# Calculating heart stroke volume

## stroke

i had a echocardiogam which shows that my stroke volume is 47 ml is it a normal value or it is a sign of heart failure.all other things are normal in my echo. my ejection fraction is 61.4%.
Stroke volume is a calculation to determine ejection fraction (EF)...the amount of blood pumped into circulation with each heartbeat. The calulation for EF is volume of blood in the left ventricle at systole (pumping) phase. Subtract the volume left in the ventricle after diastolic (filling) phase. Then divide the volume of blood during systole for a fraction...that would be EF. Your EF idicates you have a normal pumping heart. Normal is 50 to 70%.
Diastolic volume (filling phase) per stroke is blood volume measured at end of diastolic (maximum filling capacity...relaxed state) minus the volume at the end of systole (minimum amount of blood left in the chamber). Divide end diastolic volume into the difference between end systole and end diastole is the fraction (%) pumped with each stroke. CHF by definition is an EF below 29% (left side non-compliance) indicating there may be an insufficient blood/oxygen supply meeting system demand.
You post indicates about 10 to 15% is pumped out with each stroke. All or almost all of the heart is working (sometimes heart cell damage from a previous heart attack impedes heart wall movement reducing contractions), but enlargement itself can cause a weaker heart and arrhythmia.. The heart quickly enlarges when it is overworked by pumping against the high resistance of constricted blood vessels. Also, the kidneys increase the volume of blood adding to the workload.
Blood in the heart before the heart beats minus blood in the heart after the heartbeat, divided to blood in the heart before heartbeat. Or: Stroke volume / End diastolic volume. It should be between 50 and 80%. What is yours?
The difference between end-diastolic and end-systolic volumes is the stroke volume, the volume of blood ejected with each beat. Ejection fraction (Ef) is the fraction of the end-diastolic volume that is ejected with each beat; it is stroke volume divided by end-diastolic volume. Normal in a man is 50-68%. With a given sized ventricle and its volume, and its ability to contract, I fail to see how a faster or slower heart rate will affect the volume of blood leaving the chamber.
The left ventricle will dilate to increase the cardiac output with each stroke (or decrease), the heart rate will increase cardiac output (amount of blood pumped for each cycle...one minute), the blood pressure will vary and helps maintain the volume of blood in circulation, etc. If there is a shortfall or increase of any one of those factors, that will effect the other compensitory factors.
2) It's the slow heart rhythm that makes me question if the monitor is wrong (and if it's wrong calculating the slow heart rhythm it may be wrong calculating the rapid rhythm). It's impossible to exercise with a heart rate below 50. If you're running uphill, I would guess the body needs a cardiac output of at least 15 liter/minute. That would mean that your heart would have to pump 300 ml of blood for each beat. Not even top athletes have a stroke volume above 180-200 ml, so that's impossible.
Chances are, if you receive one that is significantly lower, than you’ve just done the calculations incorrectly. However, if the pulse pressure is genuinely low, it reflects a low stroke volume, which means your heart is not pumping out the amount of blood it is supposed to. This could be because of a very serious problem, such as congestive heart disorder or shock.
An echo calculates heart chamber diminsions and volume of blood pumped with each stroke. The math equation is diastole volume (peak filling) minus systole (volume after pumping) divided by diastole for the percentage. You can calculate your fraction shortening (FS) which is similar to EF but relies on dimensions rather than volume. To calculate subtract systole dimension from diastole dimension and divide by diastole for percentage. FS is an estimate of myocardial contractility.
Chances are, if you receive one that is significantly lower, than you’ve just done the calculations incorrectly. However, if the pulse pressure is genuinely low, it reflects a low stroke volume, which means your heart is not pumping out the amount of blood it is supposed to. This could be because of a very serious problem, such as congestive heart disorder or shock.
I believe that is consistant to what I said. CO is a product of blood volume per heart stroke and heart rate. The higher/better the stroke volume (weak, strong, stronger contractions) the fewer heart beats (more for weak, less for strong, least for stronger) are required to meet demand (held constant for illustration) for blood oxygen. A well-condition individual requires fewer heartbeats due to strong contractions. It is the stroke that determines EF...
Hi, By using some formulas, Fractional Shortening = (LV End diastolic diameter - LV end systolic diameter) / LV End Diastolic diameter % Ejection fraction can roughly be estimated as Fractional Shortening x 2. Stroke volume = End diastolic volume x EF. I don't remember the formula to calculate end diastolic volume, but my LV End diastolic diameter is 52 mm and my LV End diastolic volume is 135-140 ml.
End diastolic - end systolic = stroke volume. The percentage is calculated from these. You are simply looking for efficiency, then if the EF is low, you look for the cause. So I really don't understand the point you are making.
As you know EF is a measurement of the heart's pumping output in VOLUME with each stroke. The heart chambers' size is not rigid (unless hypertrohic, etc.) but elastic and varies in dimension (LV volume capacity) to maintain a compensatory balance between the right and left chambers when there is not a dysfunctional system. To better understand, there is a similar test called fractional shortening (FS).
Basically the volume of blood in a ventricle is calculated when the heart is relaxed. Then the volume of blood remaining in the ventricle after it has 'squeezed' is calculated. This then allows us to determine the volume of blood that has been pumped out of the chamber. We can work out a percentage of the total blood ejected from the heart from these numbers and that is your ejection factor. The volume in a ventricle chamber when it is relaxed(before squeezing) is the end diastolic volume EDV.
CO (volume of blood pumped with each heartbeat) there is a variation of volume of blood pumped with each stroke to maintain a balance between left and right side blood flow) and abviously PAP will vary accordingly (stronger heartbeat,higher PAP. If there are blockages (clots) in the lungs or pulmonary artery (increase of resistance the heart pumps against, and/or if there is narrowing, stiff pulmonary vessel the more resistance (increase of resistance will cause higher PAP).
It's not a measure of overall heart output from the left ventricle. That is a seperate measurement, called the Stroke Volume. Can you find out what his SV is? It sounds like his heart was damaged at a young age and has had time to adapt. What a great case study this would make though, someone should study it more to find out how his heart adapted. Given the LVEF, it doesn't sound like his LV contracts very well. SOmething has to be making up for it. Perhaps the LV grew larger?
increases blood pressure and volume of blood available to the vital organs, the heart pumps more blood (increase in blood flow) that will increase heart CO (stroke voume times heart rate.
For some insight, the pulse pressure is determined by the interaction of the stroke volume of the heart (each heartbeat), compliance (ability to expand) of the aorta, and the resistance to flow in the system arterial complex. By expanding under pressure, the aorta absorbs some of the force of the blood surge from the heart during a heartbeat. In this way the pulse pressure is reduced from what it would be if the aorta wasn't compliant.
I had an echocardiogram later in that month and everything was normal according to the report. Atria were normal size. Blood ejection volume was normal etc. I also had a heart event monitor which showed no atrial fib for the month after my episode. My GP doctor (not a heart specialist) said that he was happy with the results and decided not to do anything. I'm on 81mg of aspirin and also atenolol for high blood pressure. My cholestrol test are very good, and I have no diabetes.
From this, my stroke volume is 125 x 0,57 = 71 ml and CO at heart rate 80 = 5,7 l/hour? If my heart rate increases to 200, my CO is 14,2l/hour. Anyway, I guess my EF increases a bit during heavy exercise (mid to upper range heart rate), and reduces if HR is too high, like the torque on an engine, right? A strange thing was, during my echocardiography, I was very scared and I felt this light, but somewhat elevated heart rate (100-120), possibly lowering EF?
The pulse pressure is determined by the interaction of the stroke volume of the heart, compliance (ability to expand) of the aorta, and the resistance to flow in the arterial tree. By expanding under pressure, the aorta absorbs some of the force of the blood surge from the heart during a heartbeat.
Your report doesn't show a calculation for the amount of blood pumped with each stroke (that would be the ejection fraction...50 to 70% normal). EF calculations is based on blood volume before and after contraction. Your report shows a calculation of fractional shortening of 32%. FS is the LV dimension chamber size of the LV before and after contraction and that is normal...greater than 26%. You FS is normal at 32% Sometimes the EF is normal , but the filling capacity is limited....
amount of blood pumped with each stroke) differs from FS in that the EF calculation deals with volume (end systole volume subtracted from end diastole volume divided by end diastole vloume. Normal is 50 to 70%. The calculation shows that an increase in end diastole (dilated LV) increases EF and that accounts for the slightly higher than normal EF. FS (fractional shortening) calculation deals with left ventricle dimensions.
Athletes often have a fairly low EF at rest, as the heart relax very well, but they are able to increase stroke volume and EF quite a bit with exercise. What matters is that you are able to run, if you can run fast for a long distance without symptoms you don't have heart failure! Your FS is 22,5% (4,9-3,8)/4,9 = 0,225 EF can be roughly estimated as FS x 2, but this formula will under-estimate EF at lower FS and over-estimate it at higher FS.
I also meant to say that I have Hashimotos Hypothyroidism that is still not controlled. My last TSH was 5.99. I take 150 mcg of Synthroid.
Hello :) 1. A "normal heart rate" simply can't be defined. We are all different, and what's normal to some people isn't normal for others. Think of it as cars with diesel or gas engines, one engine has a "normal" RPM when driving in 80 km/h on the highroad (yes, in Norway that's the speed limit, ridiculous :p) of 1800 and another 2700. But the first engine will probably stop at 4500 RPM and the other on 7000 RPM.
The EF is one factor that maintains a balance of blood flow between left and right sides of the heart. Cardiac output is a function of stroke/volume for 1 minute. Pressure expands the left ventricle and increases preload (filling) . An increased preload increases stroke volume through the Frank Starling mechanism and that would be an increase in EF. Frank Starling mechanism can be campared to a hand spring as an analogy.
This process reduces stroke volume and cardiac output, causing effort intolerance. Figure 117 summarizes the pathophysiology of diastolic heart failure. In addition to providing fundamental information on chamber size, wall thickness and motion, systolic function, the valves, and the pericardium, two-dimensional echocardiography with Doppler is used to evaluate the characteristics of diastolic transmitral and pulmonary venous flow pattern.