Morphine in acute mi

Common Questions and Answers about Morphine in acute mi

avinza

Avatar f tn Complained all thru the night , given morphine 3 x with no relief. Following morning taken back found 2 of the 3 stents occluded causing acute MI , anterial wall injury with no flow phenomenon . Now have chronic heart failure with no quality of life : What could have caused the stents thrombosis in the distal side branch of lad ? Would it have made a difference if they had realized sooner than 9 hrs that I was having acute MI.
Avatar m tn Hi mi mi. LuvD is right. Do this under the strict supervision of your cardiologist. All the meds and changes can put a lot of stress on your heart. Sounds like you are going up the ladder in potency of drugs, and Im not sure I fully understand your situation. Hydro, oxy and dilaudid are potent, morphine is very potent and sub is more potent that. Why are you going on subs? I strongly suggest you don't. Id rather see you on the morphine. Can you tell us a little more of your story?
Avatar m tn The proper term is Myocardial Infarction (MI for short) and there is MI and there is Acute MI. Normal MI can be caused by a restriction of around 95% or more. There isn't enough oxygen to supply the heart muscle in a particular area, and the cells become damaged. With such cases, it is very common for heart muscle to fully recover from the damage, because they didn't reach the necrotic (dead) stage.
Avatar m tn I am currently taking plavix, 81mg aspirin AND coumadin on a daily basis. I had a stent put in in July 2008 and triple by pass in 2006. I'm 64 and had my first MI at 33. My father died of an MI at 46, my EF is under 30%. My EP (at one hospital) just suggested I eliminate the plavix while my CHF specialist (at a nearby major research university) said I would be on plavix for a "long time" (forever?).
Avatar m tn Additionally, determining the levels of cardiac markers in the laboratory - like many other lab measurements - takes substantial time. Cardiac markers are therefore not useful in diagnosing a myocardial infarction in the acute phase. The clinical presentation and results from an ECG are more appropriate in the acute situation. Quick summary of Cardiac Enzymes Troponin is released during MI from the cytosolic pool of the myocytes.
768044 tn?1294223436 In regards to your poll, my first line of defense when I have a migraine is Fiorinal C1/2, or Tylenol #3, but the Tylenol upsets my stomach. If neither work I will take either Morphine or Demerol, but this is only in severe cases in which I didnt catch the migraine at the beginning.
Avatar m tn m no doctor) frequent VPCs (PVCs) are a worrying finding in the setting of acute MI. The question is if your heart is damaged from your MI, or if it has recovered well. In the setting of structural heart disease, PVCs can trigger a sustained tachycardia due to what is called a "re-entry phenomena" causing the PVC to "loop" and fire several PVCs in a row/salvo, possible triggering more dangerous arrhythmias.
Avatar f tn I was diagnosed with an mild fixed perfusion at the cardiac apex wall near the apex and a infarct involving the apex and anterior wall near apex and my left ventricular ejection fraction is calculated to be 56%. My question is what does this mean in laymen terms and what medication, treatments or diet may help this condition?
Avatar m tn Pre cath diagnosis: CAD - ACS, Anterior wall MI, Acute LVF, HTN, DM Type-2, Severe LV Systolic Dysfunction LMCA: Normal LAD: Type 3 vessel, mid total cut off with retrograde filling, Diagonal - 60% disease LCX: Non-dominant, distal 70% stenosis. OM1 - Mild disease, OM2 - 70% Disease RCA: Dominant system, mid 70% stenosis.
Avatar n tn To support myocardial infarction (heart attack and damaged heart cells) there is marked ST elevation in the same area is consistent with a recent MI. If it persists and is present in an older infarction, it is associated with a wall motion abnormality or an aneurysm. Also, Lateral infarcts are associated with diagnostic Q waves in at least 2 of the lateral leads, I,AVL, V4,5,6.
Avatar n tn the extent of myocardial injury, the age of the infarct, its location, the presence of conduction defects, the presence of previous infarcts or acute pericarditis, changes in electrolyte concentrations, and the administration of cardioactive drugs. Nevertheless, serial standard 12-lead EKGs remain a clinically useful method for the detection and localization of MI. With an infarction changes in the QRS-complex are seen. These are manifestations of myocardial necrosis.
Avatar m tn t find anything immediately life threatening -- they do no further follow up - just pump me full of morphine (nitro has no effect in pain reduction) and send me home. Even when I have taken my complete set of detailed records and explained all previous treatments - they do the same old thing. I no longer go to the emergency room - can't afford the cost - and no longer see the point -- especially with no diagnosis - and I haven't died yet LOL.
Avatar n tn So a 100% blockage of a coronary artery may or may not result in the same tissue damage as an MI.
Avatar f tn I am a chronic pain patient and on DSHS. The only meds for pain covered by this are Morphine and Methadone (as far as the "Big Dogs" go). I take 4 30mg Morphines per day and my pain isn't even touched. Morphine has never worked for me - had a Hysterectomy 7 years ago - Morphine pump didn't work 4 pain and was switched to Dilaudid, which worked. Problem is that my Dr.
Avatar n tn Hello, Many disorders produce chest pain or discomfort. Some (e.g., acute MI, unstable angina, thoracic aortic dissection, tension pneumothorax, esophageal rupture, pulmonary embolism) are immediately life threatening. Some (e.g., angina pectoris, pericarditis, myocarditis, pneumothorax, pneumonia, pancreatitis, various thoracic malignancies) are potentially life threatening.
Avatar f tn To answer your question regarding prognosis may depend on the underlying cause. In about 90% of acute pericarditis, the underlying cause is unknown. Other common causes include infection, kidney failure, heart attack (MI). There can be a malignancy, damage from radiation, and direct injury. Thanks for your question, and if you have any further questions or comments you are welcome to respond.
Avatar m tn Hi I am 34 Year old , I had an Acute inferior MI on May - 4 at around 8.00 pm last and there was complete blockage in RCA, on 5th May 2007 around 1.00 am my Echo was done and my ECHO showed LVEF = 35% with LA Dilated, I later got a stent implanted in my RCA. I want to know if this LVEF = 35% and LA dilated was due to MI and if LVEF is improved after stent is implanted or if any way by which it can be improved.
254714 tn?1316613355 It sounds like you have either suffered a heart attack or a infection has damaged an area of heart tissue. You have a lot of symptoms and it doesn't necessarily mean there is a blockage. It could be an electrical signal problem requiring a pacemaker. You must have had an EKG? do you know what it showed? Usually with high troponin and chest pains, you are having an angiogram within the hour to investigate. Something is telling your cardiologist that this is not a blockage problem.
Avatar n tn Opana is just morphine....when i withdrew from morphine I had vomiting (enough to let myself get dehydrated) ....... and severe abdominal cramps. Use a heating pad or electric blanket for muscle aches and cramps - - drink plenty of good fluids - avoid caffeine and citrus - warm flat 7-up is good and so is white grape juice. Hot showers are a good thing. Look at the Health Pages - lwr rt hand side of page - for the Thomas Recipe and the Amino Acid Protocols .... they are both good.