Diabetic ketoacidosis blood sodium

Common Questions and Answers about Diabetic ketoacidosis blood sodium

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Avatar n tn html Anion gap Increased serum anion gap reflects the presence of unmeasured anions, as in uremia (phosphate, sulfate), diabetic ketoacidosis (acetoacetate, beta-hydroxybutyrate), shock, exercise-induced physiologic anaerobic glycolysis, fructose and phenformin administration (lactate), and poisoning by methanol (formate), ethylene glycol (oxalate), paraldehyde, and salicylates. Therapy with diuretics, penicillin, and carbenicillin may also elevate the anion gap.
Avatar n tn And this is interesting because it's been a long known fact that diabetic Ketoacidosis can develop into lactic acidosis if not treated promptly. The only forms of lactic acid in the body I'm aware of is that which is produced as the end result of glucose metabolism, and that which is delivered by Acidophillus (or B. Bacterium, Lactobacillus, etc).
Avatar m tn CT came back normal but they did some labs and her sodium was high so they decided to flush her blood and try to bring her out on today. Well today her sodium is down (still elevated but better) and I know that high sodium levels can cause the brain cells to be lacking water and therefore can cause confusion, but they are thinking it is low enough that this shouldn't be the issue now. They tried to bring her back out today, but she did the same thing.
Avatar m tn Diabetic acidosis (also called diabetic ketoacidosis and DKA) develops when substances known as ketone bodies, which are acidic, build up during uncontrolled type 1 diabetes Hyperchloremic acidosis results from excessive loss of sodium bicarbonate from the body, as can happen with severe diarrhea Lactic acidosis is a buildup of lactic acid.
Avatar n tn Other Tests more sensitive are Ketostix (more sensitive tablet version of same assay) and serum glucose measurement to confirm Diabetic ketoacidosis." Source:www.uchsc.edu/pathology/didactic/presentations/Urinalysis05-24-06.doc - Discuss this with your physician.At this point, I may suggest that you have your fasting blood glucose levels monitored. A repeat urinalysis may be done just to definitely establish the absence of protein and ketones.
Avatar n tn Thanks for the correction. I know that Diabetic ketoacidosis arises because of a lack of insulin in the body. The lack of insulin and corresponding elevation of glucagon leads to increased release of glucose by the liver (a process that is normally suppressed by insulin) from glycogen via glycogenolysis and also through gluconeogenesis. High glucose levels spill over into the urine, taking water and solutes (such as sodium and potassium) along with it in a process known as osmotic diuresis.
Avatar n tn They have done X-rays, an EKG, and various blood testing but have come up with nothing. He is also type 1 diabetic and ketoacidosis is negative. The dr tried singulair and claritin, but nothing is helping. He does not have any wheezing or cough, just the shortness of breath. I don't know if it is pulmonary, gastric, or neurologic problem. The dr will not refer him to another specialist and says to "ignore" that he has breathing difficulty!
Avatar n tn The most common incidence of fluid shifting caused by elevated insulin levels is in the treatment of diabetic ketoacidosis. While in the hyperglycemic phase, the potassium is pulled from the intracellular space to the serum and then excreted through the kidneys. The serum may indicate that potassium levels are elevated, though the stores are being lost with polyuria.
Avatar f tn diabetic ketoacidosis): Deep, rapid breathing Dry skin and mouth Flushed face Fruity breath odor Nausea or vomiting, inability to keep down fluids Stomach pain" Diabetes is diagnosed with the following blood tests: fasting blood glucose level, random (nonfasting) blood glucose level, oral glucose tolerance test, haemoglobin A1c test.
Avatar m tn Renal failure, acute and chronic Increased magnesium load (especially in presence of renal insufficiency) Magnesium-containing laxatives, antacids, or enemas Treatment of eclampsia (mother and infant) Diabetic ketoacidosis Increased renal magnesium reabsorption Hyperparathyroidism Familial hypocalciuric hypercalcemia Hypothyroidism Mineralocorticoid deficiency, adrenal insufficiency Symptoms and signs :Muscle and generalised weakness Decreased reflexes (Neuromuscular depression) Hypo