epinephrine v fib

It probably isn't the lidocaine, it's the epinephrine that is in the lidocaine. You can ask your dentist to use an anesthetic without epinephrine, I have had tons of dental work and always ask for that. Dentist doesn't mind at all and I've never had any problems. It doesn't last as long but if you start to feel anything while they are working the dentist will just give you more. I have done fine with it.

My understanding is that once a heart is damaged, the heart is more prone to v-tach and v-fib. In my Dad's case, after 4 heart attacks his heart was so damaged he developed cardiomyopathy and needed an ICD. That would zap his heart back to normal rhythm when it went out of control. Not sure how to stop v-fib. I think v-tach can be controlled to some degree with the proper medications and a pacemaker or ICD.

I have to go have a tooth filled tomorrow and I'm somewhat apprehensive about the anesthetic. I know most often the ones they use contain epinephrine because it's a vasoconstricter and causes the anesthesia to work longer. I know you can request the anesthetic without the epi but then it wears off quickly (like when they are drilling on your tooth). I asked my cardiologist and he said if I didn't get a big dose of the epi, if it was mixed, with the beta blockers I should be ok.

There is no relationship between afib and v-fib. A-fib is a very common rhythm disorder that occurs sometimes in people with healthy hearts. The abnormal rhythm is initiated in the atrium. V-fib is a ventricular rhythm disorder, initiated in a different part of your heart. Atrial arrhythmias are not going to cause you to drop dead and are not considered as serious as the ventricular ones. Sounds like you have had all the tests to rule out a serious issue. Need to try to stop worrying.

I had a-fib and a-flutter since 2004. Last year I developed episodes of V-tach ( 5 of mine were captured on an event monitor). In January I had an ablation for both the a-fib and flutter. Since my ablation I've had no a-fib, no a-flutter and no v-tach. My EP and the EP that did my ablation do not know why I suddenly started going into v-tach.

Due to the frequency of re-occurence of a fib, the MD is thinking of Sotolol but because of the risk of v-fib. I am holding off. Is this side effect a common occurence? I am tired all the time. Could that be from the a-fib or the toprol?

Hi. I'm 24 male dip a can of tobacco a day. I know. I have an anxiety and panic disorder. I think I have been getting these pvcs. It feels like my heart skips or stops for a minute. Then back to normal or heart rate may rise a little. During the skipped heart beats if lying on my left side I get a regular beat the pause then regular beat pause. I usually only get them once a day. Couple times a week. My blood pressure raises a little during pvcs plus I can feel it.

Stroke CAN kill. V-fib without prompt treatment WILL kill. Within 45 seconds after you go into v-fib - i.e. your heart stops pumping - you WILL lose consciousness. Within 3 minutes of onset of v-fib without treatment (CPR or cardioversion) your brain starts to die. Stroke CAN kill but absent VERY PROMPT treatment v-fib WILL INVARIABLY KILL YOU. Hence medication for rate control.

From what I read the protocol for v-fib is to start at 200 J and keep working up until it resolves. V-tach protocol starts at 100 J, then work up until it resolves. Could mean nothing but it might be another clue, in other words I'm still thinking it was coarse v-fib. Where, when, did this happen? How long were you out before someone with an EKG and defib got to you?

Idiopathic V-fib is almost never seen. There are no guarantees, though. You can be hit by lightning. STOP spending your entire life being afraid to die from cardiac arrhythmias. You actually slightly increase your risk when producing all this adrenaline.

IF you have not followed up with a cardiologist, you certainly should. V-fib is a deadly rhythm and people who go into V-fib without cause usually have an internal defibrilator placed. This definitely qualifies as a cardiac problem and needs to be evaluated by a specialist if you have not done so as soon as possible. Good luck.

It showed short runs of a-fib and v-tach, but very inconsistently, but surprising and a bit worrisome. I have a 15yr history of arrhythmia and also have chd and have had strokes because of the chd. I am currently on aspirin as well as other meds including a new prescription for multaq, but does the a-fib warrant coumadin? I just read about a-fib and I don't want to have another stroke.

Okay, well this turned out to be a pretty information-packed, long response, but I believe it can do a lot of good for you in understanding your situation if you go ahead and read it. I would also like to warn you that I am not a doctor. I am just a student, who happens to have a particular area of interest in the heart. Maybe someday I'll be a cardiologist or something, but I'm not one now, so take everything that I say with the knowledge that I am not a licensed doctor or anything.

I also drink beer everyday.1.Can I develop V-Fib with normal heart test?2.Can drinking alcohol cause V-Fib in someone with normal heart test?3.can pvc's turn into v-fib?4.do my symtomatic pvc's and other weird heartbeats that waken me make me more likely for v-fib?These episodes happen most that waken me from sleep,and early morning hours,especially after night of drinking, but also without drinking.

The second event, which is less common (although it becomes more common as we age into later adulthood), is what occurs if you have atrial fibrillation (a-fib) or atrial flutter, which are also types of SVT. A-fib and atrial flutter are very fast arrhythmias of that occur in the atria, in which the atrial rate can go 200 to 400, or more, beats/minute. Our A-V node can’t conduct that quickly, so our ventricles don’t beat much over 200 to 220 beats/minute.

Plus, in the absence of a good signal from the atria you can easily go into ventricular tachycardia (v-tach) which can escellate into ventricular fibrillation (v-fib). FYI, absent cardioversion by a defibrillator v-fib is pretty much 100% lethal. When the ventricals are in fib they aren't pumping. That tends to be lethal. I suspect that you're being given atenolol to control your heartrate to keep you out of v-tach. Again, I got used to it pretty quickly.

Without a shock from an ICD or paddles, you would be dead within minutes if you had v-fib. V-fib is the deadliest arrhythmia you can have short of total asystole (which at that point, can it really be called arrhythmia? Is music without sound still music?), and is just about 100% fatal without quick medical intervention. And Jerry's right, a-fib in and of itself is not life threatening. V-fib is life ending. Google ventricular fibrillation for yourself.

Should I get the EP study done or can this be benign? 6.Whats the chances of V-Fib and SCD during EP study and ablation? 7.How bad is it to have a 26 beat run of v-tach??

When a person is in v-tach and the v-tach degenerates into v-fib, is the transition abrupt (like the transition from normal sinus rhythm to v-tach) or is it gradual? By gradual, I mean the v-tach becomes progressively finer and faster until eventually it can no longer be defined as v-tach. I have looked for these stats everywhere but I can't find much of value.

I'm a 24 yr old male. Echo, stress test, stress echo, holter, and event monitors have all been normal besides "PVC's and a 26 beat run of ventricular tachycardia at 127bpms that self terminated". I seen an EP today and he said that he thought it to be benign and called it Idiopathic Ventricular Tachycardia and I started asking questions and he said that just to be safe he wants to do an EP study next week. Can you explain an EP study? How safe are they to do?

t prevent me from going into a V-Tach or even V-Fib (which is my main concern). I saw the EP doc as well and he was less than reassuring, stating that the PACs could lead to A-fib, but that I should just 'go back to cardio if I felt like I was having more PACs"? Is there a reason why I'd have these changes (in echo & now w/ PACs) in such a short amount of time? Is it dangerous to have these changes - will they progress to V-fib or A-fib?

Epinephrine v fib

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