Pancreatitis in renal transplant
Common Questions and Answers about Pancreatitis in renal transplant
pancreatitis
many things could have caused it--renal dysfunction, infection, pancreatitis, hemolytic anemia, dehydration or cancer.
We concluded that rituximab therapy is effective in cryoglobulin-related renal dysfunction in renal transplant patients but, due to chronic immunosuppression, this may be achieved at the expense of infectious complications.
AD Multiorgan Transplant Unit, University Hospital, CHU Rangueil, 1 avenue Jean Poulhes, TSA 50032, 31059 Toulouse cedex 9, France.
Hello Dr.
Blood pressure had been well controlled in the past with Norvasc 10mg 1x daily and Metroprolol 25mg 2x daily. However, most recently, experiencing high blood pressure since the Nephrologist switched the Norvasc to Lorsartan 100mg 1x daily. Readings have been in the mid 160's even in the 170's. In addition, the Metroprolol was discontinued (it wasn't really helping the heart palpations) and have been prescribed Atenolol 25mg 1x daily for the heart palpitations.
[73] found that there was no higher incidence of acute renal failure using sevoflurane among transplant recipients, although the drug is renally excreted. They suggest that in liver transplant anaesthesia, sevoflurane is as safe as propofol with respect to renal and hepatic function.
With regard to living donors undergoing partial hepatectomy, Ko et al. studied 64 adult donors and compared desflurane with isoflurane anaesthesia.
An acute pancreatitis in my experience does not normally cause ascites. FIP is a possibility but I would be more interested in looking at causes of a low serum protein. Low protein causes a buildup of fluid in the abdomen and is often associated with a condition called exocrine pancreatic insufficiency. Protein could be lost via the intestine (protein losing enteropathy) or in the urine (protein losing nephropathy).
The pathophysiological hallmark is severe renal vasoconstriction, resulting from complex changes in splanchnic and general circulations as well as systemic and renal vasoconstrictors and vasodilators. Rapid diagnosis and management are important, since recent treatment modalities including vasoconstrictor therapy can improve short-term outcome and buy time for liver transplantation, which can result in complete recovery.”
https://www.ncbi.nlm.nih.
jenna, if you post in the Transplant Forum you will be able to talk with at least two people that have had kidney transplants. One needed a transplant because of Lupus, I think the other one had Hep.C.
My husband had U/S last week and they found one septated cyst in his R kidney. He has mild symptoms of burning inside of his R quadrant . I am wondering if this has to be look after immediately....the recomendations were to check again in 3-6 months time with another U/S. Should I be worrying?
Thank you, Sunes.
second transplants that occur more than 30 days after a first transplant unequivocally carry greater morbidity and mortality due to adhesions, renal dysfunction and a myriad of other issues. The 1 year survival approximates 70% rather than 90-95% for an initial liver transplant.
When the renal function is below the optimal level then dialysis is initiated. At the end stage of renal failure renal transplant from a competent donor would be the therapy of choice.
Hope this helped and do keep us posted.
I had a liver transplant 15 years ago, been doing good until lately, yellowing, and now they say I have kidney disease..anyone know what this means? I would appreciate a reply asap...
My mother in her 50s has gotten a few blood tests done recently, that showed GGT varying from the 60s to 100s. A blood test right before this rise showed normal GGT but high serum creatine (it was high enough that if it was used to estimate glomerular filtration, it would have indicated renal failure). Since creatine was so high, she got another test right after to make sure, and the levels returned to completely normal but her GGT levels were elevated.
I would still question the advisability of steroids in an HCV setting. I know that the trend in liver transplant patients with HCV is to wean them off steroids soon post transplant and in more and more transplant facilities steroids are not being administered at all because HCV damage accelerates in patients who take steroids.
The most definitive test of the condition of the liver is liver biopsy where a sample of the liver tissue is removed and examined with state of the art equipment.
currently there is no data on its use in the post-liver transplant setting. i would be extremely cautious about using it until more data is available especially since its purported effects on tacrolimus and cyclosporine metabolism are extreme. i am quite confident that there will be clinical trials coming down the road extremely soon.
Im wondering what causes that especially in pancreatitis? I loss alot of weight a year ago, was real sick with abnormal bruising and weakness, falling down, ect, this was before pancreas started acting up again, prior to that I had only one acute episode of pancreatitis. Now its been basically non-stop, confirmed by mri, that it is chronic pancreatitis, and fatty liver.
she was diagnose with renal failure since last year.but since we live in philippines. vets here have no knowledge at all in dialysis nor kidney transplant. it really gets me on nerves because she is getting worst day by day. i hope someone will be willing to help me out by giving me a piece of advice of what to do.
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