Glucokinase pancreatic beta cells

Common Questions and Answers about Glucokinase pancreatic beta cells

glucose

Avatar m tn the article about generating liver tissue is definitely news. i only recall learning of generating abnormal beta pancreatic cells from liver stem cells from lecture last semester. amazing how fast science moves.
250084 tn?1303307435 Oops, one more thing. Have you ever heard of inserting pancreatic beta cells into the liver at time of TP? I’ve heard this can be an alternative to full pancreatic TP- the islet cells adapt to the liver environment somehow? I seem to remember reading about this, but can’t remember where it came up.
5536886 tn?1455827346 I was just reading up on pancreatic cell transplant- apparently there was a patient in Italy that received a transplant of pancreatic cells, and unlike previous attempts, they have seen her insulin free for about 15 months. They created something called a bio-hub, here's a section of the article that talks about it: "They were able to do this by creating a mini organ called a bio-hub.
Avatar n tn According to the Mayo Clinic web site: "Pancreatic cysts are abnormal, sac-like pockets of fluid on or within your pancreas. Though it may be alarming to learn you have a pancreatic cyst, the good news is that most pancreatic cysts aren't cancerous — and many don't even cause symptoms. In fact, many pancreatic cysts aren't technically cysts at all. Called pseudocysts, these pockets of fluids aren't lined with the type of cells found in true cysts.
Avatar n tn When a person has type 1 diabetes their pancreas no longer makes insulin because the body for whatever reason attacked its own cells (beta cells). This is called an autoimmune response. This is why diabetes is an autoimmune disease. Transplanting the islet cells will enable your body to produce insulin again and hopefully be the cure.
6452556 tn?1381422100 Glyburide is in a class of medications called sulfonylureas which stimulate beta cells within the pancreas. The beta cells are responsible for responding to spikes or drops in glucose. When you eat a meal, your blood glucose will increase which activates your beta cells to release insulin to then lower your blood glucose and vice versa. When you have diabetes your beta cells may lose some or all of their ability to tightly control blood glucose.
Avatar f tn A pancreatic stone is not cancer. A pancreatic stone is a stone-like crystal formed from enzymes, proteins and salts that normally are used for digestion, like a gallstone or kidneystones. Cancer are alive cells that just grow where they shouldnt and never stop. The stone probably blocks the flux of the enzymes into the intestine, and that hurts. It can even hurt more, once the tissue gets inflamed and the enzymes digest your pancreas instead of the food in your intestine.
Avatar m tn Ultrasound, X-ray unremarkable, amylase high, but not too much (140) otherwise blood normal. She had some white cells, protein and white cells in urine. Normal stools, though occasional constipation. She does not smoke or drink. She has only vomitted once on empty stomach having not eaten much for a week. She has lost 16lbs in weight. GI Endoscopy showed reflux in stomach.
Avatar n tn --- dark urine sign of jaundice -- poss symptomatic back pain -- possible enlarged head of pancreas -- terrible history of colon cancer in her family, which I understand can be a genetic cousin to pancreatic genetic proclivity -- one episode of vomiting a few weeks back Signs that are encouraging: -- pain was not in the center of abdomen and back -- no yellowing or signs of jaundice besides dark urine -- no bilirem (sp) in blood test -- no tumor/mass noted in ultra sound -- no ultras
798555 tn?1292787551 Good read LM. Another ongoing 'diabetes cure' research is 'pancreatic islet cell transplant' http://diabetes.niddk.nih.gov/dm/pubs/pancreaticislet/ For those unaware of what islet cells do follow this rather brief synopsis. Insulin is a hormone that helps the body use glucose for energy. Diabetes develops when the body doesn't make enough insulin, cannot use insulin properly, or both, causing glucose to build up in the blood. Islets within the pancreas contain ßeta cells.
Avatar m tn they first said that it was cancer and told her she had 6 to 12 months to live, but after 2 fine needle biopsy,s and 3 ultrasound scans they cannot find any signs of cancer cells. but they still think it could be cancer and the cells are that small they could be missing them. my worry,s are that it is cancer and the longer they leave her it will be to late to treat... why cant they just start the treatment to shrink it. this is really worrying because they just seem to be leaving her.
Avatar n tn Hello again, Good question.... Visit the link below and you can search for more information using the term Edmonton protocol. http://www.expressnews.ualberta.ca/article.cfm?id=6354 This procedure has not been perfected and from what I understand, patients who have undergone this procedure have to take a lot of immunosupressive drugs that could have possible side effects.
Avatar n tn Bad news first. Type 1 Diabetes is is the result of an autoimmune attack on beta cells in the pancreas. Once beta cells have been destroyed, there is no way of getting them back again. And by the time symptoms manifest and keytones appear in the urine, 80% of beta cells have already been destroyed. So it is not possible to "grow out" of T1 Diabetes.
Avatar m tn Having LDH and all other pancreatic enzymes back to normal, plus all pancreatic imaging beeing normal, excludes any severe pancreatic disease. Even, if it was pancreatitis, it was slight, and has gone until now. No cancer from these results. Doctor ordered another CT; maybe he thinks there are small metastases from some extra-abdominal cancer. May be a pancreatic (and liver) scintigraphy would show more. 3.
Avatar n tn When the liver starts to back up with toxins does it cause enlarged spleen, pancreatic bile duct enlargement, duodenum widened? Which it eventually backs up to the esophagus causing vessels to burst. I've been told I have enlarged spleen , all the stuff I listed above. They try to check each organ but never issue the liver and hep c which I think are creating the problem. I now have fatty tissue on the pancreas from 5 years ago and has gotten larger at last ct scan.
Avatar f tn This was my first IVF cycle. 3 blastocysts were transferred ( one Grade 4 8-cells blast, two 6-cells Grade 3+ blasts). I just received my BETA result: 5.8!!! The nurse told me to come back for another BETA in 3 days to see if it's doubled, but she didn't sound very optimistic. My questions are: Has anyone else receive a low BETA, but continued to have a healthy pregnancy? Is it possible that my low BETA could be a result of late implantation? Or is this a chemical pregnancy?
Avatar n tn It often spreads to the liver fast - a biopsy of a liver tumor in my brother just hours after his ultrasound produced pancreatic cancer metasticised cells. The test that prompted the ultrasound was a severely elevated pancreatic enzyme level - amylase and lipase and specifically acetyl[??-something axxx] phosphatase level were in the 10s of thousands. Pancreatic burning pain is higher-or at least it was with my brother.
Avatar m tn Typically the pancreatic lipase increases, but not to dangerous levels. It is unclear if beta cells are effected also. At longer use podocyte function needs to be carefully monitored following urine microalbumin levels.
Avatar n tn However, my Amylase continues to be elevated and I am concerned, as is my doctor, about chronic pancreatitis leading to pancreatic cancer. I do not drink Alcohol and have not for 20 years. Can you offer any ideas for cause of the pancreatic enzyme elevation and treatments?
Avatar n tn ve found a number of articles related to new onset diabetes and the presence of pancreatic cancer. Is this something I should push for? I have had discomfort up under the ribs and am being treated for H. pylori. Advice appreciated.
1126279 tn?1311895042 Hopefully if/when a pancreas is removed they take what is called islet cells from the pancreas and implant them into your liver....and that theoretically tricks the liver into producing the enzymes like the pancreas does.....but the success of this varies greatly with each individual. If it doesn't work, you will be on enzymes for the rest of your life and also be an unstable diabetic! Definately something to talk about with a specialist who has done these procedures before....
Avatar f tn Hyperkalemia secondary to beta-adrenergic receptor blockade can occur in 1% to 5% of patients and is more likely to occur in non-cardio-selective beta-blockers versus cardio-selective beta-blockers. Underlying cause can be excessive potassium intake, disturbed cellular uptake of potassium, or impaired renal excretion of potassium. My system is periodically tested to monitor renal excretion of potassium as well as liver functionality.
Avatar f tn During normal digestion, the inactivated pancreatic enzymes move through ducts in your pancreas and travel to the small intestine, where the enzymes become activated and help with digestion. In pancreatitis, the enzymes become activated while still in the pancreas. This causes the enzymes to irritate the cells of your pancreas, causing inflammation and the signs and symptoms associated with pancreatitis.
Avatar n tn Other causes are irritable bowel syndrome, celiac disease, crohn’s disease, malabsorption syndromes, pancreatic diseases, diabetes, hyperthyroidism etc. Abnormal liver function too can cause this problem. A number of times a gastroenterologist is able to diagnose these conditions by clinical examination and detailed medical history. At other times a few tests are conducted.
Avatar n tn Hi, It would had been really helpful if you had shared details about your illness and symptoms Exocrine pancreatic insufficiency can cause atrophy of the acinar cells in the pancreas. These changes could be due to any injury, inflammation, infection or malignancy in the pancreas. A clinical correlation with the test results is important. It is best to consult your doctor and discuss these results. He will explain you best since he is knowledgeable about your history and clinical findings.