Blood platelets sticking together

Common Questions and Answers about Blood platelets sticking together

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Good Morning Everyone: My doctor informed me that my platelets had dropped to 58k and that on Pegasys it calls for treatement to be stopped if they go as low as 70K. I go in for more lab work today to confirm. My neutrophils have gone down to 562 and he wants me to increase the dose of nupogen on Saturday nights. Something else was down to 31 - 33 but I had lost interest in the conversation at that point. Wishing everyone wellness.
I start a 4-week lead-in with eltrombopag (Promacta) today to try and and see if we can raise (stabilize) my platelets at 50k, where they are at now. My last Tx using Infergen (daily shot of non pegylated interferon) dropped my platelets to 6k and Tx was stopped. My 1st 48 week Peg/Riba Tx lowered my platelets from ~100 down to 50 where they have been and my 2nd Tx they stayed at ~50. Good thing is the eltrombopag works; bad thing, it is hepatoxic esp. at higher doses.
I've heard a lot about low HGM and platelets, but not much about WBC. I also called a nurse at Roche over the weekend, and she made a bet with me that it was because my medical providers were concerned about my WBC -- she was right. Any info about my low WBC at this point would be appreciated.
I am so glad to hear that others get it too. I am thin, athletic with low blood pressure. I do have low platelets and have a border line low thyroid problem. Other wise I am in good health.
If you've done more than one, you should post the series of biopsy results together with comparative blood data. There are almost no horizontal studies over time of fibrosis progression (biopsy) vs. blood scores in individuals. It's some of the info that is sorely lacking. Thanks for the suggestion, but I don't think I'll go to Brazil for Int./Riba. + Telaprevir. I realize I may come to regret it.
The drug Plavix may be a little safer than warfarin. It helps keep platelets from sticking together and forming blood clots. You can discuss this alternative with your doctor.
Plavix works like aspirin in preventing blood clots by preventing platelets from sticking together forming a blood clot. Lipitor is a statin and in Lipitor studies have been shown to reduce cholesterol levels and has been shown to reduce buildup in the heart as well. You can goggle Lipitor studies to find out more information.
i had sexual intercourse about 7 or 8 times before seeing a doctor. the same occurred except for 2 times, in which no blood appeared. but the majority of times blood came out in the sperm, or only after ejaculation during urination, or both in the sperm and the urine. once i woke up without having had sex that week and urinated blood. i tested my blood, sperm, and urine and the only abnormality was a high psa level. my dr said it is very rare and is that high in 40/50 year old.
If the baby boomers caught Hep C in the '60s but didn't know it and gave blood like normal public spirited citizens and Hep C testing of donors did not commence till 1992, shouldn't everyone who received a transfusion prior to that date get a test? The blood supply prior to that date must have been very infected! The quickest and cheapest test seems to be to attempt to give blood.
It's primarily baby aspirin, but also contains a drug that prevents platelets from sticking together. I've taken it twice a day since my stroke, and it appears to work, but who ever knows?
Both drugs are some what the same, they both work by keeping your blood platelets from sticking together and forming clots. Plavix is a adenosine diphosphate (ADP) receptor and Pradaxa is a direct thrombin inhibitor. Both do basically the same thing just in different ways.
Hi I think that you should really see a specialist haematologist to help get the correct treatment for you considering that you have evidence of neurological damage. I used to be a haematology scientist, so I have put in a bit of background below to help bring you up to speed. But please do get checked out & treated - you don't want to end up with a full on stroke.
Plavix stops platelets aggregation or prevents them from sticking together. This tends to prevent clot formation in the arteries, including coronary arteries or the arteries on the surface of heart. Its action is same as that of Aspirin, and is always better than Coumadin for someone with major risks in the arteries (heart attack, stroke). Plavix does not influence the PT/INR.. Plavix' action is not influenced by vitamin K intake.
Plavix stops platelets aggregation or prevents them from sticking together. This tends to prevent clot formation in the arteries, including coronary arteries or the arteries on the surface of heart. Aggrenox (aspirin/extended-release dipyridamole) is indicated to reduce the risk of stroke in patients who have had transient ischemia of the brain or completed ischemic stroke due to thrombosis.
GARLIC- excellent for the heart it helps the platelets from sticking together. HAWTHORN- DILATES THE CORONARY ARTERIES, AND INCREASES BLOOD SUPPLY TO THE HEART. GINGER-reduces "ANGINA" also lowers cholesterol, and helps the platelets from sticking together. "GINGER" works better taken on an empty stomach. you can eat about ten minutes after. also what I take for Anemia. it helps for when my iron is low. FOR YOUR KIDNEYS- your kidney mechanisms might be overwhelmed.
If that is the case, you should talk to your doctor about taking 81mg aspirin daily to keep your blood thin and platelets from sticking together. Once a person gets about age 80, the platelets do not stick and the person can actually have a problem with clotting. Fish oil is great to keep the arteries healthy. Make sure that you are getting enough of the vitamin B's as many people are not and it is essential for proper heart function.
Plavix works like aspirin. It prevents the platelets from sticking together causing a blood clot. Coumadin works differently and is for blood clots in the arteries and veins. Coumadin is to be used with caution in people with CHF and he needs to be monitored very closely if he is taking Coumadin. With CHF, ace inhibitors are best such as Lisinopril. Ace inhibitors can also help reduce pressure in kidneys, lungs, and eyes (if glaucoma exists).
My tx is a bit weird insofar as I added the VIC at w30 and am planning on only 18w but the sx may reflect what others will run into. The late start means my blood cell production had already been hammered by 7mo of soc so the extra drops are primarily due to the VIC. It also means there's no VL data since I was UND about w8, 22w before starting the PI. The best part is there's no ugly part. Everything is tolerable though no fun. The bad: HGB dropped 0.7 from 11.7 to 11.
The two drugs work on different types of clotting systems. Aspirin is an antiplatelet agent. It keeps platelets from sticking together and forming a clot. Coumadin keeps your liver from using vitamin k to manufacture other types of clotting factors in the blood. If it were me in your BF's situation (and I do take coumadin), I would find any way I could to get back on coumadin ASAP.
Esophageal varices develop when normal blood flow to your liver is slowed. The blood then backs up into nearby smaller blood vessels, such as those in your esophagus, causing the vessels to swell. Sometimes, esophageal varices can rupture, causing life-threatening bleeding." I have had a number of endoscopies to monitor my varices. You won't be conscious so you won't feel a think during the procedure.
The spleen have the ability to tripple itself in size so it is very rare the it will ever burst. As the blood flows through the spleen the platelets will not all make it. many will drop into the bottom of the spleen and lay there. Giving the patient a low platelet count which makes it difficult for your blood to clot when you hurt yourself. The "Ammonia" level. The body makes ammonia to help break down the bateria in the intestines.
5 on the shot and 600 mil in the morning and 600 at night and one trial pill for platelets, for the last 4 months.I'm going to miami for this trial and was treated OK in the beginning but now I have to wonder.Last month I was going again to pick up more medicine, but unfortunately the person that takes care of me, was not there even though 2 weeks before that day I told her I would be there on that friday so she said OK and that was the end of it for that conversation.
when there is little turbulence in the blood the platelets tend to want to stick together. Get enough of these stuck together platelets and you have a plug for the artery. Aspirin will help keep the platelets from sticking together. and this also includes external cuts where if you take too much aspirin the blood will not stop flowing out of that cut. And I just reminded my self that this also includes internal "cuts(?)" such as ulcers.
In many cases, people with ITP die because they don’t have enough platelets for their blood to clot. Ordinarily the “only” treatment for this is months—even years—of treatment with Prednisone, an artificial form of cortisone, which can cause bone thinning, hypertension, suppressed immune system, and cataracts. Now there may be another option. One investigation of 16 patients with ITP found that Helicobacter was present in 87 percent.
There exist other anticoagulants that can be taken orally and work by different mechanisms. Ticlid is a medicine that inhibits platelets from sticking together. This is the initial process in clot formation. Aspirin, not Tylenol or aceteminophen, works on platelets in a similar fashion to prevent them from sticking together. Coumadin, or Warfarin, is yet another medication that inhibits the coagulation cascade that occurs after platelet aggregation.
) I've been reading many of the postings and if not mistaken it's been said the Peg/INF is what reduces the platelets. Soooo... this next blood draw and determination whether I continue is on a Wednesday morning. The shot is Monday night around 23:00 giving me about 36 hours until evaluation. If I can increase the distance between shot and evaluation by a day or two, maybe that gives a little edge. I'm doing newleaf09's suggestion of exercise (not running though!
Conventional treatment has been drugs like aspirin to prevent blood platelets sticking together and clotting the artery.Surgery, although possible is very difficult high up in the neck and at the base of the skull where this problem occurs.Therefor, occasionally endovascular treatments have been considered eg.stenting. This procedure is used with success in some other large arteries in the body and early reports in the medical literature suggests a role in the carotid artery.
Dake did give me two months worth of Plavix and baby aspirin (both are anticoagulants that prevent platelets in the blood from sticking together; their functional mechanism is different and additive). I was worried that I wouldn’t be able to tell if this was working, but I do seem to be getting some effects or at least a noticeable placebo effect (see below). I am also worried about restenosis and how I would know whether I have it. Dr.
I recently had blood work, I am SVR and my doctor said they are now considering this a cure. He also said they are finding that while the liver regenerates it is also regenerating the scarred tissue. I feel better than I have in years. My mind is clear, my hair is growing back, my muscles are building back up. I am very happy that I took the chance JimJim530 came back and said he went from a 3-4 and now is normal, that is great news and I hope encouraging to others on tx.
She said my cbc's were normal but not one of the numbers was normal. I have low platelets. low rbcs. I was referred to hematologist who did more blood work. He said this was blood isssue from an autoimmune issue and not bone cancer. My ferritin and iron came back low on his tests. I'm on plaquenil and colchichine. Liver doc said I probably have autoimmune heptatitis but he wants the rheumy to dx for it because imuran is appropriate for several autoimmune issues I have so more waiting.
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