now left leg is showing less sense and not able to walk, left hand also started having less sense. I donot know what to do. Blood sample test shows all the RBC, WBC etc., with in the limit the disease is curable, Please reply early, . Thanks and regards C. MADHAVAN from Chennai India.
152264 tn?1280354657 The disease, researchers now report, is the monkey equivalent of multiple sclerosis. And it appears to be caused by a virus – adding support to the possibility that multiple sclerosis in humans can be triggered by a viral infection. Experts say the discovery could help expedite the search for more effective treatments.
Avatar f tn http://www.medhelp.org/forums/Multiple-Sclerosis/show/41 Specifically, this Health Page describes the lesions found in MS: http://www.medhelp.org/health_pages/Multiple-Sclerosis/How-MRIs-Show-Lesions-in-MS/show/23?cid=36 Also, what a typical MS work up entails: http://www.medhelp.org/health_pages/Multiple-Sclerosis/Common-Blood-Test-Youll-See-During-the-Diagnosis-Process/show/446?cid=36 Good luck and keep us posted.
Avatar n tn The other possible cause could be multiple sclerosis and is a diagnosis of exclusion. The symptoms of multiple sclerosis are loss of balance, muscle spasms, numbness in any area, problems with walking and coordination, tremors in one or more arms and legs.
333672 tn?1273792789 We seem to have this discussion interminably and yet the types of MS still seem elusive. Just to satisfy my own curiosity, I got my hands on a copy of the article on which the current four clinical types are based: Defining the clinical course of multiple sclerosis: results of an international survey by Fred D. Lublin and Stephen C. Reingold for the National Multiple Sclerosis Society (USA) Advisory Committee on Clinical Trials of New Agents in Multiple Sclerosis. In Neurology.
Avatar f tn I was have episodes of confusion, blurred vision, dizzeness, no coordination and memory loss. My PCP sent me to a rhematoid specialist then a neurologist. The neurologist sent me to have an MRI done and another cognition/memory test the following week to rule out MS. I had already scheduled my follow-up test with the neurologist for a month later to review the test results. He was convinced nothing probably would be found since I could still walk in heels seriously?).
Avatar f tn LOMS is usually associated with a faster progression to disability compared to young adult multiple sclerosis (MS) patients. Moreover in patients over 50, MS variants and atypical forms which present a difficult diagnostic problem, may be frequently encountered.
198419 tn?1360242356 //www.medhelp.org/health_pages/Multiple-Sclerosis/Relapses--Pseudo-Relapses-and-Remissions/show/372?cid=36 http://www.medhelp.
Avatar f tn Approximately 50% of patients with RRMS convert to Secondary Progressive Multiple Sclerosis (SPMS) within 10 years of disease onset. After 30 years, this figure rises to 90%. At any one time, the Relapsing-Remitting form of the disease accounts around 55% of all people with multiple sclerosis. SPMS is characterised by a steady progression of clinical neurological damage with or without superimposed relapses and minor remissions and plateaux.
Avatar n tn Everything seemed minor until the last line, which raised the possibility of demyelinating process, including but not limited to entities such as multiple sclerosis. Please follow-up with your neurologist for further workup and/or evaluation.
559187 tn?1330782856 times of the year when relapses tend to occur. If we look back over tme to when our relapses tended to occur, there seems to be some pattern. For the past 5-6 years, my worst time of the year for health problems or something going wrong has always been in the April-June timeline. Maybe it is just a coincidence, but I am not so sure. I had intended on going into this problem period in 2010 being positive that this year will be different.
1221035 tn?1301000508 I was diagnosed with Multiple Sclerosis in Jan 2010. I have a long history of severe migraines. I was telling my neuro about periods of zoning out/blank stare and rubbing my thumbs together. He did a 20-30 min in office EEG that showed spikes in my left parietal lobe. I also have had many jerks, like when your whole body jumps when your cold, but it happens a lot to me.
1029680 tn?1286919361 And, as others have suggested, you probably want to watch what you eat to try to reduce gas-producing foods. It might take some experimentation to figure out what foods are contributing most to your problems. It might also be worthwhile to lookup all your medications on drugs.com, to see if any list intestinal issues or flatulence as side-effects. You have fewer options if your problem is caused by a medication you really need to take, but at least you'd know who to blame.
Avatar m tn ) I apologize for no one posting to you yet but I think they may be a bit confused as to your question? Are you wanting to know the symptoms of MS or wanting to post Your symptoms? There is some awesome information in the top right hand corner of the page Titled "Health Pages". There is a wealth of information there BUT....Please ask if you have any questions :) Hope to hear from you soon. Again, Welcome to the Forum!
Avatar f tn t think I have MS symptoms and this all started with me complaining about a bad headache to the back of my head which was determined to be caused by neck strain due to wearing a motorcycle helmet on a 3 day road trip. The headache has not returned but stranger yet was a interesting sensitivity to my scalp at the rear center top of my head. It lasted 6 days and was moderate in pain and hurt when touching my hair or trying to sleep on that side.
Avatar m tn You do have things we experience here all the time, and it will be very important for your Neuro to start a thorough work up to start to seeing what you do or do not have. You already have been to 3 Neuros, so what are they saying? Do you know what they worked you up for so far? I'm sorry you are going through this with seemingly no guidance or info. Have you had Brain only MRIs? Any imaging for you C-spine ordered? Where is the lesion located? Do you have copies of the reports?
Avatar f tn Hi Dee, We haven't met yet and I just wanted to welcome you to the forum. Although it seems you have been lurking for a while, I haven't seen you on the forum. Please feel free to ask anything you have questions about and we will try our best to answer your questions based on our personal knowledge since we aren't doctors, with the exception of Quix who is currently on an extended leave.
Avatar f tn I WAS PARALIZED IN 1999 IN LEFT LEG, TOOK 18 MONTHS TO BE ABLE TO WALK A LITTLE. ABOUT A YEAR LATER I GOT PARALIZED IN RIGHT LEG IT TOOK A SHORTER TIME FOR ME TO BE ABLE TO WALK AGAIN. I HAD BELL PALSY IN 2000. IN MY FACE. THEY PUT ME ON CAPAXONE INJECTIONS. THE NEW DOCTOR TOOK ME OFF OF MEDICINE. I HAVE BEE SO NERVOUS ABOUT ALL THIS. I'M NOW AN OLDER PERSON. WHAT COULD I HAVE IF NOT MS? I FELT CAPAXONE HELPED ME. WHAT DO I DO NOW?
Avatar m tn i had been diagonised with multiple sclerosis in the year 2001. now i am not able to walk. i am also not able to see clearly. my speech is also not clear. i think my type of ms is primary progressive since my condition has worsened gradually. pl suggest some medicines or other alternative therapy.
Avatar n tn do you have to have active lesions in the brain on MRI in order to have M.S. My husband was diagnosed with M.S. and is driving himself crazy denying it. He had an MRI of the brain that showed multiple foci of T2 and FLAIR hyperintensity in the white matter of both cerebral hemispheres including approximately 5 small lesions in the periventricular white matter, lovated at the callosal sptal margin and oriented perpendicular long axis of the lateral ventricles.