Vioxx and heart disease
Common Questions and Answers about Vioxx and heart disease
vioxx
I had a DVT that went through my heart and into my lungs(PE).
A valve was damaged, also one chanber of my heart.
My problem is that I wasn't diagnosed as a "heart attack", but rather a "pulmonary embolism", and the lawyers representing me in my vioxx lawsuit say it has to be a heart attack, not a PE.
Isn't a heart attack part of a PE? Rather, did I suffer a heart attack as well as a pulmonary embolism?
Recently, Vioxx has been removed from the market due to heart-related problems. Celebrex and Bextra are currently being investigated for similar heart-related toxicities.
It is recommended that people with liver disease avoid using all NSAIDs. If NSAIDs are medically required for the treatment of another medical disorder, a reduced dose should be used for a limited period of time and only by people with stable liver disease.
I have had three surgeries on my left eye (SLT, filtration, and then lazer cutting of filtration sutures to keep pressures low.), by a well known glaucoma specialist. After each surgery, my vitreous became 'looser' and more floaters erupted. Before each surgery, I asked the surgeon if the procedures would cause vitreous excitation, and each time he said no. Well, not so.
Recently, Vioxx has been removed from the market due to heart-related problems. Celebrex and Bextra are currently being investigated for similar heart-related toxicities.
It is recommended that people with liver disease avoid using all NSAIDs. If NSAIDs are medically required for the treatment of another medical disorder, a reduced dose should be used for a limited period of time and only by people with stable liver disease.
As independent studies started to show a correlation between Vioxx and an increase risk of heart attack and stroke, the NIH funded the VIGOR study I believe in 2001 at the request of the Arthritis Advisory Committee to determine if there was indeed a link. What they discovered was that all NSAIDS increased the risk for heart attack and stroke with Vioxx being the worst.
13 months ago I had a HA. 100%blockage of the ALD. Stent was inserted. Prior to this event I had low BP, Low cholesterol, don't smoke, exercise and no family history of heart disease. Ef was 20-25 after the HA. last Echo put EF at 35-40. Stopped taking plavix after one year, now taking 81 aspirin every other day. Stopped taking beta blocker at one year. Still taking 2.5 Ramipril twice a day.
My BP can run 95/65 & Heart rate 60 when resting.
Hi. I am a 59 yr old male. Symptons are always have been cold, tired, joint and muscle pain worse in fingers, I took Vioxx before it was removed , high blood pressure , tinnitus, completely deaf over 2,500 cycles in both ears. Hearing aids made no difference. Dentist last month couldn't freeze my gums with 2 needles of Carbocaine.
7mm nodule on thyroid. Blood tests TSH 2.15 ( range .465 to 4.68 ) FT4 11.17 ( range 10 to 28.2 ) Total T3 2.02 ( range .92 to 2.
When used within proper dosing limits Acetaminophen (Tylenol) is the only painkiller recommended for person suffering with advanced liver disease (cirrhosis). 1000-2000mg daily for compensated cirrhosis (CPT class A or B) and about half that for persons with decompensated cirrhrosis (CPT class C)."
Here are some links about this discussion.
http://www.medhelp.org/posts/Hepatitis-C/Ibuprofen-vs--acetminophen/show/86699
http://www.medhelp.
Same here! It has been 5 years and I had my GB removed laparoscopically after a HEPA (SP?) Scan revealed a "non functional" GB (no stones) as I was in alot of pain which appeared after a 2 week Master Cleanse fast (be careful folks as apparently fasts can cause a spike in cholesterol and GB distress). I have chronic pain on my right side under the edge of the ribs all the way to my back. It is driving me insane. Finally I am going to get a CT scan. Will let you know the results.
t stop the vics), that being aside, you should read the warning leaflet and decide for yourself if the side effects are worth it. I was given vioxx years ago, read the warnings about heart attacks and such, and threw them all in the trash! Not much later it was pulled from the market. If I lost a loved one, $$ isn't going to bring the back anyway, it's all stupid. If I do myself in, I guess I will come back and haunt the manufacturers....BOOOOOOOOOOOOOO...........
The structure of the heart and that includes wall dimensions, chamber size, valves, valve compliance, and heart functionality is evaluated and measured by ultra sound referred to as echocardiogram. A doppler test can be part of the echo examination and it color codes the blood flow through the heart and any backflow is easily identified (regurgitation).
The heart functionality is determined by the percentage of blood pumped with each heartbeat and referred to ejection fraction (EF).
There may be a different perspective for analysis of Marfin disease or a bicuspid aorta valve. These conditions are a tissue disorder and are more vulnerable and there should be no heavy lifting and absolute control of blood pressure.
Thanks for your question and if you have any further questions or comments you are welcome to respond. Take care, and I wish you well going forward.
The rightside vetricle of the heart when enlarged is usually due to pumping againist high resistence of the pulmonary artery and/or lungs. This condition is oftened due to pulmonary artery thrombosis (clot) and/or pulmonary emboli (clots). Thanks for your question, and if you have any further questions or comments you are invited to respond. Keep us informed.
on a Transthoracis Echocardiogram Test I have 3 valves Mitral, Pulmonary and Tricuspid regurgatating trivial and mild in the Tricuspid Valve. Does this mean I have Pulmonary Hypertention? I also had right atrium and right ventricular dilatation on a 2007 and 2008 report. This new report says decreased in size of the right atrium and right ventricular. Does any one knows what that means? How and why would it decreased in size and why would it be dilated in the first place.
Look at the millions of people for example who have had a heart attack and some heart muscle has died and turned into scar tissue, their collaterals didn't open. My main left artery completely blocked, and yet I didn't know until years later when another vessel blocked and had an angiogram. I didn't know because lots of collaterals had opened and kept me alive.
I would just ask 2 questions because there are no guarantees here.
1. Did they use drug eluting stents or just plain ones.
After all that has been said I can also just confirm: familial cholestorol is the primary reason for CAD's (coronary artery disease) and other heart related problems. My mum has CAD since her early 30's and Ischemic heart disease she is diabetic and she has Porphyria but she smoked all her life (quit 10 yrs ago, but the damage was already done), she is obese and unhealthy diet.
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