Digoxin stroke volume

Common Questions and Answers about Digoxin stroke volume

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Avatar f tn s electrical system and decreases the heart rate, but increases each stroke volume. Wikipedia has an excellent article for Digoxin, and describes how it works very clearly. If it were me, I'd opt for a modern drug targeted for your type of SVT.
Avatar f tn I am taking Digoxin for tachycardia--my cardiologist says that while there aren't good studies available for cardiac meds while pregnant Digoxin is believed to be safe. It is not a betablocker, however the dr felt the digoxin would be safer than a betablocker while preggo. Also the changes in your blood volume while pregnant may correct the tachycardia.
Avatar m tn Some of the drugs associated with this are blood pressure medications (especially beta-blockers), heart medications (such as digoxin), some peptic ulcer medications, sleeping pills, and antidepressants. Just check out the medications and if you are not taking any then all these causes ruled out. I sincerely hope that helps. Take care and regards.
Avatar f tn 5 % reference 54-74(LV end-diastolic volume/bsa (H) value 148 ml/m\s\2 reference 29-61(LV end-systolic volume/bsa (H) value 122ml/m\s2 reference 8-24(LV ejection fraction, 1-p A4C (L) value 16.3% reference 46-78(Stroke volume, 1-p A4C value 30 ml (Stroke volume/bsa, 1-pA4C value 16.1 ml/m/s\2(LV end-diastolic volume 2-p (h) value 189 ml reference 46-106(LV end systolic volume 2-p (H) value 158 ml reference 14-42(LV ejection fraction 2-p (L) value 16.
Avatar m tn CO is a product of blood volume per heart stroke and heart rate. The higher/better the stroke volume (weak, strong, stronger contractions) the fewer heart beats (more for weak, less for strong, least for stronger) are required to meet demand (held constant for illustration) for blood oxygen. A well-condition individual requires fewer heartbeats due to strong contractions. It is the stroke that determines EF...not the CO, as CO can vary by an increase or decrease of HR.
1124887 tn?1313754891 EF is a compensating factor and helps maintain a balancing flow of blood between the right and left side of the heart. Stroke volume is inverse to the heart rate. Slow heart rate the greater the stroke volume which indicates the heart rate is also a compensating factor...the slower the heart rate the more time to fill. Wide pulse pressure (no medication, etc) can be the outcome of increased CO due to fitness, anxiety, hyperthyroidism, etc.
Avatar m tn After a (small) stroke conjectured to have been caused by atrial febrillation (accompanied by a heart rate of 160 (3x her normal rate)), my wife had a pacemaker implanted and is now on coumidin, digoxin, and metoprolol. In a brief phone conversation with her doctor, she asked how long she'd be on the coumidin. He responded, "forever.
Avatar n tn The abnormals are mitral valve leaflets thickened, aortic vavlular sclerosis, diastolic transmitral e/a ratio decreased suggesting diastolic relaxation impairment, mild mr and mild tr present I am 39 with a history of tachycardia. I am currently taking digoxin 250mcg twice a day. Even with that my heart rate is still in the high 90's resting. I have low blood pressure, Digoxin has been the only medicine that hasn't dropped my blood pressure lower.
Avatar n tn i have been suffering from shortness of breath for 5 months.recently i have a echocardiogram. it shows that my stroke volume is 47 ml . is this is a normal value or it is a sign of a heart disease? my ef is 61%.
Avatar m tn This happens by a system of vasodilation and vasoconstriction coupled with the pumping rate and stroke volume of the heart. Rather than make frequent changes in volume, the body changes the configuration of the total container (all the vessels) and increases the speed at which the resource is renewed.
Avatar m tn I have visited the Cleveland Heart Clinic in 2007. I was put on coumadin because I had a small stroke after 12 hrs. of a-fib (prior to being put on sotalol). I no longer have a-fib on sotalol. I would like to get off coumadin and take 1 or 2 81 mg. aspirin instead. Do you think this would be prudent?
Avatar m tn How do you tell if Afib converted on its own or if the medication did the conversion or if the medication was causing the Afib? I have been to the Primary doctor and Emergency room both said I was not in afib currently. Another words, when do you know when to stop taking the medications? In Nov 2013, I have Aortic heart valve replacement and a maze procedure. My old cardiologist wanted to do a cardioversion but I didn't want it, so I had to change Cardiologist.
Avatar m tn It's been 2yrs since my stroke and I have no voluntary movement in my left arm or hand. Is there any possibilty that i could recover any movement? My neurlogist told me that if within 6month of the stroke if I didnt have any movement that my hand will stay paralyzed?
Avatar m tn A faster heartbeat does not provide enough time to fill as at rest, and the timing of contractions are less. The percent of blood pumped with each heartbeat is less reducing stroke volume (lower EF). The heart rate has increased to provide the necessary blood/oxygen to meet the system's demand for the diminished stroke volume. The CT scan indicates there is substanital soft plaque buildup within the linings of the vessels.
Avatar m tn The volume of blood left in a ventricle at the end of contraction is end-systolic volume. The difference between end-diastolic and end-systolic volumes is the stroke volume, the volume of blood ejected with each beat. Ejection fraction (Ef) is the fraction of the end-diastolic volume that is ejected with each beat; it is stroke volume divided by end-diastolic volume. Normal in a man is 50-68%.
Avatar f tn After spending 8 days in the hospital getting my heart back into sinus rhythm (3 cardioversions + Tikosyn) I am back to Afib (off the Tikosyn) and have a 'normal' heart rate of 100 now. I presently take Lisinopril, Digoxin, Warfarin, and Carvedilol. Is 'my present condition' truly something that should not cause concern to me. Isn't being in constant AFib causing my chances of a stroke to rise considerably???
Avatar n tn I took Digoxin for a number of years in reponse to an atrial fibrillation event. My new physician did not feel I needed to be on it any longer. What are the side effects of discontinuing this medication? I have been off it for several months and some odd changes in my body.
Avatar n tn s explanation where he states digoxin is used to treat irregular heartbeats. I was on digoxin for a few months following my congested heart failue event. It is my understanding the medication is used to increase contractility of the pumping chambers. My medication for stablizing heart rate is a beta blocker (coreg) and an ACE inhibitor. My research: "Digoxin is contraindicated in patients with ventricular fibrillation.
Avatar m tn For a perspective, diastole dysfunction compromises left ventricle filling volume, inadequate stroke volume and operating at a high pressure regimen. LV filling volume is reduced due to increased wall size crowding out available space. Inadequate stroke volume is usually due to wall thickening and stiffening and loss of elasticity. High pressures can dilate upper chamber to compensate for gradient pressure across the mitral valve. That is the pathology.
1591333 tn?1297733419 These mechanisms would be interrrelated. The low blood supply (volume in circulation) the kidneys will increase the volume (an example would blood loss, or a low cardiac output). The left venticle will dilate to increase blood flow with each stroke and the heart rate will increase the cardiac output (amount of blood pumped per minute). If the system is dysfunctional, medication, etc. can change the dynamics.
Avatar n tn My niece had an MRI recently and one of the findings said volume loss of the posterior fossa. What does that mean exactly? She has speech, vision, hearing and balance problems that we have never really had explained as to why she has them? Are they all releated to what they found on the MRI?
720516 tn?1230646225 d expect the stroke volume to be inverse to the Heart Rate - e.g. a well-conditioned person would have a low HR with a low BP but a high stroke volume with every beat. As far as poisoning, it'd seem natural for the system to go into high gear to remove the poison - but 4 months would sure be a long time for that, unless it created some semi-permanent change in you somehow.