Graves disease and insulin resistance

Common Questions and Answers about Graves disease and insulin resistance

graves-disease

Avatar f tn I made an appt with an opthamologist who said I have no evidence of any eye disease at this time. I should also mention I was dx with insulin resistance a month ago. My questions are :1. Isn't it unusual to have neg TPO and TG in Graves? 2. Should I be worried that the lymph node present did not have well defined hilum-- how like is this cancer? 3.Does euthyroid Graves pose a significant risk to future pregnancies? And anything else you would recommend this point going forward. Thank you!
Avatar f tn In my case I was already insulin resistant (high insulin = fat storage and no fat burning) and hyperthyroidism worsened my insulin resistance and caused impaired glucose tolerance. There is an article from Natural Endocrine Solutions "Why Do Some People With Hyperthyroidism Experience Weight Gain" that has a lot of reasons why this can happen plus tips on how to lose weight.
Avatar f tn I had to stop eating refined sugar and get thyroid hormone levels back down so the insulin levels would drop. I reversed my insulin resistance and pre diabetes. I've lost 13 kg so far.
Avatar f tn I have been having difficulties quite sometime now and labs show normal but I feel terrible and eye doctor suspects graves disease (I never mentioned anything to him regarding thyroid issues). I also have insulin resistance and arthritis. Fatigue, bone pain, irritability and extreme stiffness after doing physical housework, walking and other activity are everyday symptoms I have learned to live with. Lab results were as follows: TPO AB 7 TSH 0.93 T3 Uptake 24 antithyroglobulin AB <1.
Avatar m tn It is impossible to store body fat without insulin. And once insulin is elevated you can't burn body fat. I had insulin resistance with chronically high insulin levels so I found that out the hard way. In fact, I didn't lose any body fat during my year of hyperthyroidism. Excerpt from the article Reverse Insulin Resistance in 4 Easy Steps... "Insulin resistance can be reversed. My patients do it all the time. Step 1. Stop Eating Dessert.
Avatar m tn I was hyperthyroid for a year and my poor appetite never improved and I lost no weight either on the scales (although I have muscle atrophy). The reason was due to severe insulin resistance which worsened even further due to excess thyroid hormone.
Avatar f tn Graves Disease is an Autoimmune Disease and usually when one autoimmune disease is found, there is usually a 2nd one found (this one being Diabetes). The Graves and Hyperthyroidism can be treated by either anti-thyroid meds or pernmanant treatment which will eventually stablaise the Diabetes although this is extremely hard if the Diabetes has been undetected for a long time. And something to watch for is ...Metformin can interefer with thyroid medications.
Avatar m tn Underlying glucose intolerance may be exacerbated in hyperthyroidism because overweight individuals display higher levels of insulin resistance by homeostasis modeling assessments of insulin resistance than do thinner counterparts.[20,23] In patients with Graves disease or toxic nodules, the level of elevation of T3 correlates directly to the degree of peripheral insulin resistance by homeostasis modeling assessments of insulin resistance.
Avatar n tn Ask for a full thyroid panel including TSH Free T3, Free T4 and Graves disease antibodies. I am an American and have experience with the UK system. This sounds EXACTLY like Graves / Hyperthyroid. I have graves and I had the EXACT list of symptoms. Go pay for an independent lab if you have to...the last time I was over there, I felt "off" and look into going to a private lab and think a full thyroid panel was about 150 pounds (the graves marker may be more).
1287446 tn?1313947638 I’ve done a lot of research online and when I read about insulin resistance, it seems to pinpoint nearly everything I’m experiencing. I’m not overweight, and I think that doctors tend to think you have to be overweight to have an issue (at least my current doctor does). I’m paranoid about becoming overweight and have always monitored calories, macronutrients, etc.
973741 tn?1342342773 Hi The answer comes in understanding about insulin resistance. Insulin resistance typically precedes diabetes by many years, maybe even decades. High insulin is inflammatory. This is what contributes to / causes heart disease. They also know that even slightly elevated blood glucose, still in the "prediabetic range" is associated with heart disease. How to address insulin resistance? Lifestyle for sure.
Avatar n tn 1. Why he is treating insulin resistance without diabetes so aggressively and 2. If you were that insulin resistant you wouldn't have been able to lose weight so easily that the doctor commented it was too fast. I would make sure the doctor explains to you what he is treating and why. I would absolutely want clarification (by A1C test) as to whether you have diabetes.
Avatar m tn Although insulin levels were never extraordinarily high, the increase between the 2nd and 3rd hours seems to be abnormal and indicative of delayed insulin response, a pattern associated with insulin resistance. Several years ago I had elevated cortisol throughout the day. Last year, is was below range for most of the day. It seems to have flip flopped, while DHEA has stayed consistently borderline high (but still in range) and testosterone is elevated.
Avatar m tn Ask for a 2 HR glucose tolerance test and have them check your fasting glucose, A1c, and your post drink insulin levels. That is how they will be able to test for insulin resistance. If you are not insulin resistant, it may be that your adrenals are the ones over producing male hormones in your body and not your ovaries, in that case your doctor will want to test the androgen levels produced by your adrenals and go from there.
315318 tn?1353251800 I read somewhere that fenofibrates reduce insulin resistance or increase insulin sensitivity. Is that what I am seeing ? Does anyone else have a similar experience ? Thanks.
Avatar m tn In studying the insulin resistance of 29 people with Hepatitis C, Australian researchers have confirmed that they have high insulin resistance, a precursor to diabetes. However, almost all insulin resistance was in muscle, with little or none in the liver, a very surprising finding given that Hepatitis C is a liver disease. Insulin, a hormone made by the pancreas, helps the body use glucose for energy. The two most important organs that respond to insulin are the liver and muscle.
Avatar f tn In summary, hepatitis C promotes insulin resistance and INSULIN RESISTANCE INDUCES INTERFERON RESISTANCE, steatosis and fibrosis progression. http://www.ncbi.nlm.nih.gov/pubmed/17131467?ordinalpos=14&itool=EntrezSystem2.PEntrez.Pubmed.Pubmed_ResultsPanel.Pubmed_RVDocSum "Other measures can be taken to enhance svr probabilities in the the face of negative predictors." Like what?
Avatar f tn I'll post my info over two answers as I have a lot to say lol. Insulin resistance is the most common cause of PCOS. A great article to check out... Treatment for 4 Types of PCOS. Treat the Cause by Lara Briden: http://www.acubalance.ca/blog/which-pcos-type-are-you I have had severe insulin resistance but no PCOS because of my poor appetite causing low calorie intake.
Avatar m tn Wouldn't abnormally high insulin levels be a better indication of insulin resistance? In which case diagnosis can be made a lot earlier? Like maybe when a person is still young and can make lifestyle changes easier than when they are already in their late 40s?
568322 tn?1370165440 Insulin resistance is the major feature of the metabolic syndrome and depends on insulin secretion and insulin sensitivity. In chronic hepatitis C, insulin resistance and type 2 diabetes mellitus are more often seen than in healthy controls or chronic hepatitis B patients. Hepatitis C virus (HCV) infection promotes insulin resistance, mainly by increased TNF production together with enhancement of suppressor of cytokine (SOC-3); both events block PI3K and Akt phosphorylation.