Primary progressive multiple sclerosis life expectancy

Common Questions and Answers about Primary progressive multiple sclerosis life expectancy


Primary-progressive, secondary-progressive, and progressive-relapsing are sometimes lumped together and called chronic <span style = 'background-color: #dae8f4'>progressive</span> multiple sclerosis. Benign Types of Multiple Sclerosis In addition to the four main patterns of ms, 20 percent of the multiple sclerosis population has a benign form of the disease in which symptoms show little or no progression after the initial attack. Patients with benign ms remain fully functional.
- In 1936, only 8% of patients were reported to survive beyond 20 years after onset of illness - In 1961, over 80% of Multiple Sclerosis patients were reported surviving to 20 years after onset of illness - 2002 – A patient with Multiple Sclerosis can expect to live to average population <span style = 'background-color: #dae8f4'>life</span>-<span style = 'background-color: #dae8f4'>expectancy</span> minus seven years (mean <span style = 'background-color: #dae8f4'>life</span> <span style = 'background-color: #dae8f4'>expectancy</span> - 7 years)"
No I do not like having Cancer but compared to a lot of people I have a good <span style = 'background-color: #dae8f4'>life</span>. I also have a rare form of Multiple Sclerosis which can paralize me and I am BRCA 1 positive with an 87% chance of breast cancer. If I get breast cancer the chances are it will be triple negative a harder to treat form of breast cancer. I was supposed to have a double mastectomy. I did not because I think they are coming up with new treatments for Cancer all the time.
Relapsing/Remitting ms (RRms) Secondary <span style = 'background-color: #dae8f4'>progressive</span> ms (SPms) Primary <span style = 'background-color: #dae8f4'>progressive</span> ms (PPms) <span style = 'background-color: #dae8f4'>progressive</span> Relapsing ms (PRms) Rather than describe each one, it would be easier if you can share your sister's diagnosed type.
(So, if h2s is the culprit, I should be low on sulfate in the urine b/c I'm not metabolizing my own...I think that would be the case.) -The primary location for the oxidation reaction is the liver (Deficient FMO3 enzyme in the liver = TMAU, similarity). -(p.106) There are no known methods for reducing the body burden of h2s, although reducing the intake of sulfhydryl-containing amino acides has been shown to reduce endogenous production.
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