Heart disease risk increased
Common Questions and Answers about Heart disease risk increased
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Low levels of high-density lipoprotein cholesterol (HDL-C) and raised triglycerides, affecting millions of patients worldwide, are strongly linked to significantly increased risk of coronary heart disease (CHD) even in patients who achieve or surpass current low density lipoprotein cholesterol (LDL-C) targets.
Research has shown that people with kidney abnormalities have a dramatically increased risk of heart disease. But by studying blood samples from patients with early-stage kidney disease, researchers at The Rockefeller University Hospital hope to better define the connection.
A new pilot study, launched last month, involves adults diagnosed with kidney disease and focuses on a condition called endotoxemia, a buildup of toxins in the blood that may affect cardiovascular health.
Five years ago I had a dilated left ventricle due to occluded coronary vessels. The heart became overworked and increased in size. Medication that reduced the heart's workload provided the time necessary to recover (about a year), and currently the heart is normal size.
If there is heart muscle is damaged (virus, alcohol, medication, congenital, etc.) , the outlook is not favorable and treatment would be to slow any progression, if any.
Those with mitral vale problems and those who have had the valve repelaced are at increased risk of clot formation. Hence, they need to be on blood thinners. With time, most need a change in dose.
Normally patients on blood thinners are kept at an international normalized ratio (INR) of 2.0 to 3.0. If the INR is low or near 1.00, this means the blood is pretty thick and likely to cause clot formation.
The diminished blood flow can injure the heart cells impairing heart wall movement, the heart will enlarge in an attempt to compensate, and untreated will cause the heart to eventually fail.
Medication will/should dilate the vessels enabling the heart to supply an adequate supply of blood to the heart, the heart's workload is reduced by the less resistance of unconstricted vessels. Control risk factors where possible as stated, etc.
They did the echocardiogram of my heart and the last result was increased bulk in the heart. After all those results and diagnosis I have mentioned above, my doctor did not give me any follow up visit after the hospitalization and the stress test last week. I assume he thinks I am ok .... or he just doesn't care :0). My dad died of heart attack and my mother has cardiomyopathy. I am 30 years old 5ft even and 96the lbs.
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More recently, several studies seemed to suggest that eating diets high in saturated fat did not raise the risk of heart disease—a finding that ran counter to decades of dietary advice. (21,22) One highly-publicized report analyzed the findings of 21 studies that followed 350,000 people for up to 23 years. Investigators looked at the relationship between saturated fat intake and coronary heart disease (CHD), stroke, and cardiovascular disease (CVD).
I have had CHF but medication has increased the the contractility of the pumping chamber, and the heart is functioning normally so I don't have any limitations with exertion.
You have a respiratory conditon that may effect your heart's ability to provide or compromise an adequate supply of blood oxygen when the system's demand increases with exertion. You should get some form of exercise, but it is difficult to know unless there is an examination. Your doctor should advise.
A calcium score can help RISK stratify you for major cardiac events over the long term, but it DOES not tell you specifics about the extent of your current blockages. A CT angiogram or a cath angiogram can do that--cath is still the gold standard.
Recommendations for asymptomatic individuals based on their coronary calcium sore can be summarized as follows. Calcium scores of 0 identify individuals in the "lowest-risk" group with 10 year risk for cardiac events less than 5%.
1) In people with no significant underlying heart disease, PVCs do not indicate an increased risk. 2) In patients with significant heart disease, the presence of PVCs suggests that an already increased risk may be even higher. 3) Suppressing the PVCs with antiarrhythmic drugs does not reduce risk, and actually tends to increase the risk.
Exercise capacity and heart rate recovery is a stronger predictor of risk than the severity of coronary disease, it is also a very good predictor of people at low risk who do not need aggressive treatment. Slow heart rate recovery is 12 beats or less per minute, and it is the first minute reaction time after a stress test that is the usual measurement.
Thank you for your time. I know that my LAD has no calcification. The number was zero. Your number is extremely high. I know that I have plaque halfway down the abdominal aorta vessel. I don't think that is consider heart disease maybe aorta disease. (not sure) Once again thank you for your time.
I assume you are being treated for high blood pressure, and patients with CAD and very low BP can increase the risk for a stroke and heart attack. There is a study that 110 or lower for systolic pressure and 60 or lower for diastolic pressure may increase the risk. There are other doctors who want the blood pressure as low as possible without side effects such as fainting, dizziness, etc.
The significance is if and when the heart chamber size is pathologically increased, the size can begin to lose its contractility and fail to pump adequately. The right side pumps blood to the lungs to be oxygenated, and if there is a loss of contractility the less blood supplied to the lungs reduces the amount of blood received to be pumped into circulation and the cardivascular system will begin to fail.
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